Not exact matches
Alpha - lac, the Lund and Karolinska teams have demonstrated, can persuade
mitochondria torelease
cytochrome c.
More of both support cell suicide as now sick or injured cells signal
mitochondria to release factors such as
cytochrome c into the cell cytoplasm.
All of this damage damages extremely precious mitochondrial DNA, which make the
mitochondrias dysfunctional, clogging lysosome for mitophagy, and being incapable of keeping mitochondrial membrane potential (leading to
Cytochrome C loss through mitochondrial permeability pores) and mitochondrial ATP OXPHOS respiration.
In your
mitochondria, there's also a specific molecule called
cytochrome c oxidase, which is involved in the energy production within the
mitochondria.
This toxin stops the cell respiration as a non-competitive inhibitor for an enzyme in the
mitochondria that is referred to as
cytochrome c oxidase, causing asphyxiation in the cells.