It works by activating
the inflammatory nuclear factor kappa B (NF - kB for short), which binds to your DNA and triggers a number of inflammatory cascades throughout the body.
One particularly bad guy in high amounts is
the inflammatory nuclear factor kappa B, or NF - kB for short.
Not exact matches
Over-activation of the
inflammatory protein
nuclear factor κB (NF - κB) in the brain region leads to a number of aging - related changes in mice, from cognitive decline to muscle weakness.
Nuclear factor kappa - B mediates selective induction of neuronal nitric oxide synthase in astrocytes during low - level
inflammatory stimulation with MPTP.
The Nurr1 Activator 1, 1 - Bis (3 ′ - Indolyl)-1 -(p - Chlorophenyl) Methane Blocks
Inflammatory Gene Expression in BV - 2 Microglial Cells by Inhibiting
Nuclear Factor κB.
Low - dose 1 - methyl -4-phenyl-1, 2, 3, 6 - tetrahydropyridine causes
inflammatory activation of astrocytes in
nuclear factor - $ ąppa $ B reporter mice prior to loss of dopaminergic neurons.
(2,3) Statin drugs can help with all this inflammation by down regulating
Nuclear Factor Kappa Beta (NF - κ B), the master controller of the
inflammatory response.
In addition, LA promoted
inflammatory processes activating the
nuclear factor - κB transcription
factor as well as stimulating the expression of cyclooxygenase - 2 and pro-
inflammatory cytokines.
In a recent study of 10 lean healthy individuals, both
nuclear transcription
factor - κB activation and nitrotyrosine were acutely increased ≤ 3-fold after a high GI meal by comparison with a low GI meal, suggesting that high - normal physiologic increases in blood glucose after meals could aggravate
inflammatory processes (6).
In addition to the metabolic activities described above, food flavonoids have also been shown to suppress
inflammatory signaling in another metabolic pathway called the
nuclear factor kappa - B (NF - kB) pathway.