«But we discovered that attaching microbeads to the cell's surface amplifies the cell's response during cavitation and produces the same result with much less risk
of cellular injury.»
Not exact matches
They found subtle evidence
of axonal
injury at the
cellular level in six athletes who had not been diagnosed with concussion but sustained RHI during the normal course
of play.
Some autopsy data shows that infants
of smokers have signs
of established hypoxic - ischemic
cellular injury in the brain and the heart which probably occurred in antenatal life, may have been caused by suboptimal placental function and may have been sub-clinical, but if the baby continues to be in a vulnerable environment exposed to post-natal passive smoking, this could affect autonomic nervous system function and lead to poor temperature control, and poor heart rate and respiration control.
The affected mitochondria could no longer provide the synapses with enough energy, which ultimately prevented the synapses from functioning — providing the first direct link between
cellular injury caused by amyloid protein and the characteristic breakdown
of neuronal communication that occurs in Alzheimer's patients.
A new kind
of cellular alchemy may allow cells to be converted into other tissues to treat
injuries.
«We came up with a list
of potential
cellular candidates that could be compensating for the loss
of these specific macrophages and used several different tests to determine which cells were clearing away the nerve debris after
injury,» Lindborg said.
Known to help restore ATP and maintain
cellular energy, creatine is being investigated to treat a number
of neurological conditions — including Parkinson disease, amyotrophic lateral sclerosis and spinal cord
injury.
«Proper blood cell production is dependent on functioning hematopoietic stem and progenitor cells that are destroyed during conditioning procedures for transplantation or following bone marrow
injury,» said the study's first author Kevin A. Goncalves, who performed this research as part
of his PhD studies in
cellular and molecular physiology at the Sackler School.
They are recruited to sites
of injury or infection and there turn into macrophages (literally «large eaters») that ingest pathogens, infected cells, or
cellular debris.
Previous research has described at least some
of the fundamental processes involved in healthy, on - going peripheral nerve growth regeneration, including the critical role
of mitochondria —
cellular organelles that produce adenosine triphosphate (ATP), the energy - carrying molecule found in all cells that is vital to driving nerve recovery after
injury.
Recent research suggests one benefit
of environmental enrichment at the
cellular level: it repairs brain myelin, the protective insulation surrounding axons, or nerve fibers, which can be lost because
of aging,
injury or diseases such as multiple sclerosis.
He and his colleagues at the University
of Wisconsin — Madison have begun to explore how fruit flies could help uncover the
cellular mechanisms behind traumatic brain
injury (TBI) in humans.
Topics such as the mechanisms
of cell
injury in normal and dystrophic muscle, compensatory muscle regeneration and hypertrophy, and the effects
of various therapies or voluntary exercise on muscle repair, satellite cell activation, muscle growth, bone density and age - related atrophy are examined using a large variety
of cellular, molecular and whole - animal in vivo assays
of function.
Most
of the discussion section at the end
of the paper focuses on the complexity
of the molecular and
cellular mechanisms involved with vascular
injury, especially in very advanced lesions.
The central nervous system damage seen with prenatal ZIKV infection is likely due to direct
cellular injury, as ZIKV RNA15, 17,32 and live virus15 have been identified in the brain tissue
of infants with microcephaly.
Rhamnazin has also shown protective properties against inflammation in acute lung
injury, inhibiting the metabolism
of cytokines, which are proteins secreted by the immune system in order to affect
cellular functions.
Also noted by IER studies are an increase in the expression levels
of silent mating type information regulation 2 homolog 1 (SIRT1), an NAD + - dependent deacetylase.20 The expression
of SIRT1, also increased by prolonged ER in rodents, is linked to the up - regulation
of cellular stress resistance and improved outcomes in animal models
of metabolic, neurodegenerative and inflammatory diseases.106, 107These findings have been suggestively accompanied by improvements in resilience to disease progression in rodent models
of Type 1 diabetic nephropathy 20, survival following induced ischaemic
injury 21 and a reduction in oxidative stress.105
Bananas also contain small amounts
of beta - carotene, which gives bananas with their yellow color and provides an additional source
of vitamin A. Collectively, these vitamins support a number
of physiological processes; vitamin A promotes healthy
cellular growth; vitamins C and E serve as antioxidants that help prevent disease; vitamin K helps your body form blood clots in response to
injury; and the B - vitamins support your metabolism.
Rooted in her experience as an elite athlete who used traditional foods to cure her own debilitating
injuries, and combining her research with the latest discoveries in the field
of epigenetics, Dr. Cate shows how all calories are not created equal; food is information that directs our
cellular growth.
Bee (Hymenoptera) envenomation may include stings from honeybees, bumblebees, yellow jackets, hornets and wasps — and the ASPCA Animal Poison Control Center warns that components
of venom can create
cellular injury, elicit an inflammatory response, trigger histamine release and act as a possible allergen.
The reparative phase occurs within 3 to 14 days after
injury and is characterized by the
cellular response
of fibroblasts and endothelial cells.
The departing blood will also carry away deleterious by - products
of injury and bring in fresh
cellular nutrients, allowing cells to rejuvenate faster.
Today it is very easy to take photographs
of a damaged car, an
injury or an accident scene at any location and at any time because most people have camera features in their
cellular phones.
The courts must decide at what point the
injury which is mesothelioma «arises»: is it at the point where a victim is exposed to asbestos, when no perceptible damage is evident, or at the point when
cellular mutation — the onset
of disease — actually «begins» in fact, or when it becomes symptomatically manifest?