The surge in pupil numbers is due to many different factors, including the impact of the baby boom,
increasing birth rate levels and immigration.
Not exact matches
The cultural changes that Fuchs and Reklis have in mind are
increasing individualism, growing preoccupation with individual fulfillment, wider tolerance for divorce as a solution to marital problems, and more general acceptance at all social
levels of the high
rates of out - of - wedlock
births and single parenthood.
In poor countries the
birth rate is soaring, while consumption has
leveled off (which means per capita consumption is going down); in rich countries the
birth rate has
leveled off, while consumption is
increasing.
Despite a 56 % decrease in the national incidence of sudden infant death syndrome (SIDS) from 1.2 deaths per 1000 live
births in 19921 to 0.53 death per 1000 live
births in 2003,2 SIDS continues to be the leading cause of postneonatal mortality in the United States.3 The decreased
rate of SIDS is largely attributed to the
increased use of the supine sleep position after the introduction of the «Back to Sleep» campaign in 1994.4 - 7 More recently, it has been suggested that the decrease in the SIDS
rate has
leveled off coincident with a plateau in the uptake of the supine sleep position.8 Although caretakers should continue to be encouraged to place infants on their backs to sleep, other potentially modifiable risk factors in the sleep environment should be examined to promote further decline in the
rate of SIDS.
In animal models, exposure to cigarette smoke or nicotine during fetal development alters the expression of the nicotinic acetylcholine receptor in areas of the brainstem important for autonomic function, 28 alters the neuronal excitability of neurons in the nucleus tractus solitarius (a brainstem region important for sensory integration), 29 and alters fetal autonomic activity and medullary neurotransmitter receptors.30 In human infants, there are strong associations between nicotinic acetylcholine receptor and serotonin receptors in the brainstem during development.31 Prenatal exposure to tobacco smoke attenuates recovery from hypoxia in preterm infants, 32 decreases heart
rate variability in preterm33 and term34 infants, and abolishes the normal relationship between heart
rate and gestational age at
birth.33 Moreover, infants of smoking mothers exhibit impaired arousal patterns to trigeminal stimulation in proportion to urinary cotinine
levels.35 It is important to note also that prenatal exposure to tobacco smoke alters the normal programming of cardiovascular reflexes such that there is a greater - than - expected
increase in blood pressure and heart
rate in response to breathing 4 % carbon dioxide or a 60 ° head - up tilt.36 These changes in autonomic function, arousal, and cardiovascular reflexes might all
increase an infant's vulnerability to SIDS.
The prenatal phase of the program reduced fetal exposure to tobacco, improved the qualities of women's prenatal diets, reduced
rates of pyelonephritis, improved
levels of informal social support, and reduced intellectual impairment and irritable behavioral styles associated with fetal exposure to tobacco.6, 10,11,22 Prenatal exposure to tobacco is a risk factor for early behavioral dysregulation, problems with attention, and later crime and delinquency.22 Moreover, the combination of
birth complications (and, by implication, neurological impairment) and rejecting parenting substantially
increases the likelihood of violent offenses by the time children are 18 years old.5