Not exact matches
In a new study based on
mice, scientists at Lancaster University found that a drug that
goes after three diabetes - related targets «significantly reversed the memory deficit» in
mice who got the drug, as measured by their performance in a maze
test when compared to
mice who didn't get the drug.
After they were given the drug, the
mice were asked to
go through a maze that
tested their memory.
Of course, there is still a long way to
go before this particular method will be
tested on humans (it was
tested on
mice), and an even longer way to
go before it'll be used in medical therapies (if it ever will translate into therapies), but one thing is becoming clear: We need not compromise our moral principles and rush into government - funded embryo - destructive research.
These
mice also
went on to exhibit a range of depressive symptoms in behavioral
tests.
You can look for efficacy in cell cultures, but that tells you almost nothing about what's
going to happen when you
test in a
mouse or a human.
In their experiments, Witten and her team gave two
mice a chance to socialize in a cage that limited the mobility of one of the
mice (the «social target»), so the
test mouse could choose whether or not to
go to the target for friendly behaviors like sniffing and grooming.
Meanwhile, when he and his team
tested JWH - 133 on
mice without CB2 receptors, the rodents kept
going for the cocaine, and their dopamine levels were higher.
While the UC Riverside did not look at the toxicity of
GO in their study, researchers at the Hersam group from Northwestern University did report in a paper published in the journal Nano Letters («Minimizing Oxidation and Stable Nanoscale Dispersion Improves the Biocompatibility of Graphene in the Lung») that
GO was the most toxic form of graphene - based materials that were
tested in
mice lungs.
Giving PPAR - delta a boost with an existing drug was protective in HD cells and
mice, but we'll likely need to research and
test it further before it can
go to the HD clinic.
Knowing that oxytocin has been implicated in wound healing [47] and immune health [24], we
went on to
test roles for oxytocin during L. reuteri - induced wound repair events in our
mouse models.