There are two other
genes, on different chromosomes, that
code for different forms of the enzyme, and the team is now
investigating whether mutations in these
genes might be responsible for the other cases of ALS.
We further
investigated the source of the conflict in the protein -
coding genes (SM11) and found that trees using all codon positions from the 10 % most compositionally homogeneous (low - variance) exons (n = 830) were most congruent with the c12 tree and, thus, more similar to the TENT than to the c123 tree (Figs. 2 and 6A; cladograms in fig.