Malignant adrenal cancers may spread through the body by invading the adjacent blood vessels and seeding
new tumors in body cavities and other organs.
Not exact matches
Tumors are notoriously difficult to study
in their natural habitat —
body tissues — but a
new synthetic tissue environment may give cancer researchers the next - best look at
tumor growth and behavior.
Researchers at the University of Leeds have made a
new synthetic anti-cancer molecule that targets two key mechanisms
in the spread of malignant
tumors through the
body.
In a
new study, a team found that injecting mice with tiny magnets and cranking up the heat eliminated
tumors from the animals»
bodies with no apparent side effects.
It is well established that melanoma cells can spread through the blood to accumulate and form
new tumors (metastases)
in other parts of the
body away from the original
tumor.
Many
tumors spread: Single cancer cells migrate with blood flow through the
body before they settle
in new tissue.
Only a few cells
in a cancerous
tumor are able to break away and spread to other parts of the
body, but the curve along the edge of the
tumor may play a large role
in activating these
tumor - seeding cells, according to a
new University of Illinois study.
Metastasis is the process
in which cells from a primary
tumor break - off, enter the blood stream and create
new tumors elsewhere
in the
body.
This allows cancer cells to break off from
tumors, spread throughout the
body (
in blood or other fluid) and form
new tumors at distant sites — a process called metastasis.
Siva Vanapalli, an associate professor
in the Department of Chemical Engineering, at Texas Tech University, recently received two grants from the Cancer Prevention and Research Institute of Texas (CPRIT) to study the movement of
tumor cells throughout the
body and
new methods of detecting cancer cells.
Research from Rutgers Cancer Institute of
New Jersey examining difficult to treat cancer
tumors through genomic profiling shows that
tumors with alterations
in a signaling pathway responsible for cell regulation may respond to targeted therapy regardless of where the
tumor originated
in the
body.
What followed is an illuminating tale of how one woman's intersection with experimental research helped open a
new frontier
in cancer treatment — with approval of a drug that, for the first time, capitalizes on a genetic feature
in a
tumor rather than on the disease's location
in the
body.
The
new iPS cells passed the standard tests for pluripotency: They formed
tumors called teratomas when injected into immunocompromised mice, and they could differentiate into cells from the three main tissue types
in the
body, including neurons, muscle and gut epithelium.
We are leaders
in the field of cancer immunotherapy and the development of
tumor vaccines and
new antibodies, working to activate the human
body's own immune system to fight cancer.
He adds that recent
new drugs on the market or
in clinical trials are attempting to disable Tregs
in tumors to help the
body's own immune system fight cancer.
CXCR4 is a receptor that cancer cells use like antennae to feel their way from their parent
tumor to distinct sites
in the
body where they will establish a
new tumor.
The aggressive basal - type breast cancers often metastasize, seeding
new tumors in distant parts of the
body.
Once
tumor cells strike out on their own and metastasize to
new sites
in the
body, drugs and other therapies rarely do more than prolong a patient's life for a few years.
Metastatic disease refers to cancer that has spread through the blood or lymph system to form
new tumors in other parts of the
body distinct from the original site.
A story
in last Friday's
New York Times highlights research on «cancer stem cells»: a fraction of cells
in a
tumor that are especially resistant to chemotherapy and resemble the
body's non-cancerous stem cells
in their ability to renew themselves.
The chronically stressed mice had decreased immune function and experienced
tumor development significantly earlier than the non-stressed mice.16 Other mouse studies of ovarian cancer showed that chronic stress resulted
in increased cancer growth as well as increased angiogenesis, the process with which cancer forms
new blood vessels to feed itself nutrients for growth and metastases.17 Chronic stress has also been shown to decrease our
body's ability to mount an attack against foreign invaders, including viruses.18 As we know that several viruses can cause cancer (HPV and cervical cancer, and EBV and nasopharyngeal cancer), we can extrapolate that any decrease
in immune function could increase cancer risk.
Others may experience
new tumors, located
in the bladder or
in other areas of the
body.
When they have moved (spread) to a
new location
in the
body they are called metastatic
tumors.