The phrase
"influenza infection" refers to a condition where a person gets sick due to a specific kind of virus called influenza. It's a common illness that causes symptoms like fever, cough, sore throat, and body aches.
Full definition
Researchers in my lab think that this impact
of influenza infection on muscles is another unintended consequence of the immune response to the virus.
Seeing
influenza infections in pigs is very common, and the transfer of influenza virus from humans to pigs is known to occur.
We found no association between ASD risk and
influenza infection during pregnancy or influenza vaccination during the second to third trimester of pregnancy.
This is important, since a decrease in leg stability and strength could result in older folks being more prone to falls during recovery
from influenza infection.
Even influenza infection increases your susceptibility to disease (most commonly pneumonia, but infection overall), and it's worse in the elderly.
Researchers have discovered an inherited genetic variation that may help identify patients at elevated risk for severe, potentially
fatal influenza infections.
Whether signs are shown or not,
equine influenza infection can result in days or weeks off work and the virus rapidly spreads to other horses by nasal aerosol (coughing).
A study led by St. Jude Children's Research Hospital found that obese mice are not protected against
influenza infections by vaccines that include adjuvants, raising concerns about vaccine effectiveness in obese humans who are known to be at an increased risk for severe flu.
Avian influenza infections in cats appear to be associated with outbreaks in domestic or wild birds by way of eating raw meat infected with the virus.
«The clinical implications of this work are that we might be able use existing drugs that alter Hedgehog signaling to
treat influenza infection,» said Bier.
I am a researcher who specializes in immunology at the University of Connecticut School of Medicine, and my laboratory focuses on
how influenza infection affects the body and how our bodies combat the virus.
Most
influenza infections affect the respiratory system, but there have long been suspicions that they are also linked to neurological disorders.
Examining 37 years of data on hospital admissions for appendicitis and the flu, he and his colleagues have found that appendicitis rates vary roughly in parallel
with influenza infections.
«Most healthy people who
develop influenza infection do just fine by getting plenty of fluids and rest and do not need an antiviral medication unless they have severe infection,» says Pritish Tosh, MD, an infectious diseases physician and researcher at the Mayo Clinic in Rochester, Minnesota.
To make their discovery, Dr. Prantner and his colleagues used isolated mouse and human macrophages in vitro, and a
mouse influenza infection model.
In order to properly
combat influenza infection, the cytokines and chemokines produced by the innate immune cells in the lungs become systemic - that is, they enter the bloodstream, and contribute to these systemic symptoms.
Our study in an animal model found that
influenza infection leads to an increase in the expression of muscle - degrading genes and a decrease in expression of muscle - building genes in skeletal muscles in the legs.
In another study published this week the group investigated influenza pH1N1 virus - host cell interaction in detail and found that certain host function could be temporally inhibited with small molecule MK2206 to
block influenza infection in cell culture.
Heaton et al. now report that a special type of epithelial cell in the lungs of mice — called club cells —
survive influenza infection.
In one study, Dartmouth's Hamilton found that arsenic exposure at 10 ppb compromised the immune systems of mice so much that they could not fend off an
ordinary influenza infection.
As a natural host of influenza A viruses (including H5N1), the duck is known to often remain asymptomatic
under influenza infection.
In mice with a lethal
Haemophilus influenzae infection, the researchers found glucocorticoids upregulated IRAK - M expression by enhancing the protein NF - κB p65 and glucocorticoid receptor binding to the IRAK - M promoter.