Not exact matches
Of those, eight
people from five families —
including two children — would be diagnosed
with symptoms that were
milder than those of almost all the American patients: nosebleeds, dizziness, headaches and insomnia.
The study
included 121
people from Japan and 252 from Australia, and both groups involved individuals
with normal brain function,
mild cognitive impairment and Alzheimer's.
A
person with mild cognitive impairment (MCI) has a slight but noticeable and measurable decline in cognitive abilities,
including memory and thinking skills.
A
person with a
mild reaction to the drug may break out in hives, but more severe signs can occur,
including swelling of the lips, tongue and throat, as well as asthma - like symptoms.
Hepatic encephalopathy occurs when the liver can not remove certain toxins and chemicals, such as ammonia, from the blood.1 These toxins and chemicals then build up and enter the brain.1 Hepatic encephalopathy is one of the major complications of cirrhosis (scarring of the liver), and a leading cause of hospital re-admission due to its recurrence, despite treatment.1 It can occur suddenly in
people with acute liver failure, but is seen more often in those
with chronic liver disease.1 Symptoms of hepatic encephalopathy
include mild confusion, forgetfulness, poor concentration and personality or mood changes, but can progress to extreme anxiety, seizures, severe confusion, jumbled and slurred speech and slow movement.1 The first step in treatment is to identify and treat any factors that cause hepatic encephalopathy.2 Once the episode has resolved, further treatment aims to reduce the production and absorption of toxins, such as ammonia.1 Generally, there are two types of medication used to reduce the likelihood of another hepatic encephalopathy episode — lactulose and rifaximin.2 However, it remains a leading cause of hospitalisations and re-hospitalisations in cirrhotic patients, despite the use of the above - mentioned standard of care treatment.
Hye and his colleagues analysed 26 proteins in blood from 1,148
people,
including 476
people with Alzheimer's, 220
with mild cognitive impairment and 452 elderly healthy controls.
The operationalization of
mild cognitive impairment (MCI) led to targeting earlier symptomatic cases of the illness and treatment strategies based less on pathology and more on a chance to halt or slow decline than there would be earlier in the disease.1
With the development of amyloid imaging, MCI due to AD diagnosis was refined, 2 and early - stage AD was extended further to
include preclinical AD, 3 wherein a positive amyloid positron emission tomography (PET) scan or diagnostic low levels of cerebrospinal fluid β - amyloid (Aβ) indicated the presence of pathology in
people who were cognitively normal.
Each group
included some
people with Alzheimer's disease, some
with mild cognitive impairment, and some
with no signs of mental deterioration.
But I can't help sharing that you also almost perfectly describe my (large and growing) chronic patient community as well Dr. Brogan, which
includes persons of all types usually
with hypermobility (often diagnosed as fibromyalgia, and occasionally but rarely
with Hypermobile Ehlers - Danlos Syndrome), depression, anxiety,
mild autistic traits (or related to
people on the spectrum), driven, Type A (for adrenergic, smile), perfectionistic, high achieving, driven, artistic, and creative who eventually succumb to secondary aotuimmune disease and all manner of issues from chronic inflammation.
In a
person with a
mild brain injury, the behavioral issues could be relatively minor,
including irritability, fatigue, and mood swings.
In the blogosphere, it generally represents a parenting approach that does not use punishment, however some
people use it to refer to parenting styles that still make use of rewards / incentives
with kids, and
milder forms of punishment
including time - outs and withdrawal of privileges.