Sentences with phrase «increased oxidation of fatty acids»

Increased glucagon appears to contribute to development of severe hyperglycemia by mobilizing hepatic stores of glucose and to development of ketoacidosis by increasing the oxidation of fatty acids in the liver.

Not exact matches

Phosphorylation of both acetyl CoA carboxylase and AMP - activated protein kinase was increased, thus explaining the increase in fatty acid oxidation.

As a summary, based on numerous studies, if your goal is to increase mitochondrial density and as a result increase the rate of fatty acid oxidation or fat loss, focus your training program around strength training and HIIT, and eat a diet which is rich in protein and low - glycemic carbs.

Aside from studies on obese mice, limited research has also shown reduced blood pressure, increased fat burning and increased fatty acid oxidation later in the fast, and increased metabolic rate due to the release of epinephrine and norepinephrine (again, later in the fasting period).

More specifically, these acids have the ability to increase the rate of fat burning by stimulating the activity of enzymes in the liver involved in fat breakdown and oxidation and diminishing the activity of enzymes that promote fat storage, which leads to an improved fatty acid and glucose metabolism.

Also observed, was the growth hormone's actions of increasing fatty acid oxidation, as indicated by rises in free fatty acids and ketone bodies.

4Michelle Olson «Tabata Interval Exercise: Energy Expenditure and Post-Exercise Responses» Department of Exercise and Science, Auburn University,... continue One study found marked increases in fatty acid oxidation after just two weeks!

Among these, FGF21 increases insulin sensitivity and causes a corresponding decrease in basal insulin concentrations; FGF21 increases hepatic fatty acid oxidation, ketogenesis and gluconeogenesis; and, FGF21 sensitizes mice to torpor, a hibernation - like state of reduced body temperature and physical activity.

Furthermore, Cy -3-g substantially induced AMPK downstream target ACC phosphorylation and inactivation, and then decreased malonyl CoA contents, leading to stimulation of CPT - 1 expression and significant increase of fatty acid oxidation in HepG2 cells.

The oxidation of fatty acids increases further, directly through the release of adiponectin.

• widespread effects on circulation, muscles and sugar metabolism • raised heart rate • increased heart output • increased rate and depth of breathing • increased metabolic rate • increased force of muscular contraction • delayed muscular fatigue • reduced blood flow to bladder (muscular walls relax and sphincters contract) • reduced blood flow to intestines • increased blood pressure • increased sugar (glucose) in the blood • increased break - down of glucose for energy *, especially in muscle cells • increased free fatty acids in the blood * • more oxidation of fatty acids to produce energy * • more ATP (the cells» primary energy compound) produced * • blood vessels constrict

«Lipolysis» (the conversion of fatty acids) and «ketogenesis» (creation of ketone bodies) increase significantly due to fatty acid mobilization and oxidation.

The free fatty acids inhibit the oxidation of glucose for energy, creating insulin resistance, the condition that normally increases with aging, and that can lead to hyperglycemia and «diabetes.»

It thus even ensures the utilization of protein in better fashion, increased storage of glycogen and fatty acid oxidation in the cells.

-- Increase intake of Omega 3 fatty acids: Chlorella, ground flax seed, soaked in water chia seeds, good quality organic Omega 3 fish oil with antioxidant to prevent oxidation (HealthAid offers a very good quality molecularly distilled Omega 3 fish oil >).

I think it is most appropriate to borrow the words from someone far smarter and well versed on this topic here (Dr. Kevin Hall), «A logical consequence of the carbohydrate - insulin model is that decreasing the proportion of dietary carbohydrate to fat without altering protein or calories will reduce insulin secretion, increase fat mobilization from adipose tissue, and elevate oxidation of circulating free fatty acids.

To real important finding from this study was that an increase in fatty acid oxidation (one of the measurements that is considered a benefit of the ketogenic diet) doesn't necessarily result in more fat loss, in fact it may be the opposite.

This happens — more or less quickly depending on the individual — once beta - oxidation of fatty acids has increased to a point where fatty acids can't be utilized directly for adenosine triphosphate (ATP) production.

Lactate, which increases during starvation, can induce hepatic ketogenesis.2 Low - carbohydrate, fat - rich meals can enhance alpha - cell secretion of glucagon and lower insulin concentrations.3, 4 Plasma fatty acid concentrations can be twice as high during low - carbohydrate diets as compared with the usual carbohydrate intake in the postabsorptive period.5 Increased concentrations of free fatty acids in the absence of carbohydrate - induced inhibition of beta - oxidation of fatty acids and in the presence of an abnormally high ratio of glucagon to insulin and elevated concentrations of lactate may have caused ketoacidosis in our patient, who was trying to avoid all dietary carbohydrates.

While plasma glucose, insulin, and carbohydrate oxidation did not change significantly, plasma free fatty acid levels rose from 432 ± 31 to 848 ± 135 µEq / liter and were accompanied by significant increases in fat oxidation during the last hour of the test.