Sentences with phrase «increase free fatty acids»

Adenovirus - induced hyperleptinemia rapidly depletes body fat in normal rats without increasing free fatty acids and ketogenesis, implying that fat - storing adipocytes are oxidizing the fat.

Having coffee and / or caffeine before workout can actually be very helpful at breaking down and increasing free fatty acids in your body.

• widespread effects on circulation, muscles and sugar metabolism • raised heart rate • increased heart output • increased rate and depth of breathing • increased metabolic rate • increased force of muscular contraction • delayed muscular fatigue • reduced blood flow to bladder (muscular walls relax and sphincters contract) • reduced blood flow to intestines • increased blood pressure • increased sugar (glucose) in the blood • increased break - down of glucose for energy *, especially in muscle cells • increased free fatty acids in the blood * • more oxidation of fatty acids to produce energy * • more ATP (the cells» primary energy compound) produced * • blood vessels constrict

Not exact matches

Severity of pancreatitis in obesity may be attributed to increased oxidative stress, free fatty acid production, fat necrosis and increased inflammation [8].

Mice fed on a high - fat diet showed increased levels of free fatty acids in their blood.

A reduction in TG levels also did not cause hepatic steatosis (Figure 3, A and B) or increase the production of ketone bodies (Figure 3C), and at most had only a modest effect on circulating nonesterified free fatty acid (NEFA) levels (Figure 3D).

Sometimes I do add some egg whites from a carton to my whole eggs to both save a little money and to increase the protein content without losing the nutrient benefits that you get from eating the yolks with the whites.Not to mention that the egg yolks from free range chickens are loaded with healthy omega - 3 fatty acids.

Also observed, was the growth hormone's actions of increasing fatty acid oxidation, as indicated by rises in free fatty acids and ketone bodies.

Interestingly, free fatty acids, which indicate available fats to burn during exercise, increase in children during exercise, indicating a very strong ability of children to mobilize fat stores for energy and possibly even use energy sources that have higher amounts of fat.

Cortisol is also released during high - intensity exercise to increase the production of glucose by the liver as well as free fatty acids into the bloodstream for energy production.

Omega - 3s as re-esterified triglycerides, natural triglycerides, and free fatty acids have somewhat higher bioavailability than ethyl esters, but consumption of all forms significantly increases plasma EPA and DHA levels [33,35].

The free fatty acids inhibit the oxidation of glucose for energy, creating insulin resistance, the condition that normally increases with aging, and that can lead to hyperglycemia and «diabetes.»

Basically, it will increase the amounts of «free fatty acids», which are utilized by the body to produce energy.

Swelling of the brain, resulting from increased vascular permeability and the entry of free fatty acids, reduces its circulation and oxygenation; lactic acidemia causes swelling of glial cells.

Smoke point depends on refinement and origin of the oil and it increases * with the decrease * of free fatty acids and the growth of refinement temperature.

Low intensity morning cardio potentially increases the amount of free fatty acids used up as fuel.

Growth hormone is a peptide stress hormone that tends to increase glucose, free fatty acids and insulin growth factor - 1 (IGF - 1).

After 1 — 2 d of the 5 % carbohydrate diet and persisting through the 7 - d period, serum β - hydroxybutyrate increased from 0.1 to 0.4 mmol / L and free fatty acids increased from 0.2 to 0.4 mmol / L.

These changes suggest that there is a shift from the use of glucose to the use of ketones and free fatty acids as metabolic fuels, and that glycogen formation increases from baseline.

I think it is most appropriate to borrow the words from someone far smarter and well versed on this topic here (Dr. Kevin Hall), «A logical consequence of the carbohydrate - insulin model is that decreasing the proportion of dietary carbohydrate to fat without altering protein or calories will reduce insulin secretion, increase fat mobilization from adipose tissue, and elevate oxidation of circulating free fatty acids.

These powerful emotions trigger adrenal hormone release — the physiological effects of which include activation of adipocyte lipase (resulting in mobilization of free fatty acids) and partial inhibition of protein synthesis, i.e., the plasma amino acids which are normally (readily) utilized by nonmalignant cells for protein synthesis are only partially used resulting in an increase in the availability of amino acids to meet tumor cell metabolic needs.

Lactate, which increases during starvation, can induce hepatic ketogenesis.2 Low - carbohydrate, fat - rich meals can enhance alpha - cell secretion of glucagon and lower insulin concentrations.3, 4 Plasma fatty acid concentrations can be twice as high during low - carbohydrate diets as compared with the usual carbohydrate intake in the postabsorptive period.5 Increased concentrations of free fatty acids in the absence of carbohydrate - induced inhibition of beta - oxidation of fatty acids and in the presence of an abnormally high ratio of glucagon to insulin and elevated concentrations of lactate may have caused ketoacidosis in our patient, who was trying to avoid all dietary carbohydrates.

While plasma glucose, insulin, and carbohydrate oxidation did not change significantly, plasma free fatty acid levels rose from 432 ± 31 to 848 ± 135 µEq / liter and were accompanied by significant increases in fat oxidation during the last hour of the test.

Increased amino acids to nourish and repair muscles, glucosamine for joint health and Omega 3 fatty acids also combine to help active dogs stay active and pain - free.

The therapeutic dotential of dietary precursor modulation by a fish - oil - supplemented diet (n - 3 fatty acids), such as eicosapentaenoic acid (C20: 5,n - 3) and docosahexaenoic acid (C22: 6,n - 3) in the therapy of ulcerative colitis has been shown to result in a 35 % to 50 % decrease in neutrophil production of LTB4.28 Significant improvement in symptoms and histologic appearance of the rectal mucosa has been observed in several small series of patients with Crohn's disease and ulcerative colitis given fish oil at 3 to 4 g daily for 2 to 6 months in uncontrolled studies.29 However, a larger, randomized, double - blind trial comprising 96 patients with ulcerative colitis failed to reveal any benefit in remission maintenance or treatment of relapse on 4.5 g of eicosapentaenoic acid daily, despite a significant reduction in LTB4 synthesis by blood peripheral polymorphonuclear cells.30 It should be emphasized, however, that the anti-inflammatory actions of the fish oils, in addition to inhibition of LTB4, include suppression of IL - 1 and platelet activating factor synthesis and scavenging of free oxygen radicals.30 The impact of increased lipid peroxidation after fish oil supplementation should be considered when altering the n - 6: n - 3 fatty acid ratio.31 Antioxidant supplementation may be able to counteract the potentially adverse effects of n - 3 fatty acids.