Not exact matches
In 2010, researchers from the University of Michigan Comprehensive Cancer Center published a study in the journal Clinical Cancer Research showing that sulforaphane had the ability to kill breast cancer stem cells in mice and in lab cultures, and it also prevented the growth of new tumor cell
In 2010, researchers from the University of Michigan Comprehensive Cancer Center published a study
in the journal Clinical Cancer Research showing that sulforaphane had the ability to kill breast cancer stem cells in mice and in lab cultures, and it also prevented the growth of new tumor cell
in the journal Clinical Cancer Research showing that sulforaphane had the ability to
kill breast cancer stem cells
in mice and in lab cultures, and it also prevented the growth of new tumor cell
in mice and
in lab cultures, and it also prevented the growth of new tumor cell
in lab cultures, and it also prevented the growth of new
tumor cells.
In human cells and in mice, the virus infected and killed the stem cells that become a glioblastoma, an aggressive brain tumor, but left healthy brain cells alon
In human cells and
in mice, the virus infected and killed the stem cells that become a glioblastoma, an aggressive brain tumor, but left healthy brain cells alon
in mice, the virus infected and
killed the stem cells that become a glioblastoma, an aggressive brain
tumor, but left healthy brain cells alone.
In the Cell study, Dr. Massagué, with Fellow Manuel Valiente, PhD, and other team members, found that in mouse models of breast and lung cancer — two tumor types that often spread to the brain — many cancer cells that enter the brain are killed by astrocyte
In the Cell study, Dr. Massagué, with Fellow Manuel Valiente, PhD, and other team members, found that
in mouse models of breast and lung cancer — two tumor types that often spread to the brain — many cancer cells that enter the brain are killed by astrocyte
in mouse models of breast and lung cancer — two
tumor types that often spread to the brain — many cancer cells that enter the brain are
killed by astrocytes.
The researchers also used their ultrasound technique
in mice to image Salmonella bacteria, which could be used to deliver cancer -
killing drugs to
tumor cells.
One form of pancreatic cancer has a new enemy: a two - drug combination discovered by UF Health researchers that inhibits
tumors and
kills cancer cells
in mouse models.
Dr. Cripe and his colleagues at The Ohio State University, the University of Pittsburgh School of Medicine and Cincinnati Children's Hospital Medical Center tested how well the oncolytic viral therapy — a cancer -
killing form of the herpes simplex virus, called oHSV — infected and
killed tumor cells
in mice with and without a healthy immune system.
Much of the cancer -
killing effect of the oHSV injections was lost when the researchers tested the therapy
in tumors of
mice with defective immune systems.
Increasing expression of a chemical cytokine called LIGHT
in mice with colon cancer activated the immune system's natural cancer -
killing T - cells and caused primary
tumors and metastatic
tumors in the liver to shrink.
In experiments with cancer cell lines, the PIM1 inhibitors killed cells in a MYC - dependent manner, and in two different mouse models — one in which mice were implanted with patient tumors and the other in which a genetic alteration of MYC predisposes the mice to tumor development — the administration of PIM1 inhibitors resulted in significant tumor regressio
In experiments with cancer cell lines, the PIM1 inhibitors
killed cells
in a MYC - dependent manner, and in two different mouse models — one in which mice were implanted with patient tumors and the other in which a genetic alteration of MYC predisposes the mice to tumor development — the administration of PIM1 inhibitors resulted in significant tumor regressio
in a MYC - dependent manner, and
in two different mouse models — one in which mice were implanted with patient tumors and the other in which a genetic alteration of MYC predisposes the mice to tumor development — the administration of PIM1 inhibitors resulted in significant tumor regressio
in two different
mouse models — one
in which mice were implanted with patient tumors and the other in which a genetic alteration of MYC predisposes the mice to tumor development — the administration of PIM1 inhibitors resulted in significant tumor regressio
in which
mice were implanted with patient
tumors and the other
in which a genetic alteration of MYC predisposes the mice to tumor development — the administration of PIM1 inhibitors resulted in significant tumor regressio
in which a genetic alteration of MYC predisposes the
mice to
tumor development — the administration of PIM1 inhibitors resulted
in significant tumor regressio
in significant
tumor regression.
In a four - year study conducted on the mouse model in advanced breast cancer metastasis in the eye's anterior chamber, Petty and colleagues found that the new nanoparticle not only killed tumor cells in the eye, but also extended the survival of experimental mice bearing 4T1 tumors, a cell line that is extremely difficult to kil
In a four - year study conducted on the
mouse model
in advanced breast cancer metastasis in the eye's anterior chamber, Petty and colleagues found that the new nanoparticle not only killed tumor cells in the eye, but also extended the survival of experimental mice bearing 4T1 tumors, a cell line that is extremely difficult to kil
in advanced breast cancer metastasis
in the eye's anterior chamber, Petty and colleagues found that the new nanoparticle not only killed tumor cells in the eye, but also extended the survival of experimental mice bearing 4T1 tumors, a cell line that is extremely difficult to kil
in the eye's anterior chamber, Petty and colleagues found that the new nanoparticle not only
killed tumor cells
in the eye, but also extended the survival of experimental mice bearing 4T1 tumors, a cell line that is extremely difficult to kil
in the eye, but also extended the survival of experimental
mice bearing 4T1
tumors, a cell line that is extremely difficult to
kill.
Meng and Nel also collaborated with Dr. Timothy Donahue, chief of gastrointestinal and pancreatic surgery and a Jonsson Comprehensive Cancer Center member, to demonstrate that treatment with the iRGD peptide can enhance
tumor cell
killing for patient - derived pancreatic cancers, growing subcutaneously
in a
mouse model.
The treatment
killed cancerous cells
in the
mice, shrunk their
tumors and left them with fewer side effects.
Researchers demonstrated that the drugs pemetrexed and gemcitabine
killed cells from
mouse and human brain
tumors, called group 3 medulloblastoma, growing
in the laboratory.
Furthermore, when the team, lead by cell biologist Valeria Fantin, implanted LDH - A-normal cancer cells and LDH - A-deficient cancer cells
in mice, LDH - A-deficient
tumors grew more slowly and took two - and - a-half times longer, on average, to
kill the
mice than LDA - A-positive
tumors.
So
in the
mice we found that each of the CAR T cells
killed about 40
tumor cells, and as I said
in humans they
killed more than 1,000
tumor cells.
In mice, the study found this helped kill tumors hiding elsewhere in the body — not just at the site of the injectio
In mice, the study found this helped
kill tumors hiding elsewhere
in the body — not just at the site of the injectio
in the body — not just at the site of the injection.
A virus not known to cause disease
kills triple negative breast cancer cells and
killed tumors grown from these cells
in mice, according to Penn State College of Medicine researchers.
The
tumors in the active
mice contained more of two types of cancer -
killing cells, explains Quartz, «double the number of T - cells and five times the number of natural killer cells.»