In fact, in another study published last year, National Institute on Aging (NIA) researchers discovered that people with what's called a CR1 gene variant — the presence of which heightens Alzheimer's disease risk — had much lower levels
of amyloid protein compared with those without the mutant gene.
Not exact matches
In a culture dish microglia that were modified to make a lot
of TREM2 gobbled more
amyloid and removed more dying neurons,
compared with microglia having less
of the
protein.
These results indicated that, under our experimental conditions, other amyloidogenic
proteins do not induce significant accumulation
of IAPP
compared with
amyloid aggregates composed
of IAPP.