Sentences with phrase «in sodium restriction»

Why is there a difference in sodium restriction from patient to patient?

As we move forward in the sodium restrictions we have found that removing sodium from certain products give sub-par quality.

Not exact matches

«These are clinically significant findings, with this magnitude of blood pressure reduction being comparable to that expected with the addition of an anti-hypertensive medication and larger than effects usually seen with sodium restriction in people without CKD,» said McMahon.

The article is entitled «A Randomized Crossover Trial of Dietary Sodium Restriction in Stage 3 - 4 Chronic Kidney Disease.»

In 79 % of participants, dietary sodium was reduced during the restriction phase, and 65 % of patients reduced their intake by > 20 %.

9 A comment published in the Sao Paulo Medical Journal stated that «recent evidence from the Cochrane systematic review has cast doubt on the efficacy of dietary sodium restriction.»

Many holistic health experts and functional clinicians believe we may be seeing more people affected by thyroid deregulation due to sodium restriction advised by the media which has resulted in a reduction in iodine.

For example, KBs were recently reported to act as neuroprotective agents by raising ATP levels and reducing the production of reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseasein neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseasein the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseasein a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseaseIn addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseasein most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseasein the treatment of convulsion diseases.

Salt substitutes have little or no sodium in them and are typically used by people who have a medical restriction to salt.

The level of sodium restriction required in advanced heart failure is usually only found in veterinary prescription diets, however, many pets will not accept the palatability of foods this restricted.

While severe sodium restriction was historically recommended for pets, it is now known that in earlier stages of heart disease and heart failure, this approach may actually be detrimental.

In pets with mild heart disease (those that are not showing any symptoms), only mild sodium restriction is recommended.

No sodium restriction is necessary in pets with heart disease unless the condition has progressed to retention of fluid in the lungs (night cough) or in the abdomen (tummy).

Moderate sodium restriction is important in dogs with moderate heart disease, but severe heart disease requires very strict restriction.

Sodium restriction is key in management of Cushings Disease, as sodium levels will tend to be high in Cushings patSodium restriction is key in management of Cushings Disease, as sodium levels will tend to be high in Cushings patsodium levels will tend to be high in Cushings patients.

This could be a problem in patients with congestive heart failure or other patients who require sodium restriction May stunt growth if used in young, growing animals May lower seizure threshold and alter mood and behavior At high doses, this medication can cause birth defects early in pregnancy, be irritating to the stomach or cause higher than normal blood sugar levels If your pet has received high doses, it should not be vaccinated without your veterinarian's advice as the vaccine may not work or it may actually give your pet the disease you are trying to prevent Less common side effects include weight gain, insomnia, panting, diarrhea, vomiting, elevated liver enzymes, pancreatitis and behavior changes Serious side effects are not expected with routine use.

Robertson GL: Evaluation of the plasma vasopressin, plasma sodium, and urine osmolality response to water restriction in normal cats and a cats with diabetes insipidus.

In addition to protein restriction, renal diets contain additional potassium, lower sodium, low phosphorus and, in some cases, added Omega - 3 fatty acidIn addition to protein restriction, renal diets contain additional potassium, lower sodium, low phosphorus and, in some cases, added Omega - 3 fatty acidin some cases, added Omega - 3 fatty acids.