Experiments in Muc1 knockout mice and mice with Muc1 knockdown have shown that under Helicobacter infection, mice deficient in Muc1 develop far more cancer - promoting
inflammation than normal mice.
Not exact matches
When the researchers examined
mice with disrupted IL - 27 function, they found that they were more likely
than normal mice likely to die when infected with the virus, and that they died as a consequence of rampant lung
inflammation.
Expression of CXCL16 was higher in the colon and lung tissue of GF
mice than in
normal mice, and blocking that expression reduced the numbers of iNKT cells and the amount of
inflammation in those tissues.