Not exact matches
A
host can often be
infected with more than one type of virus and, as viruses replicate in the
host's
cells, the genetic segments of the progeny viruses can be shuffled into
new combinations.
The prM cleavage process is important for
new viral particles to be able to
infect a
host cell.
New research out of Duke University shows that RNA viruses are littered with N6 - methyladenosine tags which affect the ability of these viruses to
infect cells and, ultimately, their human
hosts.
The phages infiltrate bacterial
cells, where they commandeer the
host machinery to make thousands of
new phages; then they escape through the bacterial
cell wall — killing the
host — and spread to
infect their next victims.
«Bacterial and
host cell proteins interact to regulate Chlamydia's «exit strategy»:
New research suggests major role for calcium ion signaling in release of Chlamydia from
infected cells.»
An international study led by researchers at Monash University» Biomedicine Discovery Institute (BDI) has shone light on the way the Hepatitis C Virus (HCV) hijacks the communication systems in the
host cells it
infects, uncovering potential
new therapeutic targets for the disease.
«Nathanael sent us his
new molecule, and we put it in our
host cells,
infected them with HCV and found that while the
cells were fine, they didn't support virus replication anymore,» Dr Reza Haqshenas said.
Groundbreaking discovery challenges the conventional theory that
infected host cells control latency and could open the door to
new pathways
Soon the
host cell becomes a virus factory, replicating thousands of
new viral organisms to go forward and
infect new cells.
Inhibiting neuraminidase effectively locks the
new viral organisms within their
host cell, imprisoning them so that they can not
infect new cells.
With more CD4 +
cells at the scene of the infection, there are more
cells for HIV to target, increasing its chance of successfully
infecting a
new host.