Sentences with phrase «of cu»

Prior to joining the company, he was chief lending / originations officer of CU Business Partners, LLC, one of the nation's largest credit union service organizations (CUSO).

At the end of December 2014, a video circulated that referred to CU as an «appraisal time bomb» and renewed concerns among NAR members about the implementation of CU.

NAR is watching the roll - out of CU closely and have asked members to give us feedback as soon as the tool is available and used by lenders at the end of January.

Reduced amygdala responses to fearful facial expressions have been reported in adolescents with CD or conduct problems, and elevated CU traits compared to typically - developing controls (Jones et al. 2009; Marsh et al. 2008), whereas increased amygdala responses to fearful faces have been demonstrated in children with conduct problems and low levels of CU traits compared with their high CU traits counterparts (Viding et al. 2012).

Adolescents with conduct disorder (CD) and elevated callous - unemotional (CU) traits have been reported to present with a more severe and persistent pattern of antisocial behaviour than those with low levels of CU traits.

However, we note that over 130 studies have used self - report measures of CU or psychopathic traits such as the ICU (Frick et al. 2014) and there is still no consensus whether parents, teachers or the young people themselves are better informants about the presence or absence of CU traits and the guidance in the DSM - 5 is that information from all three sources can be used in clinical settings (American Psychiatric Association 2013).

In addition, we examined for correlations between a continuous measure of CU traits and FA, AD, RD and MD in order to test the reliability and validity of the group - based analysis described above.

However, relatively few studies have investigated whether there are differences in brain structure between these subgroups.We acquired diffusion tensor imaging data and used tract - based spatial statistics (TBSS) to compare adolescents with CD and high levels of CU traits (CD / CU +; n = 18, CD and low levels of CU traits (CD / CU -; n = 17) and healthy controls (HC; n = 32) on measures of fractional anisotropy (FA), axial (AD), radial (RD) and mean (MD) diffusivity.

Thus, study findings warrant further exploration of how trauma might impact or maintain the development of CU traits over time and indicate that issues of differential diagnosis must be considered in order to more effectively tailor treatment for trauma - exposed CU youth.

We propose that one important factor responsible for the inconsistent findings described above is the varying levels of CU traits in CD samples.

However, when the CD participants were split according to their level of CU traits, a different picture emerged.

This is in line with the lack of association of CU traits and amygdala VBM measures reported here.

However, results show that high levels of CU traits predict elevated externalizing behavior problems, but only for toddlers exhibiting either high baseline respiratory sinus arrhythmia (RSA) or little to no RSA suppression in response to a fear stimulus.

Functional magnetic resonance imaging (fMRI) studies have provided evidence that individuals with conduct problems and high levels of CU traits (CD / CU +) process emotional stimuli differently from those with conduct problems and low levels of CU traits (CD / CU --RRB-.

Participants with high levels of CU traits were significantly more likely to be heterozygous (G / T) than participants low in CU traits.

Studies investigating the effects of CU traits on facial emotion recognition have also yielded mixed findings, with some studies showing that CU traits are associated with deficits in recognizing facial expressions signalling distress (i.e., fear and sadness; Dadds et al. 2008; Fairchild et al. 2009, 2010), whilst other studies have reported superior fear recognition in those with high versus low levels of CU traits (e.g., Woodworth and Waschbusch 2008).

Given the marked developmental changes that can occur at these early ages, when empathy is developing, the stability of CU difficulties is noteworthy.

Therefore, they could represent the first step in identifying the presence of CU traits, and the possible developmental pathways to antisocial behaviours, and in promoting early interventions.

A strong genetic liability, in interaction with parenting and relevant environmental factors, can lead to elevated levels of CU traits in children.

Furthermore, people with high levels of CU traits (psychopathic personality) have been shown to have three specific cognitive and emotional deficits; a poor conditioned fear response, reduced ability to recognise fear, and deficits in stimulus - reinforcement tasks (see Moul et al. [12] for a review).

These finding suggest that antisocial behaviour problems characterised by high levels of CU traits may have unique aetiological mechanisms associated with specific cognitive and affective impairments that are heavily dependent on genetics.

The purpose of the present study was to evaluate the specific contribution of both CU traits and ODD, cross-sectionally and longitudinally, to several psychological characteristics in a large community sample of preschoolers.

Indeed, we acknowledge that the present findings may be considered surprising given previous work showing that empathy deficits are more pronounced in those with CD and elevated CU traits than those with lower levels of CU traits (Jones et al. 2010; Schwenck et al. 2012) and theories predicting that affective empathy deficits are uniquely related to CU traits (Blair 2005, 2013).

General Linear Models (GLM, for psychological quantitative measures) and logistic regressions (for binary measures) assessed the specific contribution of CU levels and the presence of ODD diagnosis on the psychological measures.

Recently, Ray et al. [54] studying a sample of adolescents, have pointed out that the unemotional scale contributes to an overall CU factor but is weakly associated with other subscales; they recommend further research to conceptualize it within the broader construct of CU behaviors.

Additionally, the results of the GLM, adjusted by the covariates of the study, measuring the association of the CU raw score on the ODD measures also at age 3 for the children diagnosed with ODD at baseline (n = 61) showed that CU levels did not achieved significant contribution on the ODD level (p ≥.810; R2 ≤.006).

Although the CU trait levels tend to decreases across childhood and adolescence in a large number of children, those showing an elevated level of CU traits are at higher risk for keeping them in older ages.

Childhood antisocial behaviour problems characterised by high levels of CU traits have been found to be highly heritable [3], [4].

In addition, given the high heritability of antisocial behaviour problems in the presence of high levels of CU traits, it was hypothesized that CU traits would be significantly associated with functional polymorphisms of the serotonin - system.

Viding et al. [4] found that genetic variability accounted for 71 % of the variance in antisocial behaviour problems in 9 year old children with high levels of CU traits but only 36 % in those with low levels of CU traits.

Scores on the parent - and teacher - reported ICU were internally consistent and combined CU scores showed expected associations with an alternate measure of CU traits and measures of empathy, prosocial behavior, conduct problems, and aggression.

In order to comprehend the aetiology and development of CU traits it is necessary to get a better understanding of how these traits are characterised by the function of neurochemical systems in males and females across all developmental stages.

In addition, low levels of CU traits at the intermediate evaluation promoted an increase of positive parenting 1 year later.

This direction of the association, however, is concordant with the idea that the minor allele of SNP rs11568817 engenders a risk for higher levels of CU traits, which in turn were found to be associated with lower levels of peripheral serotonin.

It has been suggested that the levels of CU traits are also associated with a generalized impairment in the natural allocation of attention to emotionally salient stimuli that results in cascading errors in recognizing other people's emotions [33].

However, these studies raise the issue of translating the measures of CU traits into DSM - 5 criteria of CD with LPE.

For this modeling, the measures of CU (ICU - total raw score) and ODD (binary diagnosis present / absent) were considered as the independent variables and the analyses were adjusted by the covariates family SES, children's sex and ethnicity, presence of comorbidities other than ODD and the number of DSM - IV CD symptoms.

Recently, Waller et al. [67] applied Belsky's model in relation to the development of CU behaviour.

We also examined for effects of CU traits using a dimensional approach by testing for correlations between CU traits and EA, emotion recognition, and affective empathy (using either parametric or non-parametric bivariate correlations, as appropriate).

Disentangling CD patients through an evaluation of the presence of CU traits is a crucial component of the assessment.

Current evidence, however, is inconsistent and the precise role of CU traits versus antisocial and aggressive behaviour, and other comorbidities, is unclear.

This result supports the idea that previously reported mixed findings regarding the relationship between serotonin and antisocial and aggressive behaviour in children may be linked to variation in the levels of CU traits within and between samples.

Furthermore, the high heritability of CU traits and their association with more chronic and serious aggression and antisocial behaviour problems make them a strong candidate for the driving force behind the familial transmission of aggressive behaviour that Halperin et al. [38] argue is mediated, in part, by reduced central serotonin function.

They use observational measures to score guilt (in breaking a valued object situation), empathy (hurting the parent during play) and fearlessness in toddlers and preschoolers, and related these findings tolater parent - rated measures of CU traits and externalizing behaviours during scholar and pre-adolescent age.

The DSM - 5 [5] includes subtyping of conduct disorder considering the presence of CU traits on the basis that these traits identify a group of children with severely disordered conduct [6].

In all, the results from this study suggest that the function of serotonin - system genes is important in the aetiology of CU traits.

Viding, Fontaine, Oliver, & Plomin [56], in their study on monozygotic twins, found that negative parental discipline operates as a non-shared environmental risk factor for developing CP during the transition to early adolescence, but not for the development of CU traits.

We also expect significant stability of the CU traits.

The same statistical procedure (GLM and logistic regression into the CS system) also assessed the specific contribution of CU levels for the subsample of children diagnosed with ODD at baseline (n = 61).

It should be noted that heightened levels of aggression and antisocial behaviour do not demarcate high levels of CU traits.