Sentences with phrase «in diabetic control»

In the diabetic controls nineteen out of twenty marriages were satisfactory, although the fear of genetic transmission existed in that disease also.

Not exact matches

The are extremely high in fibre which is great for your digestive system as well as diabetics as fibre helps control the release of sugars to the blood stream.

They added that the fact meat free diets have fewer calories was helpful in helping diabetic people control their weight.

Canola oil: Recent research shows that this versatile cooking oil improves blood sugar control in type 2 diabetics.

So don't worry about the sugar when eaten in moderation unless you are a diabetic and even then dragon fruit can help with diabetes blood glucose control.

He contributes to other large - scale clinical trials including SAVE (evaluating whether treatment of obstructive sleep apnoea with continuous positive airways pressure can reduce the incidence of serious CV events in patients with established CVD), and CREDENCE, a randomised, double - blind, placebo - controlled trial assessing whether canagliflozin can slow the progression of diabetic nephropathy in patients with type 2 diabetes.

For all diabetic moms good blood glucose control is key to avoiding problems in pregnancy labor and birth.

I was diagnosed as a diabetic in 1994, my control was very poor and by 2007 I was warned that I was entering a high risk zone for a stroke or heart attack.

Compared with newborns of non-diabetic women, children of diabetic mothers with poorly controlled glycaemia show neurophysiological impairment and have a higher risk for metabolic syndrome, obesity and type 2 diabetes mellitus in later life.»

In trials involving two groups of diabetic patients, adults and adolescents, the bionic pancreas provided tighter control of blood glucose than standard measures such as insulin pumps and blood - sugar monitors.

«One of them is the level of sugar control; if you don't control your sugar well your kidney disease progresses faster or if you leak protein in the urine and you have proteinuria, it tends to be an independent predictor of kidney disease in diabetics.»

The species also controlled blood sugar in diabetic mice.

Now, Thomas Jefferson University researchers have found that a different blood - sugar marker is able to predict patients — both diabetic and non-diabetic - with highest risk of complications more accurately, and detect changes in glucose control much faster, which could potentially change clinical practice.

In view of their most recent results, it seems the key lies in the fact that chronic melatonin consumption not only induces the appearance of «beige fat» in obese diabetic rats, but also increases its presence in thin animals used as a control grouIn view of their most recent results, it seems the key lies in the fact that chronic melatonin consumption not only induces the appearance of «beige fat» in obese diabetic rats, but also increases its presence in thin animals used as a control grouin the fact that chronic melatonin consumption not only induces the appearance of «beige fat» in obese diabetic rats, but also increases its presence in thin animals used as a control grouin obese diabetic rats, but also increases its presence in thin animals used as a control grouin thin animals used as a control group.

The study, one of the first to evaluate hormonal contraception and health outcomes in women with a chronic condition, should encourage physicians to include implants and IUDs in birth control discussions with diabetic patients.

While controlling blood pressure, blood sugar and LDL - cholesterol levels reduces the risk of cardiovascular disease in people with diabetes, only 7 percent of diabetic participants in three major heart studies had recommended levels of these three factors, according to research from the Heart Disease Prevention Program at the University of California, Irvine School of Medicine.

More concerning, niacin was associated with an increased trend toward death from all causes as well as significant increases in serious side effects: liver problems, excess infections, excess bleeding, gout, loss of control of blood sugar for diabetics and the development of diabetes in people who didn't have it when the study began.

Controls aged three years and older were characterised by a higher fraction of butyrate - producing species within Clostridium clusters IV and XIVa than was seen in the corresponding diabetic children or in children from the younger age groups, while the diabetic children older than three years could be differentiated by having an unusually high microbial diversity.

The researchers found that in children younger than three years, the combined abundance of the class Bacilli (notably streptococci) and the phylum Bacteroidetes were higher in diabetic children, whereas the combined abundance of the important (usually beneficial) Clostridium clusters IV and XIVa was higher in the healthy controls.

We become less good at controlling the amount of sugar in the blood; it has been said that if doctors did not allow for age, they would have to classify half of the over-sixties as diabetic.

Until now, it hadn't been clear whether strict control of blood sugar and fats, or lipids, could still deter retinopathy in patients who had been diabetic for as long as a decade.

In immunocompromised individuals (chemotherapy patients, for example) or poorly controlled diabetics, upper airway Aspergillus infections can invade the mucosa and underlying tissue and become lethal, so it is particularly critical to understand its interactions with the airway epithelium.

Glycaemia monitoring is a basic element in the treatment and control of diabetic patients.

«Finding a way to control angiogenesis not only provides a target for the development of anti-cancer therapies, but may also prove useful in similarly starving abnormal blood vessel growth elsewhere in the body, like in diabetic eye disease.»

To rule out that the induction observed was due to other diabetic - associated alterations in the pancreas, we performed a control experiment in which we injected Tg - hIAPP mice with pancreas homogenate from a diabetic model not associated with IAPP aggregation.

The results indicated that the administration of diabetic pancreas homogenate, containing IAPP aggregates or synthetic IAPP aggregates, prepared in vitro did not produce any significant changes in the weight of major organs, compared with the controls treated with buffer (Fig. 8 B).

In the study, Joslin researchers examined samples of vitreous (eyeball) fluid from 61 patients with diabetic retinopathy or control group with a non-diabetic form of macular damage.

Fig. 3 shows dose - response curves for the impact of insulin on the fractional velocity FV0.1 of GS in type 2 diabetic (Fig. 3B) and control cultures (Fig. 3A).

In this study, increased risk of death from several causes, including cardiovascular reasons were also described at an older age and for milder stages of renal disease in diabetic women that included a further deterioration due to worsening of glycemic controIn this study, increased risk of death from several causes, including cardiovascular reasons were also described at an older age and for milder stages of renal disease in diabetic women that included a further deterioration due to worsening of glycemic controin diabetic women that included a further deterioration due to worsening of glycemic control.

The aim of the present study was to evaluate glucose transport and GS activity in human satellite cell cultures established from type 2 diabetic and control subjects.

ΔA0.5 is lower for the diabetic group than the control group, but this difference disappears because ΔA0.5 in cultures established from type 2 diabetic subjects significantly increased with increasing insulin concentration.

GS activity in cultures established from diabetic and control subjects at day 8 are shown in Fig. 2.

Acute insulin stimulation significantly increased GS activity at 0.1 mmol / l in both diabetic (P < 0.0001) and control (P < 0.0001) cultures; however, GS activity at 10 mmol / l was unaffected by acute insulin stimulation in both culture types (P > 0.55 in diabetic and control cultures).

S100A4 mRNA expression (A) and DNA methylation (B) differ significantly between diabetic compared with nondiabetic subjects both in the discordant twins and in case - control cohort 2.

C: The absolute differences between the basal and the insulin - stimulated state (ΔFV0.1) in control (▴) and diabetic cultures (⧫).

Glycogen content was significantly decreased in cultures established from diabetic patients compared with those established from control subjects (Fig. 6C).

Furthermore, ΔA0.5 correlated with ΔFV0.1 in control cultures (r = 0.45, n = 64, P < 0.01) and in diabetic cultures (r = 0.54, n = 64, P < 0.01).

Figure 4 shows dose - response curves of the impact of insulin on the activity constant A0.5 of GS in type 2 diabetic (Fig. 4B) and control (Fig. 4A) cultures.

GS activity measured at 0.1 and 10 mmol / l G6P was significantly lower in diabetic cultures than in control cultures (P < 0.0001).

The U-formed shape of the dose - response curve for type 2 diabetic cultures and the parallel curves for high insulin concentration in control cultures indicate that the mechanism for the primary defect is different from the mechanism responsible for the induced reduction in acute insulin stimulation of GS.

SLC37A2 mRNA expression (C) and DNA methylation (D) differ significantly between diabetic compared with nondiabetic subjects both in the discordant twins and in case - control cohort 2.

However, subgroup analysis revealed that as opposed to male groups, carotid atherosclerosis was more prevalent in newly diagnosed diabetic women than in nondiabetic female controls.

We determined the glycogen synthase (GS) activity; the content of glucose -6-phosphate, glucose, and glycogen; and the glucose transport in satellite cell cultures established from diabetic and control subjects.

Acute insulin stimulation significantly increased the fractional activity FV0.1 of GS in control cultures in the range of 0.1 pmol / l to 0.1 nmol / l (P < 0.05) and in diabetic cultures in the range of 0.1 pmol / l to 1.0 nmol / l (P < 0.05), whereas higher insulin concentrations diminished the effect of acute insulin stimulation in a dose - dependent pattern.

Higher insulin concentrations (> 1 nmol / l) significantly reduced the ΔA0.5 in control as well as diabetic cultures (P < 0.05) in a parallel manner.

Higher insulin concentrations (> 1 nmol / l) significantly reduced the ΔFV0.1 in control as well as diabetic cultures (P < 0.05) in a parallel manner.

Preculturing myotubes for 4 days at increasing insulin concentrations did not change the basal FV0.1 in diabetic or control cultures (Figs. 3A and B).

We evaluated the capacity for glucose uptake in control and diabetic cultures precultured under three different insulin concentrations and found that when precultured with 1 pmol / l and 1 nmol / l insulin, the basal glucose uptake was reduced by 30 % (P < 0.05) in diabetic cultures compared with control cultures (Fig. 5).

Eight obese type 2 diabetic patients (49.5 ± 1.8 years) and eight young lean control subjects (26.9 ± 0.2 years) participated in the study.

To get further insight into the differences between diabetic and control cultures, we compared the absolute differences between the basal and the insulin - stimulated state (ΔFV0.1) in control cultures and in those established from type 2 diabetic subjects (Fig. 3C).