Insulin opens your cells enabling your cells to use sugar from your bloodstream, moving it into your cells.
Because it's
the insulin opening the cell up.
Not exact matches
Insulin can
open up a
cell's outer membrane like a key in a door lock and set in motion the machinery that feeds the
cell glucose, which is converted to energy.
In these
cells, glucose metabolism inhibits the ATP - dependent potassium channel (KATP channel) and
opens voltage - dependent calcium channels (VDCCs), resulting in the exocytosis of
insulin - containing granules [41].
The glucose, like all of the nutrients, soon gets absorbed into the bloodstream creating a peak in what we call «blood sugar levels», which results with the releasing of more
insulin from the pancreas in order to push glucose to the
cells, basically «commanding» the
cells to
open up and absorb it, where it gets used as an energy source.
Insulin helps regulate your blood sugar levels by attaching to either fat
cells, nerve
cells (or neurons), or muscle
cells and ordering them to
open up and let the glucose in.
Once
insulin interacts with the cellular door it
opens and the sugar is able to enter the
cell.
If there's no calories, remember
insulin's the door that
opens up the
cell for the calories to go in.
Our
cells and our body, knows this; so the little doors - receptors on the
cells that happily
open for NORMAL amounts of
insulin, close and say NO WAY to excessive amounts of
insulin; they're self - preservating.
Healing the
cell so that the doors / receptors
open normally to
insulin and all our other hormones, is the next step.
Insulin is a messenger which circulates to all the
cells of the body giving them the message to
open trillions of tiny doors in the
cells, saying «let the sugar in, please».
Secondary messengers acts to repair the doorbell so that the
cell doors
open in response to glucose, resulting in less
insulin needing to be secreted.
Insulin resistance happens when the cells essentially don't open the door when insulin comes kn
Insulin resistance happens when the
cells essentially don't
open the door when
insulin comes kn
insulin comes knocking.
Meanwhile,
insulin also works on fat
cells similar to how it works on muscle
cells, signaling the gates to
open and nutrient storage to commence.
Once the
insulin molecule docks onto the receptor, it signals the muscle
cell to
open up gates.
Secondary messengers acts to repair the doorbell so that the
cell doors
open in a timely response to glucose, resulting in less
insulin needing to be secreted.
You can imagine
insulin as being an automatic gate that
opens whenever fat, glucose, and amino acids arrive to get into your
cells.
Unable to
open the door to the
cells,
insulin lets sugar build up in the blood.
Well when you think of
insulin as some annoying dude who continually knocks at the door, and your
insulin receptors attached to
cells are the guy inside who
opens the door..
Chromium is a part of so called Glucose Tolerance Factor (GTF) and as such it helps
insulin to attach to the
insulin receptors and be more effective in
opening the
cell's door for sugar, thus lowering its blood levels.
They become resistant because there is too much
insulin «knocking on the
cell door» telling it to
open up and let the sugar in.
Some research points to excessive fat in the body, blood, and diet as a contributor to
insulin resistance by preventing it from doing its job, i.e.,
opening the pores on your
cell membranes to allow sugar to pass into them.
They describe
insulin as a «glucose doorman» that travels around the body
opening cell doors so glucose can enter and do its job, which is maintaining proper blood - sugar levels.
One way to think about it is that the
cells become «resistant» to
insulin trying to
open them up and get glucose in.
Without the help of
insulin to
open the
cell doors, the glucose molecule is just too big to enter the various
cells of the body.
It makes sense from the perspective if the
insulin's role was to
open the
cells to accept glucose and, in the diabetic or someone with
insulin resistance, that was not happening, the
cell would die, would it not?
Magnesium has been found to regulate and improve blood sugar control, play a vital role in the secretion and function of
insulin, is necessary for
insulin to
open cell membranes for glucose and helps the body digest, absorb, and utilize proteins, fats, and carbohydrates.
Insulin attaches to receptors on the surface of
cells and
opens «pores» in the
cell wall that allow glucose molecules to leave the bloodstream and enter the
cell's interior.
Without an adequate amount of
insulin to «
open the door,» glucose is unable to get into the
cells, so it accumulates in the blood, setting in motion a series of events that result in diabetes mellitus.
The role of
insulin is much like that of a gatekeeper: It stands at the surface of body
cells and
opens the door, allowing glucose to leave the blood stream and pass inside the
cells.