Sentences with phrase «models on depressive symptoms»

Thus, we conducted another test to examine the effect of changes in different exercise models on depressive symptoms, as shown in table 4.

Not exact matches

Results have shown this model may reduce depressive symptoms by up to 50 % compared with usual care, and it is on this model that we base our study.

We applied generalised linear mixed models via PROC GLIMMIX to estimate the effects of different transitional patterns of exercise on depressive symptoms with HLDS as the event, after adjusting for the previous CESD score, age, gender, level of education, marital status, smoking, physical function, emotional support, social participation, self - rated health, economic satisfaction, employment and 10 chronic conditions.

The impacts of different amounts of exercise on depressive symptoms were analysed using generalised linear mixed models.

Addressing this gap, the present study examined multiple factors longitudinally that link parental depressive symptoms to adolescent adjustment problems, building on a conceptual model informed by emotional security theory (EST).

While there is strong evidence supporting effectiveness of collaborative care for adult depression, Richardson et al's study adds to results of two other studies in supporting the value of collaborative care models for adolescent depression: (1) using a similar model, Asarnow et al1 found significant advantages for collaborative depression care versus usual care (UC); (2) using a stronger medication treatment as usual condition, collaborative care with psychotherapy plus medication yielded a marginal advantage on depressive symptoms and significant advantage on mental health - related quality of...

Prospective Effects of Marital Satisfaction on Depressive Symptoms in Established Marriages: A Dyadic Model.

The results of the study performed on the model suggest that both parents and children contribute to the development of depressive symptoms.

[jounal] Beach, S. / 2003 / Prospective effects of marital satisfaction on depressive symptoms in established marriages: A dyadic model / Journal of Social and Personal Relationships 20: 355 ~ 371

This model included the additional time - varying covariates (Level 1) representing (a) the effect of patient self - rated health on spouse depressive symptoms and (b) the effect of spouse self - rated health on patient depressive symptoms and the time - invariant effects (Level 2) representing (c) the effect of mean patient self - rated health on mean spouse depressive symptoms and (d) the effect of mean spouse self - rated health on mean patient depressive symptoms.

In order to identify the number and patterns of these trajectories, depressive symptoms should be measured on at least three different times and modeled using a group based trajectory approach.

The findings from these studies converge on the theme that attachment theory has considerable utility in potentially extending and refining current cognitive vulnerability models through a consideration of interpersonal context and the cognitive mechanisms by which negative interpersonal experiences may confer increased risk to later anxious and depressive symptoms.

Future studies could test hypotheses based on this model to improve our understanding of the development of adolescents» depressive symptoms in both boys and girls.

In line with biopsychosocial models, results indicate that the effect of pubertal timing on depressive symptoms must be conceptualized through complex interactions between characteristics of adolescents» interpersonal relationships and prepubertal vulnerabilities.

Second, the dependent variable (depressive symptoms) was regressed on the independent variable (marital status) in Model 1 (Tables 4 and 5).

Fourth, the dependent variable (depressive symptoms) was regressed on both the independent variable (marital status) and the mediator (social ties) in Models 5 — 7.

Third, the dependent variable (depressive symptoms) was regressed on the mediator (social ties) in Models 2 — 4.

In Models 5 — 8, the variables related to social ties were added to Model 1 as the fourth step in regressing depressive symptoms (dependent variable) on both marital status (independent variable) and social ties (mediator).

The impact of marital status on depressive symptoms was less pronounced in Model 7 than in Model 1 in both men (Model 1, β =.109; Model 7, β =.097) and women (Model 1, β =.082; Model 7, β =.040).

Model 1 included the marital status variable and covariates (age, education, equivalent household income, at least one disability, number of chronic diseases, number of children, participation in economic activity, and number of social activities) as the second step in regressing depressive symptoms (dependent variable) on marital status (independent variable).

Based on the above - described considerations, we tested a theoretical model (Fig. 1) in which the association between self - esteem and depressive symptoms is partly mediated by approach and avoidance motivation and social factors.

Multi-level modeling analyses indicated that, on average over the course of treatment, variable and less severe obsessive — compulsive symptoms significantly predicted a decrease in depressive symptoms.

Group - based trajectory modeling analyses conditional on risk and protective factors identified four trajectories of depressive symptoms across adolescence: moderate stable (MS; 54.57 % of the sample), low stable (LS; 27.16 %), moderate increasing (MI; 11.30 %), and high declining (HD; 6.97 %).

The fitted model suggests that while measures of neuroticism are contaminated by the effects of short term mental state on the reporting of personality, there is still a fairly substantial relationship between trait neuroticism (corrected for the effects of mental state contamination) and reports of depressive symptoms.

Once the outcome models were derived, inverse probability weighting (IPW)[47] was used as a sampling weight to investigate the possible influence of selective participation on our estimates of association between parental alcohol and offspring conduct problems and depressive symptoms, respectively.

Past research relying on either cross-sectional or main effect models makes the assumption that low levels of perceived control will uniformly and consistently predict higher levels of depressive symptoms without taking into account contextual and environmental factors such as stress (see Alloy et al. 1988).

Findings from recent studies indeed provide support for a mediating model by which children's poor peer experiences have an indirect effect on depressive symptoms via feelings of loneliness.

This study investigated the role of parental Autism spectrum disorder (ASD), attention - deficit / hyperactivity disorder (ADHD), and depressive symptoms on parenting stress in 174 families with children with ASD and / or ADHD, using generalized linear models and structural equation models.

There was a significant difference in model fit between Model 1 and Model 1a, Δ χ 2 (1, N = 356) = 5.440, p < 0.05, indicating that the effect of maternal depressive symptoms on peer social preference was only partially mediated through maternal wamodel fit between Model 1 and Model 1a, Δ χ 2 (1, N = 356) = 5.440, p < 0.05, indicating that the effect of maternal depressive symptoms on peer social preference was only partially mediated through maternal waModel 1 and Model 1a, Δ χ 2 (1, N = 356) = 5.440, p < 0.05, indicating that the effect of maternal depressive symptoms on peer social preference was only partially mediated through maternal waModel 1a, Δ χ 2 (1, N = 356) = 5.440, p < 0.05, indicating that the effect of maternal depressive symptoms on peer social preference was only partially mediated through maternal warmth.

We also compared this model with a model in which the direct effect of maternal depressive symptoms on children's social preference was set at zero (Modelmodel with a model in which the direct effect of maternal depressive symptoms on children's social preference was set at zero (Modelmodel in which the direct effect of maternal depressive symptoms on children's social preference was set at zero (ModelModel 1a).

We also compared these models with a model (Model 2a) in which the direct effect of maternal depressive symptoms on peer social preference was set at model (Model 2a) in which the direct effect of maternal depressive symptoms on peer social preference was set at Model 2a) in which the direct effect of maternal depressive symptoms on peer social preference was set at zero.