Sentences with phrase «in oxidation levels»

When tasted side - by - side you can literally taste the difference in Oxidation levels.

Not exact matches

The high - level of antioxidants and other phytochemicals found in nuts and seeds help prevent chronic and inflammatory diseases by protecting the body against cell oxidation.

At standard levels in which one might add pure vanilla extract in recipes, the vanillin concentration is sufficient enough to protect against oxidation and lipid peroxidation.

Depending on where in the kiln the pieces are placed, the scattering of the ash, the flame path and the oxidation and reduction levels have varying effects in color and texture.

Questions that can't be answered in the lab Sunday and her team also suggest more work should consider a species» response to multiple environmental changes, such as increased temperature and oxidation levels, as well as multiple stages of life.

Rising sea levels, for example, would result in increased sulfate, which could fuel greater rates of anaerobic oxidation.

These whiffs of oxygen likely happened in the following 100 million years, changing the levels of oxygen in Earth's atmosphere until enough accumulated to create a permanently oxygenated atmosphere around 2.4 billion years ago — a transition widely known as the Great Oxidation Event.

We are looking for candidates interested in combining experimental and theoretical approaches to study the regulation of protein oxidation at molecular and cellular levels.

The enhanced hiPS - HEP cells express PCSK9 and other genes that regulate blood cholesterol levels, as well as principal genes involved in uptake, synthesis, and beta - oxidation of fatty acids.

Understanding the kinetics and mechanisms causing intergranular oxidation at the atomic level can improve manufacturing for more durable, corrosion - resistant materials designed for safe application in service environments, for example, in a light - water (nuclear) reactor within its decidedly high - temperature, reactive environs.

The scientists sought to determine the oxidation levels of the magmas that formed these ancient zircons to quantify, for the first time ever, how oxidized were the gases being released early in Earth's history.

Understanding the level of oxidation could spell the difference between nasty swamp gas and the mixture of water vapor and carbon dioxide we are currently so accustomed to, according to study lead author Dustin Trail, a postdoctoral researcher in the Center for Astrobiology.

To do this Trail, Watson, and their colleague, postdoctoral researcher Nicholas Tailby, recreated the formation of zircons in the laboratory at different oxidation levels.

This chemical weathering process is too slow to damp out shorter - term fluctuations, and there are some complexities — glaciation can enhance the mechanical erosion that provides surface area for chemical weathering (some of which may be realized after a time delay — ie when the subsequent warming occurs — dramatically snow in a Snowball Earth scenario, where the frigid conditions essentially shut down all chemical weathering, allowing CO2 to build up to the point where it thaws the equatorial region, at which point runaway albedo feedback drives the Earth into a carbonic acid sauna, which ends via rapid carbonate rock formation), while lower sea level may increase the oxidation of organic C in sediments but also provide more land surface for erosion... etc..

Those levels rose sharply during periods of rapid brain development and in response to radiation, oxidation and other factors known to cause breaks in DNA.

In addition, high - intensity cardio will increase your resting metabolic rate for upward of 24 hours after exercise, increase growth hormone levels, improve insulin sensitivity in the muscles and spike levels of fat oxidation in muscle cellIn addition, high - intensity cardio will increase your resting metabolic rate for upward of 24 hours after exercise, increase growth hormone levels, improve insulin sensitivity in the muscles and spike levels of fat oxidation in muscle cellin the muscles and spike levels of fat oxidation in muscle cellin muscle cells.

Iron can reach toxic levels in the body with regular use and becomes a pro-oxidant which causes stress, oxidation and eventually disease.

Stress resistance has not been assessed however and so the biological relevance of this finding is currently unknown.32 Several IER trials (75 - 85 % ER on restricted days) in overweight / obese populations have reported reductions in various markers of oxidative stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin overweight / obese populations have reported reductions in various markers of oxidative stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin various markers of oxidative stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research DirectioIn a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Direlevels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research DireLevels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin AOPP in both groups after three months.48 Summary and Future Research Directioin both groups after three months.48 Summary and Future Research Directions

And since the link between oxidative and non-oxidative pathways, and heart rate is mediated by hormone levels, aerobic function isn't really going to be mediated by cardiovascular fitness: the hormonal changes that result in a reduction of oxidation and increase in anaerobic function occur at a very high heart rate reserve (long, long before your cardiovascular fitness maxes out).

Studies in rodents have additionally described post-treatment elevations in the rate of fatty acid oxidation within both the liver and skeletal muscle of mice maintained on IER (100 % ER / alternate days).13 In a comparison of CER (40 % ER / day) and IER (100 % ER / alternate days), the authors of this 20 - week study noted a doubling in the Î ² - hydroxybutyrate levels (a marker of mitochondrial fatty acid oxidation) in IER - fed mice which was not present in the CER group.15 Both IER and CER interventions led to comparable reductions in fasting levels of glucose and insuliin rodents have additionally described post-treatment elevations in the rate of fatty acid oxidation within both the liver and skeletal muscle of mice maintained on IER (100 % ER / alternate days).13 In a comparison of CER (40 % ER / day) and IER (100 % ER / alternate days), the authors of this 20 - week study noted a doubling in the Î ² - hydroxybutyrate levels (a marker of mitochondrial fatty acid oxidation) in IER - fed mice which was not present in the CER group.15 Both IER and CER interventions led to comparable reductions in fasting levels of glucose and insuliin the rate of fatty acid oxidation within both the liver and skeletal muscle of mice maintained on IER (100 % ER / alternate days).13 In a comparison of CER (40 % ER / day) and IER (100 % ER / alternate days), the authors of this 20 - week study noted a doubling in the Î ² - hydroxybutyrate levels (a marker of mitochondrial fatty acid oxidation) in IER - fed mice which was not present in the CER group.15 Both IER and CER interventions led to comparable reductions in fasting levels of glucose and insuliIn a comparison of CER (40 % ER / day) and IER (100 % ER / alternate days), the authors of this 20 - week study noted a doubling in the Î ² - hydroxybutyrate levels (a marker of mitochondrial fatty acid oxidation) in IER - fed mice which was not present in the CER group.15 Both IER and CER interventions led to comparable reductions in fasting levels of glucose and insuliin the Î ² - hydroxybutyrate levels (a marker of mitochondrial fatty acid oxidation) in IER - fed mice which was not present in the CER group.15 Both IER and CER interventions led to comparable reductions in fasting levels of glucose and insuliin IER - fed mice which was not present in the CER group.15 Both IER and CER interventions led to comparable reductions in fasting levels of glucose and insuliin the CER group.15 Both IER and CER interventions led to comparable reductions in fasting levels of glucose and insuliin fasting levels of glucose and insulin.

It is generally advisable to build a high level of Glutathione in ones blood to protect the body from oxidation (damage to the tissue by free radicals).

And you know, the conventional view is that they have that higher risk because of higher levels of LDL, but actually the most recent research and our recent understanding of what causes both Alzheimer's and heart disease suggests that the reason they have a higher risk is that ApoE4 carriers are more susceptible to oxidative damage and inflammation, and it's the oxidation of the LDL particle that's the common thread in both increased risk of heart disease and increased risk of Alzheimer's and dementia.

The researchers found that all the women who participated in the study increased their fat oxidation levels after periods of religious fasting.

Keeping the randall effect in mind, in the case of a headache, blood levels of free fatty acids are inhibiting glucose oxidation by cells.

Originated 1970s by Dr. Henry Lemon, who tested estrogen levels in 24 hour urine samples and found that an EQ > 1 strongly correlated with a higher survival rate after breast cancer.24 Further research conducted by Lemon, Heidel, et al., a meta - analysis of published fractional estrogen excretion collected from 2,846 healthy women worldwide aged 15 to 59 years, with a risk of breast cancer varying five-fold, found that an EQ < 1 reflects increased rates of oxidation of estrone or estradiol to 4 - OH catechols (also referred to in the literature as the 3,4 - catechol estrogen quinones), which have been identified as the principal proximal human mammary carcinogens after menarche, while an EQ > 1 reflects conversion to protective 2 - OH estrogen metabolites.2526

Oleuropein not only lowers levels of harmful LDL, but prevents its oxidation — which is one of the first steps in the development of atherosclerosis.

This serum in particular keeps skin clear and decongested, yet hydrates on a cellular level to prevent oxidation and aging.

It accomplishes this by increasing the levels of antioxidant enzyme systems in your body helping the body to achieve an overall lowering and balancing of the oxidation levels, and thereby helping to bring the body back to a state of health.

Ramos O, Carrizales L, Yanez L, Mejia J, Batres L, Ortiz D, Diaz - Barriga F. Arsenic increased lipid per oxidation in rat tissues by a mechanism independent of glutathione levels.

Much of the damage done in diabetes is done by the circulating sugar and / or high insulin levels causing oxidation, inflammation and acidosis.

• EXOGENOUS INSULIN: Although insulin has been shown to improve memory and cognition acutely, chronically high insulin levels are known to impair brain function.42, 64 Exogenous insulin would serve to inhibit IDE more strongly, thereby preventing the clearance of Aβ, causing it to linger in the brain interstitial fluid even longer, where it is subject to glycation and oxidation.

Not just used to reduce nausea and improve digestion, gingerols have shown to support healthy blood sugar levels, and increase lipid oxidation slightly, great to add to green smoothies raw or use in stir - fry.

Curcumin has been attributed with extraordinary benefits for heart health, including an ability to lower cholesterol levels and prevent the oxidation of cholesterol in the body, thus preventing cholesterol from damaging blood vessels.

The key difference is that the athletic activity in indigenous cultures provides the perfect mechanism to reduce insulin levels: as soon as the volume of athletic activity creates a drop in blood sugar (which happens a lot in indigenous cultures) insulin levels drop, which means that leptin levels rise: fat oxidation (and oxidation in general) increases dramatically.

Similar to the physical and cellular burn of exercise that creates some oxidizing free radicals, the «stress» from cruciferous vegetables stimulates our mitochondria to produce homemade antioxidants to counterbalance the oxidation and protect our vital DNA.28 Even cancer cells have caught on to this central role of our mitochondria — the bastards can amplify their levels of some of these same pathways — further stressing their importance for survival.25 However, our cells still seem to have the upper hand, as multiple studies show that the organosulfur chemicals in many of these anticancer vegetables can severely stress cancer cells, leading to oxidative damage, and ultimately, death.29

For example, a 2007 Study in the Journal of Applied Physiology reported that young women who performed seven HIIT workouts over a two - week period experienced a 30 - percent increase in both fat oxidation, and in the levels of muscle enzymes that enhance fat oxidation.

Due to high levels of antioxidants, it reduces oxidation during exercise and also helps in quick muscle recovery.

Fortunately, nowadays we can find more and more research linking vegetable oils to a high level of oxidation present in the body.

Furthermore, passion flowers - and, to some extent, the fruit of the plant - contain flavonoids that prevent the oxidation of testosterone and have a positive effect on estrogen levels in women.

This is because ACV may help lower triglyceride levels and blood pressure, and also protect LDL particles from becoming oxidised (oxidation is a crucial step in the heart disease process).

Polyunsaturated fats such as corn oil and soybean oil are highly unstable and vulnerable to oxidation — and oxidation, in turn, is linked to cancer — while the omega - 6 fatty acids that predominate in vegetable oils have been shown to accelerate the growth of tumor cells.6 As Fallon and Enig explain, 4 excessive consumption of toxic polyunsaturated oils — not red meat — represents a known mechanism for colon cancer: «Colon cancer occurs when high levels of dietary vegetable oils and hydrogenated fats, along with certain carcinogens, are acted on by certain enzymes in the cells lining the colon, leading to tumor formation» [Emphasis added].

The benefits are lengthy, from memory enhancement, neuron health, increases acetylcholine levels, anti-oxidant role in the brain, and of course can aid in mitochondrial fat oxidation.

Consensus statements from leading governing bodies generally recommend that carbohydrate quantities should range from 3 to 12 g kg 1 body weight.9 - 10 However, recent work suggests that LCHF diets that raise blood ketone levels can increase fat oxidation rates and markers of health and exercise performance.11 - 12 In addition to increased fat oxidation, other potential benefits of LCHF eating plans for endurance athletes include improved training and racing energy, lowered incidence of delayed onset of muscle soreness, reductions in exogenous caloric requirements during training and competition, and the reduced incidence of serious gastrointestinal complaints.13 Although nontraditional, an LCHF eating plan approach has been recommended for athletes in a variety of sports for nearly 40 years, 14 and this report is representative of other caseIn addition to increased fat oxidation, other potential benefits of LCHF eating plans for endurance athletes include improved training and racing energy, lowered incidence of delayed onset of muscle soreness, reductions in exogenous caloric requirements during training and competition, and the reduced incidence of serious gastrointestinal complaints.13 Although nontraditional, an LCHF eating plan approach has been recommended for athletes in a variety of sports for nearly 40 years, 14 and this report is representative of other casein exogenous caloric requirements during training and competition, and the reduced incidence of serious gastrointestinal complaints.13 Although nontraditional, an LCHF eating plan approach has been recommended for athletes in a variety of sports for nearly 40 years, 14 and this report is representative of other casein a variety of sports for nearly 40 years, 14 and this report is representative of other cases.

As we have reported in other places, lipid oxidation is a much more important factor to consider when choosing dietary oils than the saturation level.

The experimental data gathered by Venables et al. measured carbohydrate and fat oxidation in both men and women at various levels of exercise intensity, where exercise intensity was measured as a percentage of VO2max.

A factoid from ACS's Journal of Agricultural and Food Chemistry indicates that naturally occurring polyphenols in cocoa, the fundamental component in chocolate, actually boost levels of HDL, the «good» cholesterol, while at the same time reducing the atherogenicity of so - called «bad» variety — LDL — by preventing its oxidation.

Sesame and their lignans have a broad range of applications in human health, including increasing the anti-inflammatory index of fish oils, decreasing LDL oxidation, inhibiting lipid peroxidation, lowering LDL levels in humans, and guarding against DNA mutating toxins.

Since the race, he also participated in the FASTER study along with a handful of other keto - adapted athletes, demonstrating that extreme levels of fat oxidation during exercise are indeed possible and not detrimental to endurance performance [10].

One study showed that after a 3 day juice cleanse, participants showed significant changes in the intestinal microbiota that are associated with weight loss, decreased lipid oxidation (free radical damage), and higher levels of nitric oxide in the blood and urine which play a significant role in reducing cardiovascular disease.

This little piggy is involved in the regulation of glucose levels and the oxidation of fats.