Sentences with phrase «of cellular calls»

But everything appears to be up to the sadly low standards of cellular calls.

and the metadata of every cellular call I've ever made, including time and duration pic.twitter.com/Ykr3o 0gZFu

Not exact matches

The police made the discovery after a 17 - year - old girl escaped the house in Perris, about 70 miles east of Los Angeles, and used a cellular phone she had found in the house to call them, the Riverside County Sheriff's Office said in a statement released online.

Founded in 2017 and based in Emeryville, California, Finless Foods is one of a number of startups using so - called cellular agriculture to replace age - old methods of food production: farming, animal husbandry, and, in this case, fishing.

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If the device is connected to a Wi - Fi network, Google will route calls and data to Wi - Fi to further reduce the use of more expensive cellular signals and help customers save up on data costs.

This aura is our spirit and when death occurs, it is called back to its» origins within our cellular embodiment to live out its» remainder of life.

Given a system or composite of individuals, all on a comparable level, for instance molecular or cellular, then in some cases, given integration of activities (thus molecules in a cell, or cells in a vertebrate animal), there may be what Leibniz called a «dominant monad» or what Whitehead calls a «society of presiding occasions.»

A NYC - based reader call in to report that he had received a mysterious robocall today on his cellular phone that appeared to be from Gov. Andrew Cuomo's corruption - busting Moreland Commission and name checked a Bronx assemblywoman whose dubious slot machine winnings have been making headlines of late.

Growing demand for cellular data by smartphones, tablets and other devices has led wireless carriers across the country to deploy a new kind of wireless transmitter called «small cells» to provide better coverage in densely populated areas.

To help fix this, a team led by Elizabeth Belding at the University of California, Santa Barbara, designed a cheap, local cellular network called Kwiizya — which means «to chat» in Tonga, the native language in Zambia's Southern Province.

Degenerative brain diseases like mad cow disease (officially known as bovine spongiform encephalopathy, or BSE), scrapie in sheep, and vCJD in humans are thought to be caused by prions, misfolded versions of a normal cellular protein called PrPC.

Now 24, he is a first - year graduate student in the department of cellular and structural biology at the University of Texas Health Science Center in San Antonio (UTHSCSA), where he is studying the role of oxidative damage — the wear and tear inflicted upon the cell by toxic molecules called free radicals — in the aging process.

They all use RNA molecules as messengers to transfer the information from DNA to cellular factories called ribosomes, which then build proteins, which in turn drive our metabolisms and form the structures of our cells.

The gene signature identified by Hancock and his colleagues relates to a special type of immune suppression called cellular reprogramming and suggests that treating inflammation in sepsis is a bad idea.

Called mTOR in mammals, for the term «mammalian target of rapamycin,» this pathway has a critical evolutionary value — it helps an organism avoid too much cellular expansion and growth when energy supplies are insufficient.

When cells become cancerous, tremendous stresses are placed on the cellular machinery responsible for maintaining protein equilibrium — and that machinery is the target of anti-cancer drugs called proteasome inhibitors.

And the first thing that begins to happen is carbon dioxide builds up inside the body, and with it there is a rise in acidity; that acidity rise contributes to cellular membranes decaying and then collapsing and then digestive enzymes that were already always present in the cells begin to slosh around to the body and [it] begins a state of what's called self digestion, so the body begins to liquefy inside rather literally.

And last is the so - called dry stage, which is stage four, from days 51 to 64 or so where tissue is all removed and the bones then begin their own process of cellular decay and decomposition.

New findings published in the Proceedings of the National Academy of Sciences showed in lab studies that supplementing an epigenetic cancer drug called decitabine with vitamin C enhanced the drug's ability to impede cancer cell growth and trigger cellular self - destruction in cancer cell lines.

In a process called cellular reprogramming, researchers at Icahn School of Medicine at Mount Sinai have taken mature blood cells from patients with myelodysplastic syndrome (MDS) and reprogrammed them back into iPSCs to study the genetic origins of this rare blood cancer.

Researchers did not understand what normally kept ras genes» protein production in check — and why certain ras mutations led to such chaotic, uncontrolled cellular activity — until the discovery of a snippet of genetic material called microRNA (miRNA).

But cellular immunity involves other players — such as macrophages, the network of chemical messengers called cytokines, and so - called natural killer cells — that have received scant attention.

Like removing unnecessary lighting from a financially strapped household to save on electricity bills, the drug — called rapamycin — prolongs the survival of diseased neurons by forcing them to reduce protein production to conserve cellular energy.

This so - called microRNA disrupts the expression of two key cellular genes called TGF - β and SMAD3, in a process known as RNA interference (RNAi).

A forward genetic screen in Drosophila melanogaster (fruit flies) identified mutant copies, or alleles, of a gene called cacophony associated with defects in autophagy and cellular homeostasis.

Called 6 diazo -5-oxo L norleucine, or DON, the drug blocks the cellular use of the protein building block glutamine.

But a string of recent discoveries indicates that a class of RNA molecules called small RNAs are actually in charge of many cellular functions.

Proteins called kinesins, for example, are natural nanomotors that support cellular functions such as mitosis (the chromosomal process that creates two nuclei from one parent nucleus) and meiosis (when the number of chromosomes per cell is reduced by one half).

Many of those efforts have focused on drugs that target cellular receptors in the brain called NMDA receptors.

The Brain Trauma Indicator measures blood plasma levels of the UCH - L1, a protein scientists believe helps dispose of cellular waste in the brain, and GFAP, a structural protein found in non-neuronal cells called astrocytes.

This breakdown of laminB1 and other nuclear material leads to a cellular state called senescence, or literally «getting old.»

Most important of all seem to be the tiny powerhouses called mitochondria that fuel cellular development.

They found that by attaching to a mitochondrial protein called TOM20, alpha - synuclein prevented the mitochondria from functioning optimally, which resulted in the production of less energy and more damaging cellular waste.

SIK3 phosphorylates and activates a key enzyme of glucose metabolism called Glucose -6-phosphate dehydrogenase G6PD, which is an important regulator of cellular redox balance.

Led by Dr. Peter Glazer, chair of therapeutic radiology, Dr. Mark Saltzman, chair of biomedical engineering, and Dr. Marie Egan, professor of pediatrics and of cellular and molecular physiology, the collaborative team used synthetic molecules similar to DNA — called peptide nucleic acids, or PNAs — as well as donor DNA, to edit the genetic defect.

Researchers looking at the white blood cells called eosinophils found a new weapon in the immune arsenal: toxic webs of DNA flung in less than a second by a kind of cellular catapult.

The controversy began in December 2010, when NASA astrobiology fellow Felisa Wolfe - Simon and colleagues described online in Science a microbe called GFAJ - 1, which grew, albeit slowly, in the presence of arsenic, leading the authors to conclude the bacterium had taken up the toxic element and incorporated it into its cellular components.

Previous studies in the lab showed that once HCMV is inside the cell, it quickly becomes latent by entering the cell's nucleus and co-opting a cellular protein called Daxx — part of the intrinsic immune system — to shut down its own replication, the process of reproducing its genetic material to make more copies of itself.

Functional measurements of the cells showed that a drug called avanafil reversed some of the cellular imbalances caused by the mitochondrial defect.

This insulation, called myelin, is made up of numerous layers of cellular membranes — seen in cross section, an axon insulated in this way resembles the rings of a tree.

This cycle of activation, production and stop / block goes around once a day and is also influenced by a compound called FBXL3, which flags CRY for degradation by cellular enzymes.

Some of these molecules are a complex of proteins called the Polycomb Repressive Complex 2 (PRC2) that is involved in «silencing» genes so that they are not «read» by the cellular machinery that decodes genetic information, effectively keeping the genetic information in the «off» state.

This association of the RNAi machinery and a gene's message is prelude to destruction of the message before it arrives at a cellular protein factory called the ribosome.

That is when studies finally convinced scientists that the minuscule RNA snippets they had taken to calling «microRNA» were regulating cellular and genetic processes throughout the human body and were critical factors in the determination of health and disease.

But they are daring again with growing confidence, buoyed by new insights and technologies to fight a foe that Jay Levy, codiscoverer of HIV, compares to a «biological Trojan horse» and Jay Lalezari calls «a cellular bioterrorist that kills your first responders first.»

These small signaling molecules, called cytokines (SIGH - toe - kines), spur a cascade of cellular reactions that help the body fight infection.

Because of that, many different components are present in minute quantities, which inevitably leads to random variations in cellular processes which are also called «noise» by chemists.

The new enzyme converts A to a base called inosine, or I. Either a cellular repair mechanism or the process of the DNA copying itself then changes the I to a G. «The big deal here is engineering the TadA enzyme to do something fairly unnatural,» says George Church of Harvard, who studies CRISPR.

Bex acts on a «master regulator» protein called retinoid X receptor (RXR), which controls a network of other cellular proteins.