1) A genetic Condition called Familial Hypercholesterolemia which affects 1 in 500 people, where a person's liver expresses much
less LDL receptors and is therefore unable to clear «used» cholesterol in LDL particles from the blood
Obviously, the less total fat in the diet,
the less LDL - raising saturated fat there will be in the body.
Since CVD is an inflammatory process, I assume
less LDL in one's artery walls would equate to a reduced need for WBCs.
Mitchell L. Jones, M.D., the head researcher of the probiotic study said that the probiotics «broke up the bile salts, leading to reduced cholesterol absorption in the gut and
less LDL cholesterol in your blood» (the bad cholesterol).
When the cholesterol level in the liver goes up, it expresses
less LDL - receptor for the simple reason that it does not need any more cholesterol.
Not exact matches
Does this mean refined coconut oils are
less healthy and lead to raised
LDL cholesterol levels?
The ability to boost HDL makes it «
less bad,» yet it's not the best choice of fat overall for heart health, as any fat that raises
LDL should be limited.
Ideally,
LDL cholesterol («bad cholesterol») levels should be
less than 100 mg / dL.
Compared to
LDLs treated with the oxidant alone, those mixed with a beverage experienced
less oxidation.
The researchers compared measurements of the three key factors to American Diabetes Association guidelines that were in effect at the time — blood pressure under 130/80 mmHg,
LDL (or bad) cholesterol
less than 100 mg / dL and blood HbA1c (glycated hemoglobin) under 7 percent.
The liver and, to a
lesser extent, the intestine produce apolipoprotein C - III (ApoC - III), an 8.8 - kDa glycoprotein associated with TRLs,
LDL, and high - density lipoproteins (HDLs)(4).
A study from an international research team finds that familial hypercholesterolemia — a genetic condition that causes greatly elevated levels of
LDL cholesterol throughout life — accounts for
less than 2 percent of severely elevated
LDL in the general population but also increases the risk of coronary artery disease significantly more than does elevated
LDL alone.
In general, decreasing your total cholesterol levels is
less important for optimizing your health than improving the ratio of HDL to
LDL.
So can a genetic predisposition and even menopause: As estrogen levels drop, we make
less HDL, and more
LDL and triglycerides.
On top of that, there is the «good» (HDL) and «bad» (
LDL) kind of cholesterol — according to the American Heart Association, the first one contributes to the production of thick, hard deposits that can clog arteries and make them
less flexible, increasing the risk of heart attacks and other cardiovascular diseases.
The two types of cholesterol are low - density lipoprotein (
LDL), or the bad cholesterol because it contributes to plaque, a hard deposit that can clog arteries, making them
less flexible, and high - density lipoprotein (HDL), the good cholesterol which is able to reverse the effects of
LDL cholesterol in the body.
For people with heart disease,
LDL should be
less than 100 mg / dL and possibly even
less than 70 mg / dL for those at super-high risk.
LDL is a cholesterol - carrying protein that can deposit cholesterol in your blood vessels, so the
less you have of it, the better.
While the fat content in food is now thought to be a more important player in determining the cholesterol level in your body, you should still limit your dietary cholesterol to
less than 300 mg / day if you are healthy, and
less than 200 mg / day (the amount found in one egg yolk) if you have cardiovascular disease, diabetes, or elevated
LDL cholesterol.
In one study, people who took a fermented papaya supplement for 14 weeks had
less inflammation and a better
LDL: HDL ratio than people who were given a placebo.
The high - fat group also had the most improvements in cholesterol, including lower triglycerides, lower
LDL, and lower levels of PIA - 1, which shows
less likelihood of having blood clots or inflammation.
Called «bad» cholesterol,
LDL can build up in the arteries, forming plaque that makes them narrow and
less flexible, a condition called atherosclerosis.
Although both are the fats of whole milk, ghee (clarified butter) has a different fat content, which actually lowers
LDL («bad») cholesterol and promotes heart health when consumed at moderate levels (
less than 2 tablespoons per day).
I read that a low rate of TG / HDL may indicate that the
LDL particles are of the bigger, fluffier type
less dangerous.
People with large, buoyant
LDL are said to have Pattern A, which is typically considered
less artherogenic, meaning
less likely to cause plaque buildup in the arteries.
The levels of
LDL will decrease which will make the body
less prone to suffer fro Type2 Diabetes.
Even more astounding, 50 percent of patients had
LDL less than 100 mg / dL — considered optimal levels!
1) Niacin — increases HDL, reduces small
LDL, and reduces Lp (a) 2) Elimination of wheat, cornstarch, and sugars — Best for reducing small
LDL;
less potent for Lp (a) reduction.
Getting your
LDL total down to around 70 mg / dl or
less is definitely achievable for many people on a high - carbohydrate, whole foods plant diet.
Even though an
LDL level of 100mg / dl or
less is considered optimal by the mainstream medical establishment, it has been shown that a truly ideal total level is probably 50 or 70mg / dl and apparently the lower the better.
I'm
less concerned about
LDL - C as i agree it's the particle size that matters most, but with ApoB being elevated, I was curios to see what your thought were on this being on a LCHF diet.
A 2005 study in the Journal of Nutrition found that raw - foodists were far
less likely than the general population to register high levels of «bad»
LDL cholesterol.
If you don't smoke, and your
LDL to HDL ratio is
less than three to one and your triglycerides are under 150, even if your total count is 250, that's a real good indication your lipid metabolism is working correctly and, here's the real point, your arteries are likely to be nice and clean.
I suspect if you keep carbs under 100 gm per day, or
less if you need to lose weight (30 - 70 is ideal), your
LDL will drop.
I've been on the the paleo - gluten - dairy - free diet for 7 months and my inflammation marker c - reactive protein is
less than 0.200, my triglycerides are at 49, my HDL at 47, but my
LDL is at 158 + probably from all the meat I've been eating,,, Does this cookbook have guidelines for this kind of balancing?
You may not feel your blood pressure improving [27], your
LDL becoming
less oxidized [28], the dimming of your inflammatory status [29], or the large number of HDL particles coursing through your veins [30], but you'll notice your health markers improving on lab tests and doctor visits.
(See Good Calories Bad Calories, by Gary Taubes, and Chapter 9 of my book, Deep Nutrition) The chance of this woman, who has no other risk factors for heart disease aside from high
LDL cholesterol (a poor predictor by itself), benefitting from Crestor's ability to prevent heart attacks is
less than one in a thousand.
So, even though my
LDL / HDL isn't
less than 3:1, I can still eat eggs?
Yet these fats are
less susceptible to oxidation, and this may be why they show up in
LDL — because they are of higher quality and therefore should preferentially be delivered to the tissues for functional roles rather than as fuel (i.e., free fatty acids).
«What we did find is in late mid-life, when we analysed their health, the animals on the best diet, the low - protein / high carbohydrate diets, had better blood pressure, had better
LDL cholesterol, had better glucose tolerance,
less diabetes and so on,» he said.
Of course, a low fat, high carbohydrate diet decreases ApoA1, but this doesn't mean it's bad if you're insulin sensitive and have low TGs (and low
LDL) eating such a diet, as many people do; the lower lipid circulation all round probably just means that
less ApoA1 will be required for equilibrium.
Coconut oil was found to raise total cholesterol and
LDL cholesterol (the «bad» kind of cholesterol shown to be correlated with a higher risk of heart disease) more than safflower oil and beef fat, but
less than soybean oil and butter = maybe not so good.
It didn't grew from April to now with
LDL around 120 - 130, was maybe a little
less.
Some say its a numbers game, the more
LDL particles crashing the walls of the arteries... some say the
LDL is only a sign of vascular damage, but if my
LDL goes down on a low fat diet, then that is bs, some say that it may be a problem metabolizing
LDL, intake / production is outpacing
LDL receptor activity, some say its not
LDL but
LDL that stays in the blood too long and oxidizes, some say about 20 % carbs (I was
less than 5 %) will produce just enough insulin to help metabolize cholesterol, but the hard core low carb guys, say the whole cholesterol thing is a scam and cholesterol under 500, without insulin resistance is nothing to worry about.
Green tea: Studies have found that those who regularly drink green tea have
less inflammation, lower
LDL cholesterol and better endothelial function than those who do not.
We know that (1) hunter gatherers eat much
less palmitic acid than many modern day «paleo dieters» and (2) they tend to have low, rather than high
LDL cholesterol.
The majority of studies observe that habitual tea drinkers, whether it be green or black, who consumed two cups or more a day, have
less stroke incidences,
less heart disease occurrences and, lower overall and
LDL («bad») cholesterol.
This optimization in
LDL cholesterol was
less pronounced in the low fat group compared to the low - carb ketogenic diet group.
In contrast, large reviews of previous trials suggest that very LC diets may have
less favorable effects on
LDL cholesterol than conventional high - carb, low - fat diets (13, 14).
My diet virtually always increases
LDL particle size (I have measured this for over a decade), such that they are not harmful (
less oxidized), but rather beneficial, and can be delivered as necessary to make new cell membranes and cells.