Sentences with phrase «more about human diseases»

At the Bateson Centre at the University of Sheffield, biologists and hospital doctors have teamed up to use zebrafish embryos to learn more about human diseases.

Not exact matches

With the advances in knowledge that are almost certain to be gained from the Human Genome Initiative — or, if its critics should win the day and it lose support, from more piecemeal genetic - research — we will know more and - more about genetic factors causally related to health and disease and to other important aspects of life, such as intelligence and emotional states.

«This may contribute to antibiotic resistance, [and] scientists are more concerned about disease - causing bacteria that develops resistance in farm animals to then infect human beings.

Learn more about this topic in our essay Opening Pandora's Bread Box: The Critical Role of Wheat Lectin in Human Disease.

Leendertz and colleagues from several different scientific disciplines traveled to Meliandou hoping to find the animal source, or reservoir, and learn more about how the disease might have spilled over into humans.

Svendsen is more optimistic about his team's work involving human tests of a novel stem cell approach to treat ALS, a degenerative motor neuron disease in which cells that transmit messages from the brain and spinal cord to the muscles wither or die.

Membrane proteins make up about one third of all proteins in the human body, and their malfunction is associated with more than 500 diseases.

This will allow to understand more about genes we currently know very little about, and open up new avenues for research into the genetics of human disease.

Human cases are rare, about five to 10 cases a year, but 35 % to 50 % of those with the disease die, and more than a third of its survivors have lasting neurological damage.

The researchers say they'd now like to learn more about how these genes influenced humans» ability to survive and what implications they might have for disease.

The gift accompanies a paper published online today in Nature from researchers at Broad and worldwide, which identifies more than 100 areas of the human genome associated with schizophrenia, based on samples from almost 37,000 people with schizophrenia and about 113,000 without the disease.

The genomic data is important, say researchers, because it serves as a key reference point for when and where the parasite existed in humans, and provides more information about the evolution of human disease.

Developing the capability to experiment with prions in bacteria could help to reveal more about the behavior of human prions, which may be linked to diseases such as Alzheimer's and Parkinson's, says Jeffrey Roberts, a molecular biologist at Cornell University in Ithaca, New York.

Unravelling this mechanism could also help scientists understand more about human cell dysfunctions linked to disorders such as diabetes, Parkinson's and other neurodegenerative diseases.

We wondered whether we could find out a lot more from these patients about basic human immunology and how their immune systems might be keeping these diseases at bay.»

Whether you want to discover new mechanisms of human disease, need a genome assembly for an organism you study, or just want to know more about us, Dovetail Genomics is here to help.

The use of NHP in biomedical research is justified in the studies of human diseases, to learn more about their causal pathways, their development, their infectiveness and to develop therapeutic and preventive strategies.

«Because scientists can selectively switch off genes in mice, more will be learnt about human disease from the mouse genome than from the human genome.

The stem cell research community hopes that as more is understood about STAP cells, they will join embryonic stem and iPS cells as another reprogramming tool for use in their collective quest to understand and treat human disease.

Health improvement (allowing to post - pone / escape the diseases and thus live, healthier / disease - free longer, but not above human MLSP of around 122 years; thus these therapies do not affect epigenetic aging whatsoever, they are degenerative aging problems not regular healthy aging problem (except OncoSENS - only when you Already Have Cancer - which cancer increases epigenetic aging, but cancer removal thus does not change anything / makes no difference about what happens in the other cells / about what happens in the normal epigenetic «aging» course in Normal non-cancerous healthy cells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to MLSP).

Genetically modified mice are often used to learn about human diseases, but in some cases primates will be more informative.

Studying how genomes are alike — or different — teaches us more about the diversity of life on Earth, how life evolved, even the genomic links to human disease.

He taught me a lot about evolutionary medicine and nutrition in general, opened many doors and introduced me (directly and indirectly) to various players in this field, such as Dr. Boyd Eaton (one of the fathers of evolutionary nutrition), Maelán Fontes from Spain (a current research colleague and close friend), Alejandro Lucia (a Professor and a top researcher in exercise physiology from Spain, with whom I am collaborating), Ben Balzer from Australia (a physician and one of the best minds in evolutionary medicine), Robb Wolf from the US (a biochemist and the best «biohackers I know»), Óscar Picazo and Fernando Mata from Spain (close friends who are working with me at NutriScience), David Furman from Argentina (a top immunologist and expert in chronic inflammation working at Stanford University, with whom I am collaborating), Stephan Guyenet from the US (one of my main references in the obesity field), Lynda Frassetto and Anthony Sebastian (both nephrologists at the University of California San Francisco and experts in acid - base balance), Michael Crawford from the UK (a world renowned expert in DHA and Director of the Institute of Brain Chemistry and Human Nutrition, at the Imperial College London), Marcelo Rogero (a great researcher and Professor of Nutrigenomics at the University of Sao Paulo, Brazil), Sérgio Veloso (a cell biologist from Portugal currently working with me, who has one of the best health blogs I know), Filomena Trindade (a Portuguese physician based in the US who is an expert in functional medicine), Remko Kuipers and Martine Luxwolda (both physicians from the Netherlands, who conducted field research on traditional populations in Tanzania), Gabriel de Carvalho (a pharmacist and renowned nutritionist from Brazil), Alex Vasquez (a physician from the US, who is an expert in functional medicine and Rheumatology), Bodo Melnik (a Professor of Dermatology and expert in Molecular Biology from Germany, with whom I have published papers on milk and mTOR signaling), Johan Frostegård from Sweden (a rheumatologist and Professor at Karolinska Institutet, who has been a pioneer on establishing the role of the immune system in cardiovascular disease), Frits Muskiet (a biochemist and Professor of Pathophysiology from the Netherlands, who, thanks to his incredible encyclopedic knowledge and open - mind, continuously teaches me more than I could imagine and who I consider a mentor), and the Swedish researchers Staffan Lindeberg, Tommy Jönsson and Yvonne Granfeldt, who became close friends and mentors.

And although more clinical trials about the benefits of curcumin for humans are needed, it's been linked to lower total cholesterol levels and improved liver function after liver disease or damage.

At first, I had difficulty believing what I was reading, but it's true and you can read more information about the study on clinicaltrials.gov: Inoculating Celiac Disease Patients With the Human Hookworm Necator Americanus: Evaluating Immunity and Gluten - sensitivity.

It is our hope to help you gain more understanding of True Health by studying about the disease, what is it, what causes it, and most of all, its effect to the human body.

The more I protested about this ambiguity, the more Joanna pointed out to me that it was both a terrible and wonderful part of life: terrible because you can't count on anything for sure — like certain good health and no possibility of cancer; wonderful because no human being knows when another is going to die — no doctor can absolutely predict the outcome of a disease.

By contrast, cats, humans and dogs are naturally more resistant to the disease, and it is estimated that only about 15 % of bites from affected animals will result in the rabies virus in humans and domestic animals.

You asked me earlier about whether human habits of concentrating poultry made the disease more dangerous.

In classes like Human Body in Health and Disease and Cardiopulmonary Resuscitation, you can learn more about how the human body works and how to perform lifesavingHuman Body in Health and Disease and Cardiopulmonary Resuscitation, you can learn more about how the human body works and how to perform lifesavinghuman body works and how to perform lifesaving CPR.