Sentences with phrase «in retinal»

Modern interest in the retinal art movement stems from 1965 when a major Op Art exhibition in New York, entitled «The Responsive Eye,» caught public attention.

[5] Harrison's writing reveals an interest in the retinal perception of color, and in tonal harmony; he believed that the term Impressionism was descriptive not merely of the recent movement in French painting, but referred to any work done «honestly and sincerely» before nature.

When looking through the Fujifilm FP - 100c instant color film datasheets, the similarities between the layering of materials in the film and the layering of cells in the retinal is striking.

A characteristic component of the phenotype associated with T4R mutation is the dose — response relationship that has been demonstrated between light exposure and the early alterations in retinal tissue that occur in affected animals.

Despite these limitations, the available tissues still provided valuable information about changes in retinal architecture in advanced disease which may contribute to a better understanding of the etiopathogenesis.

When the ophthalmologist views the retina with an instrument called an indirect ophthalmoscope, changes can be seen in the retinal blood vessel pattern, the optic nerve and the tapetum (the reflective portion of the eye that is responsible for «eyeshine»).

It plays a role in retinal health so the right food can save your dog from suffering certain diseases.

Kidney failure and resulting hypertension can result in retinal detachment and blindness.

Too much sorbitol trapped in retinal cells, the cells of the lens, and the Schwann cells that myelinate peripheral nerves can damage these cells, leading to retinopathy, cataracts and peripheral neuropathy, respectively.

In my view the primary benefit is to prevent damage to the DHA essential fat in your retinal pigmented epithelium.

(2005) Pressure - Induced Production of Inflammatory Cytokines and Activation of NF B Is Modulated by Calcium Channels in Retinal Glia (Investigative Ophthalmology & Visual Science, 46: ARVO E-Abstract 3579).

(2010) Dendritic Morphology Changes Prior to Uptake and Transport Dysfunction in Retinal Ganglion Cells of the DBA / 2J Mouse Model of Glaucoma (Investigative Ophthalmology & Visual Science, 51: ARVO E-Abstract 2141).

This is the first time that that this assay has been used to ascertain retinal function after cellular therapy in a retinal degenerate animal.

Scientists from Massachusetts Eye and Ear have identified a new transcription factor involved in retinal neovascularization: Runt - related transcription factor 1 (RUNX1).

(2008) Neurotrophin Receptor Expression in Retinal and Optic Nerve Microglia Changes With Glaucoma and Age in the DBA / 2J Mouse Model (Investigative Ophthalmology & Visual Science, 49: ARVO E-Abstract 3703).

The findings, reported in the Journal of Biomedical Optics, suggest that fluorocoxib A, or its derivatives, can be used to image COX - 2 expression in vivo as a biomarker for inflammation in retinal diseases.

Citation: Carr A-J, Vugler AA, Hikita ST, Lawrence JM, Gias C, Chen LL, et al. (2009) Protective Effects of Human iPS - Derived Retinal Pigment Epithelium Cell Transplantation in the Retinal Dystrophic Rat.

In retinal diseases including age - related macular degeneration (AMD) and diabetic retinopathy, the growth of new blood vessels damages the retina and causes blindness.

(2007) Chronic Minocycline Treatment Decreases Microglial Activation and Improves Retrograde Axonal Transport in Retinal Ganglion Cells in DBA / 2J Retina (Investigative Ophthalmology & Visual Science, 48: ARVO E-Abstract 3291).

Furthermore, we describe activity in retinal ganglion cell terminals and superficial inhibitory interneurons in the tectum during looming and propose a model for how temporal dynamics in tectal periventricular neurons might arise from com - putations between these two fundamental constitu - ents.

CFC investigators have also established a link between glial activation and oxidative damage in retinal ganglion cells.

Andrew Tsin, an expert in retinal neurobiology, «worked tirelessly, one - on - one, with research trainees in his lab, sustained over a period of more than 30 years.»

There was also a Special Interest Group on «The Role of Astrocytes and Other Glial Cells in Retinal Function and Glaucoma.»

Head of the therapeutics department and director of the research team «Inflammation, degeneration and vascular remodelling in retinal disease» at the Vision Institute in Paris.

Although the genes implicated in these disorders are involved in a variety of pathways, most of them share a molecular phenotype: high (and often transcript ‐ specific) expression in retinal tissue.

When tested in retinal electroporation studies, DsRed reporter expression was induced in ~ 76 % of GFP + cells, and 100 % of DsRed + cells also expressed GFP.

Among the newly funded investigations are: the development of animal models of diabetic eye disease to learn how fenofibrate, a drug clinically - approved to treat cholesterol, protects the eye from diabetes - related damage; the development of a specialized camera to capture how the retina's rods and cones (responsible for turning light energy into shapes and colors) work in health and how they fail in retinal diseases; and the exploration of mechanisms by which intestinal tract bacteria impact the health of the eye.

The CFC has delved into the nature of degenerative changes in retinal ganglion cells.

In glaucoma, EDN signaling increases in retinal cells before any damage occurs, and blocking it lessens cell death.

Then of course we don't know the outcome of the virtual reality stimulation studies yet, but we like to think that the study that we're now conducting and studies like it from people outside the CFC are raising the conversation and are raising the importance of neural activity in retinal ganglion cells as a way to both keep ganglion cells alive and perhaps even to bring back some that are really on the brink of dying.

Although participants in all three injection groups had a greater decrease in retinal thickness after one year than with laser treatment alone, patients who received triamcinolone injections had greater complication rates.

Dr. Vetter is committed to better understanding the role of microglia in retinal ganglion cell pathology in glaucoma.

My lab is currently moving our interest in retinal circuit formation in a new and related direction: how these circuits need to be protected over time to ensure proper visual function.

In the past, neural stem and progenitor cells from various sources were introduced into eyes with the thought that they might differentiate and replace photoreceptors lost in retinal disease [13]--[19].

CFC research findings have shown that there are many changes in the retinal ganglion cell akin to what is seen in other neurodegenerative diseases such as Alzheimer's.

The treatment could help people who have a fault in a gene called RPE65, which causes problems in the retinal pigment epithelium (RPE), a thin layer of cells that support and nourish photoreceptors.

In addition, they are developing a new type of imaging test with the goal of isolating changes in the retinal ganglion cells that are first affected in glaucoma.

His research focuses on understanding the role of inflammation in retinal degenerative diseases and seeks to target inflammatory pathways in these diseases using gene therapy.

The critical secondary endpoint of the change in retinal ganglion cell macular volume measured from baseline to week 48 demonstrated a statistically significant difference (p = 0.0189) between all GS010 - treated eyes and all sham - treated eyes, with untreated eyes losing 0.038 cubic mm of macular ganglion cell volume while treated eyes preserved their ganglion cell volume -LRB--0.003 cubic mm).

What's important is that the CFC has shown that there are many changes in the retinal ganglion cell akin to what is seen in other neurodegenerative diseases such as Alzheimer's.

Why that's important is that we showed that there are many changes in that retinal ganglion cell that are akin to what you see in other neurodegenerative diseases.

Dr. Nociari uses animal models, cell culture, and clinical approaches to study from different perspectives the impact that lipofuscin accumulation has in retinal disease.

With age, our eyes accumulate waste in retinal pigment epithelium (RPE), which supports the life and function of photoreceptors (light sensitive cells in the eye); in advanced stages, RPE and photoreceptors die.

Lead author of the study is physiology professor Julia Busik and she says: «Our study presents an unexpected finding that the connections between cells in the retinal blood vessels contain unusual, long - chain lipids that may keep vessels from leaking, possibly preventing diabetic retinopathy from occurring.»

Helena Edström More information: The study Platelet - derived growth factor over-expression in retinal progenitors results in abnormal retinal vessel formation is related to the Science For Life Laboratory, SciLifeLab, at Uppsala University and was carried out in collaboration between researchers at Uppsala University in Sweden and at the University of Murcia in Spain.

We discovered in the laboratory, our lab and others, that mitochondria — these are the little energy powerhouses inside the cells, inside all cells including retinal ganglion cells — they fragment and stop moving in retinal ganglion cell axons very early in glaucomatous insults.

And Alf Dubra is now using adaptive optics to image what we think are the same mitochondria moving in retinal ganglion cell axons.

Expressed in the retinal pigment epithelium, RPE65 plays a critical role in regenerating «11 - cis retinal,» the color - giving component (chromophore) in rods and cones, after it is bleached during light absorption.

Multiple Ca2 + - Dependent Mechanisms Regulate L - Type Ca2 + Current in Retinal Amacrine Cells.

Patch - clamp Capacitance Measurements and Ca < sup > 2 + Imaging at Single Nerve Terminals in Retinal Slices.