Organophosphate insecticides inhibit acetylcholinesterase causing excess
levels of acetylcholine at the nerve end paralyzing the muscle.
This ambiguity was explained by JJ Noval at NAMRL in Pensacola, when he showed that magnetic fields that vibrated in the ELF range would increase
the levels of acetylcholine in the rat brainstem directly.
Huperzine A extracted from the Chinese club moss is a herbal supplement that has been shown to maintain healthy
levels of acetylcholine.
Hypothyroidism causes lowered
levels of acetylcholine production which leads to forgetfulness and often constipation.
The nutrient DMAE works to maintain healthy
levels of acetylcholine, an important neurotransmitter for cognitive function.
Galantamine works by boosting brain
levels of acetylcholine, which helps brain cells communicate.
Tony and his clientele admit to taking stronger «smart drugs» than those he's selling tonight, drugs such as piracetam (which supposedly boosts
levels of acetylcholine in the brain), hydergine and propanolol (both said to work by increasing blood flow through the brain), L - Dopa (a chemical precursor of the neurotransmitter dopamine), phenytoin, vasopressin, deprenyl, the list seems almost endless.
To investigate the longer - term effects of higher - than - normal acetylcholine levels on the brain, Hermona Soreq of the Hebrew University of Jerusalem and her colleagues first induced high
levels of acetylcholine by forcing 26 mice to swim, an activity stressful to mice.
Not exact matches
In animal models, exposure to cigarette smoke or nicotine during fetal development alters the expression
of the nicotinic
acetylcholine receptor in areas
of the brainstem important for autonomic function, 28 alters the neuronal excitability
of neurons in the nucleus tractus solitarius (a brainstem region important for sensory integration), 29 and alters fetal autonomic activity and medullary neurotransmitter receptors.30 In human infants, there are strong associations between nicotinic
acetylcholine receptor and serotonin receptors in the brainstem during development.31 Prenatal exposure to tobacco smoke attenuates recovery from hypoxia in preterm infants, 32 decreases heart rate variability in preterm33 and term34 infants, and abolishes the normal relationship between heart rate and gestational age at birth.33 Moreover, infants
of smoking mothers exhibit impaired arousal patterns to trigeminal stimulation in proportion to urinary cotinine
levels.35 It is important to note also that prenatal exposure to tobacco smoke alters the normal programming
of cardiovascular reflexes such that there is a greater - than - expected increase in blood pressure and heart rate in response to breathing 4 % carbon dioxide or a 60 ° head - up tilt.36 These changes in autonomic function, arousal, and cardiovascular reflexes might all increase an infant's vulnerability to SIDS.
In the Gulf War, soldiers were both exposed to stress and given AChE inhibitors to prevent nerve gas damage, which might combine to create a brief burst
of unusually high
acetylcholine levels — and a long - term
acetylcholine deficit, Soreq points out.
AChE inhibitors have also been given to Alzheimer's patients in an attempt to boost
acetylcholine levels and improve cognitive function, she says — but her research suggests that the long - term effect may be the opposite
of that intended.
Such weapons form a covalent bond to and inactivate acetylcholinesterase (AChE), an enzyme that breaks down
acetylcholine in synapses, thereby regulating
levels of the neurotransmitter.
Also in the mix is the neurotransmitter
acetylcholine, higher
levels of which are associated with aggression.
The brain is well buffered against the effect
of arbitrary increases or decreases in circulating chemicals so that simply consuming food additives which are
acetylcholine precursors will not normally increase your brain
acetylcholine level.
In VD neurons, which do not remodel, another transcription factor maintains high
levels of OIG - 1 to block the relocation
of acetylcholine receptors.
It eases dementia symptoms by protecting
levels of the brain chemical
acetylcholine.
Clinically depressed people with low
levels of dopamine often see a shrinkage in penis size which seems to stop if they take a dopamine enhancer like L - dopa or mucuna pruriens or
acetylcholine.
This process
of deep memory consolidation requires
acetylcholine, and so compounds that can help increase
acetylcholine levels in the brain are commonly included in memory pills.
Ginsenosides increase
acetylcholine levels in the brain by blocking the inhibition
of the cholinergic pathway.
A-GPC crosses the blood - brain barrier to boost choline
levels in the brain, and it is involved in the synthesis and release
of acetylcholine (AC).
The benefits are lengthy, from memory enhancement, neuron health, increases
acetylcholine levels, anti-oxidant role in the brain, and
of course can aid in mitochondrial fat oxidation.
A study from the Journal
of Neurochemistry [1] found that whilst DMAE does enhance
acetylcholine levels, it doesn't necessarily mean that it makes it more bioavailable.
It increases the
levels of neurotransmitters such as dopamine and
acetylcholine; it boosts the uptake and use
of glucose in brain cells; and improves communication between brain cells.
It measures the
level of antibodies to the dog's
acetylcholine receptors that are circulating in its blood.