When a neuron sends a message it releases the neurotransmitter serotonin, which is detected by
the next neuron receiving the message.
It is due to these proteins that the information arriving at a synapse can be transmitted and then received by
the next neuron.
Neurons sense a signal through branching dendrites, carry this signal to the cell body, and send it onwards through a long axon to signal
the next neuron.
Some signals that the dendrites receive do not continue to
the next neuron; instead they seem to change the way that the neuron handles the subsequent signals.
Neurons communicate with a series of electrical pulses; chemical signals transiently change the electrical properties of individual cells, which in turn trigger an electrical change in
the next neuron in the circuit.
In the brain, interneurons are usually inhibitory: when they receive a signal, they make
the next neuron down the line less likely to continue the transmission.
When an electrical signal arrives at a nerve terminal and needs to be passed along to
the next neuron, neurotransmitters, or chemical messengers, packed in vesicles mediate this process.
With each successive stimulus, Lømo found, the response in
the next neuron became stronger — an effect that lasted for hours.
So when the neurons secrete serotonin,
the next neuron is able to actually connect with that very easily and realize «hey, this is the message that we need to get across.»
Coincidentally, a ketogenic diet reduces intracellular sodium concentration as well, mainly due to slightly increased acidity of the blood; it also decreases the level of excitatory neurotransmitters (which excites
the next neuron), further dampening neuronal activation.
The messengers are then captured by
the next neuron, and can either excite or inhibit it depending on their chemical makeup.