Medical malpractice plays a large role when it comes to
neonatal hypoxic ischemic encephalopathy.
Addition of vitamin D to hypothermia and NAC following
neonatal hypoxic ischemia improves functional outcomes and preserves brain volume in male rodents, report researchers at the Medical University of South Carolina (MUSC) in the September 1, 2017 issue of Neuropharmacology.
«Multi-mechanism approach to treating
neonatal hypoxic ischemia: Vitamin D added to N - acetylcysteine and hypothermia following
neonatal hypoxic ischemia improves functional outcomes, particularly in males.»
In preclinical studies, hypothermia and anti-oxidant treatment with N - acetylcysteine (NAC) are neuroprotective following
neonatal hypoxic ischemia in females, but less so in males.
The poster entitled Home birth and risk of
neonatal hypoxic ischemic encephalopathy, to be presented at the forthcoming February meeting of the Society of Maternal - Fetal Medicine looks at precisely this issue.
Not exact matches
There's HIE, or
hypoxic neonatal encephalopathy.
History of preterm birth at < 34 completed weeks gestation, Recurrent miscarriage, Moderate to severe pre-eclampsia (see detailed guidelines), Intra Uterine Growth Restriction (see detailed guidelines), Caesarean section, previous unexplained stillbirth, eclampsia, uterine rupture, placental abruption, PUPP, Obstetric cholestasis, 3rd or 4th degree tear, definitive PPH, manual removal of placenta, shoulder dystocia,
neonatal death, infant with
hypoxic ischaemic encephalopathy, midtrimester miscarriage · Previous gynaecological history.