Maternal obesity increases hypothalamic leptin receptor expression and sensitivity in juvenile obesity - prone rats.
Not exact matches
Is the
increased maternal mortality caused by the high C - Section rate, or just associated with it and caused by the same factors, such as
obesity, GD, high blood pressure, multiples,
maternal age, and so on?
Research has shown that
obesity during pregnancy is associated with a significantly
increased risk of
maternal and fetal complications, including:
What can be done: Researchers believe that the uptick in the U.S.
maternal mortality rate is due mainly to three factors: a decline in overall health, including
increased rates of
obesity and cardiovascular disease, inadequate prenatal care, and inadequate postpartum care.
I made a quick search for «
maternal death rate increase» Everything I found points to the same thing: Maternal death rate increased in the USA because of the increasing rates of obesity, diabetes and hypertension and other kind of risk factors (Which raises the risk of pre
maternal death rate
increase» Everything I found points to the same thing:
Maternal death rate increased in the USA because of the increasing rates of obesity, diabetes and hypertension and other kind of risk factors (Which raises the risk of pre
Maternal death rate
increased in the USA because of the
increasing rates of
obesity, diabetes and hypertension and other kind of risk factors (Which raises the risk of pregnancy).
Some cite elevated rates of
obesity and rising
maternal age, which both
increase the C - section risk.
Maternal obesity causes a twofold
increased risk of atonic postpartum hemorrhage more than 1,000 mL.
Disturbances in
maternal glucose metabolism, such as
increased insulin resistance or decreased insulin production, may be a key factor in the observed relations between older
maternal age, larger birth weight,
obesity, and delayed OL.
Other factors, such as older parents and
maternal obesity, can also
increase the risk of autism.
«While it is known that these drugs can
increase the risk of
obesity in adults, it is unknown whether a woman's antidepressant use during pregnancy
increases the risk of metabolic disturbances in her children,» Holloway says, adding the goal of their project was to determine whether
maternal exposure to a commonly used antidepressant is related to the development of fatty liver, an outcome commonly seen with
obesity, in the offspring.
Overall, the authors found that the rates of low Apgar scores
increased with
maternal Body Mass Index (BMI): overweight (BMI 25.0 - 29.9) was associated with a 55 %
increased risk of low Apgar scores at 5 minutes;
obesity grade I (BMI 30 - 34.9) and grade II (BMI 35.0 - 39.9) was associated with a 2-fold
increased risk; and
obesity grade ΙII (BMI ≥ 40.0) was associated with a more than 3-fold
increase in risk.
Analysis of the data indicated that
maternal overweight (body mass index [BMI] of 25 to 29.9) and
increasing grades of
obesity (BMI 30 or greater) were associated with
increasing rates of cerebral palsy.
It is uncertain whether risk of cerebral palsy in offspring
increases with
maternal overweight and
obesity severity and what could be possible mechanisms.
Although the reasons for
increased risk to male babies are not known, they could include developmental differences in the growth and function of the placenta, or
increased sensitivity of male fetuses to environmental factors experienced by the mother, including
obesity, smoking, advanced
maternal age, and social deprivation.
The association of
maternal obesity during gestation and chronic conditions in children is beginning to be explored, 37,38 and previous studies alluded to an
increased rate of health problems generally in caregivers of children with disabilities.39, 40 Associations between male sex and poverty and behavior / learning problems are congruent with other studies.41 - 43 The association of minority race / ethnicity with asthma and
obesity and the inverse relationship of minority race / ethnicity with other physical conditions and behavior / learning problems are consistent with previous studies.12,43 - 45
Some evidence suggests that
maternal stress,
obesity, pesticides, and advanced parental age may
increase the risk of ASDs but Dr Baxter said that better recognition of the disorders and changed diagnostic criteria were more likely to explain different study findings over time.
Conclusions Chronic
maternal IPV is associated with
increased risk of
obesity among preschool - aged children.
Testing the hypothesis that certain
maternal feeding behaviors
increase children's adiposity is difficult because the suspect feeding behaviors tend to cluster within low - income and minority populations, which have a higher risk of childhood
obesity (22, 23).
The association of
maternal obesity during gestation and chronic conditions in children is beginning to be explored, 37,38 and previous studies alluded to an
increased rate of health problems generally in caregivers of children with disabilities.39, 40 Associations between male sex and poverty and behavior / learning problems are congruent with other studies.41 - 43 The association of minority race / ethnicity with asthma and
obesity and the inverse relationship of minority race / ethnicity with other physical conditions and behavior / learning problems are consistent with previous studies.12,43 - 45
We can not determine from this cross-sectional study whether either
maternal obesity or
maternal depressive symptoms cause
increased TV viewing in children and, if so, what other factors may mediate these relationships.
Based on prior empirical and theoretical work, this study investigated the following hypotheses: (1)
maternal exposure to IPV will be associated with higher odds of
obesity at age 5 years in their children; (2)
maternal exposure to IPV will be associated with feeding practices and behaviors that elevate risk for childhood
obesity; and (3)
maternal perception of lower neighborhood safety will
increase the effect of IPV on childhood
obesity risk.
It also aims to investigate longitudinal associations, thus
increasing the understanding of the development of
maternal and child
obesity, glucose intolerance and mental health problems.
Stratified analyses indicated
increased risk for
obesity among girls with a
maternal history of chronic IPV (adjusted odds ratio = 2.21; 95 % confidence interval, 1.30 - 3.75) compared with boys (adjusted odds ratio = 1.66; 95 % confidence interval, 0.94 - 2.93) and a larger effect of any
maternal IPV on
obesity among children living in less safe neighborhoods (adjusted odds ratio = 1.56; 95 % confidence interval, 1.03 - 2.36).
Maternal pre-pregnancy
obesity has been linked with an
increased risk for negative emotionality and inattentiveness in offspring in early childhood.