The release of inflammatory cytokines, or intercellular signaling molecules such as interleukin - 1 (IL - 1), interleukin - 2 (IL - 6), and tumor necrosis factor alpha (TNF - α) at the site of immune activation causes other immune cells migrating throughout the lymphatic vessels of the body to express
more cell adhesion molecules (CAMs).
Not exact matches
Now a
more fine - grained picture of
adhesion mechanics is emerging, thanks to a new tool developed in Illinois in recent years called a «tension gauge tether,» which allows scientists to measure
cell mechanics at the single -
molecule level.
The constitutive expression of these endothelial
cell adhesion molecules was very similar for most of the lines derived from different organs, i.e., very low expression levels of E-selectin and VCAM - 1 and
more pronounced expression of ICAM - 1.
The endothelial
cells in insulin - receptor knockout mice expressed
more of the VCAM - 1
adhesion molecule that helps white blood
cells grab onto the growing plaques, and blood vessels gathered four times as many white blood
cells.