The cause
of calcinosis circumscripta is unknown, but there is often a genetic component.
Not exact matches
Chefetz stayed at the Israeli Institute
of Technology for her PhD, but decided to switch to more clinically oriented research and joined dermatologist Eli Sprecher's lab to study familial tumoral
calcinosis (FTC).
A key nutrient is vitamin K2 — Dr. Price's famous Activator X. I love this study on vitamin K2: The Effect
of Vitamin K2 on Experimental
Calcinosis.
Here is an extensive treaty
of the research: https://www.seleneriverpress.com/historical/wulzen-calcium-dystrophy-syndrome-in-guinea-pigs/ Treatment
of 10 cases
of scleroderma and
calcinosis with sources and concentrates
of the anti-stiffness factor indicates only a feeble beneficial effect on scleroderma and none at all on
calcinosis.
Another condition
of the skin that may be observed is called
calcinosis cutis, in which calcium deposits occur within the skin.
If
calcinosis cutis is the problem it will be necessary to try to adjust the dosage
of mitotane (Lysodren Rx) to better control the Cushing's disease or to consider switching to one
of the other treatments.
Yet this is not without risk considering the recent recall
of pet food containing toxic levels
of Vitamin D that caused systemic
calcinosis in cats.
Calcinosis cutis due to overactivity
of adrenal hormones may be associated with loss
of body muscle and swelling
of the abdomen due to laxity
of the muscles.
Calcinosis circumscripta is deposition
of calcium at bony prominences or in the footpads and mouth.
Calcinosis circumscripta is a disease
of large dog breeds.
Calcinosis circumscripta is inclined to local recurrence, especially in the tongue, but the lesions usually cease development at maturity (approximately two years
of age).
Calcinosis cutis is most common in connection with adrenal hormone problems, including those
of treatment with the hormones.
A common side effect
of Cushing's is hair loss, which can then cause a side effect, such as
Calcinosis Cutis.