Sentences with phrase «lacrimal glands»

Causes of KCS can include feline Herpes virus, canine distemper, Sulfa - based antibiotic use, trauma to the lacrimal glands, and cherry - eye surgery.

Each eye of a dog actually has 2 tear glands (also called lacrimal glands), unlike people (who have one).

In the majority of KCS cases, an underlying etiology is not identified and in these cases it is believed an immune - mediated phenomenon creates inflammation and destruction of the lacrimal glands.

Additional therapeutic effects may be achieved via CsA and tacrolimus» anti-inflammatory effects, stimulatory effect on lacrimal glands, proliferative effect on mucin - producing conjunctival goblet cells, and inhibitory effect on lacrimal cell apoptosis.

The disease is usually caused by inflammation of the lacrimal glands, but can also be caused by the toxic effect of certain drugs on the lacrimal glands or by lack of nerve innervation to the lacrimal glands.

Dry eye can be caused by a variety of causes, but the most common cause is an autoimmune destruction of the lacrimal glands around the eye.

Other sources of dry eye include drug toxicity (antibiotics such as sulphadiazine and sulphasalazine can cause temporary or permanent dry eye in some animals), drug - induced reaction (atropine and topical anesthetics temporarily reduce tear production), neurological impairment (damage to the nerves leading to the lacrimal glands), removal of the third eyelid (see Cherry Eye), systemic disease (e.g. distemper), chronic conjunctivitis, trauma to the tear glands, hypothyroidism, congenital disease (some dogs are born without lacrimal glands), breed predisposition.

Your dog's lacrimal glands produce the tears necessary to keep his eyes in healthy condition.

Until the lacrimal glands start to work again, tear replacement with drops that keep the eyes hydrated and lubricated is an important initial part of treatment for KCS as well.

Tears are produced in specialized glands called lacrimal glands.

I am wondering if anyone has had any experience with Sjögren's syndrome, which is an autoimmune disease affecting primary the salivary and lacrimal glands (i am pretty sure that many of those suffering from dry eyes might actually have Sjögren's disease).

With these cells in hand, the researchers injected them into the lacrimal glands of mouse models of Sjogren's syndrome, an autoimmune disease that results in ADDE, dry mouth and other symptoms.

The Scripps Research Institute team found «progenitor» cells isolated from healthy lacrimal glands can improve the appearance and function of diseased tissue.

A new study in animal models, led by scientists at The Scripps Research Institute (TSRI), suggests that the eye's lacrimal glands can be repaired by injecting a kind of regenerative «progenitor» cell.

«The evaporated tear film on the front of the eye can't be replenished by simply supplying more new tear fluid from the lacrimal gland,» Braun noted.

The stem of the T cartilage is surrounded by an accessory lacrimal gland, which produces a substantial portion of the tear film.

If the lacrimal gland is injured or damaged by aging, pollution or even certain pharmaceutical drugs, a person can experience a debilitating condition called aqueous deficiency dry eye (ADDE)-- sometimes called «painful blindness.»

Further tests suggested that epithelial cell progenitors helped by restoring the connection between cells called myoepithelial contractile cells and the lacrimal gland's secretory cells, which produce tears.

«This is the first step in developing future therapies for the lacrimal gland,» said TSRI biologist Helen Makarenkova, who led the study.

If injured, a healthy lacrimal gland naturally regenerates itself in about seven days.

In this study, the researchers used progenitor cells that were poised to become epithelial tissue, a key component of the lacrimal gland.

The eye's lacrimal gland is small but mighty.

In the new study, Makarenkova and her colleagues looked at whether they could kick start regeneration by injecting progenitor cells into the lobes that make up the lacrimal gland.

In addition to Makarenkova and Gromova, authors of the study, «Lacrimal Gland Repair Using Progenitor Cells,» were Dmitry A. Voronov of TSRI, the Russian Academy of Sciences and the A.N. Belozersky Institute of Physico - Chemical Biology of the Lomonosov Moscow State University; Miya Yoshida and Suharika Thotakura of TSRI; Robyn Meech of Flinders University; and Darlene A. Dartt of the Schepens Eye Research Institute / Massachusetts Eye and Ear, Harvard Medical School.

Garg A, Bansal M, Gotoh N, Feng G - S, Zhong J, Wang F, Kariminejad A, Brooks S, Zhang X. Alx4 relays sequential FGF signaling to induce lacrimal gland morphogenesis.

In most cases, the lacrimal gland will begin adequate tear production.

Other causes include dysfunction of the nerve that runs to the lacrimal gland (neurogenic KCS) and the rare side - effects of certain medications.

In most dogs, this can be done by administering eye drops that suppress the immune - mediated infiltration of the lacrimal gland.

In dogs with neurogenic KCS, the lacrimal gland will resume tear production only if the underlying neurologic problem can be treated.

The most common cause of inadequate tear secretion is the infiltration of inflammatory cells from the immune system into the cells of the lacrimal gland.

KCS can also be a congenital condition in dogs when they are born with poor lacrimal gland function.

In immune - mediated lacrimal disease, the balance between T - suppressor and T - helper cells plays an important role in lacrimal gland regulation.

The orbital lacrimal gland produces 60 % of the tears for the eye, and the third eyelid gland produces 40 % of the tears.

The lacrimal gland of the third eyelid is held in place by tissue fibers but some individuals have weaker fibers than they should so the gland protrudes.

The lacrimal gland of the third eyelid is held in place by tissue fibers but some individuals have weaker fibers than they should and the gland protrudes.

It is a common eye condition resulting from inadequate production of the aqueous portion of the tear film by the lacrimal gland and / or gland of the third eyelid gland.

The gland is a tear producing gland, and produces about 30 % of the tears, while the main orbital lacrimal gland produces the rest.

It is thought that should the main orbital lacrimal gland be damaged later in life that there is no «backup» for tear production.

Not exact matches

BioTears Oral GelCaps are designed to help inhibit the LASIK induced dry eye by supporting post-surgical wound healing response, enhancing lacrimal and meibomian gland output, as well as enhance the post-op acetylcholine neurotransmitter blink response.

Here we sought to quantify T and B lymphocyte populations in the lacrimal tissue of the nictitans glands of dogs with iKCS those with neurological KCS (nKCS) and also in dogs with tear production within the recognized normal levels and no ocular surface signs of KCS.

The orbit is the bony «socket» that contains the eyeball and associated structures like the lacrimal (tear producing) gland, nerves, blood vessels, and extraocular muscles.

The condition is caused when the tear (lacrimal) gland in the third eyelid moves out of position.

The oil comes from glands lining the outer eyelids, the mucus comes from glands in the conjunctiva (the pink part inside the eyelids), and the water comes from tear (or lacrimal) glands.

Dogs have two tear - producing (lacrimal) glands in each eye.