Sentences with phrase «of cellular oxidation»

Supplying antioxidants for your pet can help by neutralizing the harmful by - products of cellular oxidation.

The study authors concluded that the Mediterranean diet plus CoQ10 «improves oxidative DNA damage in elderly subjects and reduces processes of cellular oxidation.

Not exact matches

In schizophrenia, excessive oxidation — which involves the same type of chemical reaction that causes metal to corrode into rust — is widely thought to cause inflammation and cellular damage.

«We are trying to understand the causes of aging by linking the mechanisms that control the oxidation of proteins at the cellular level with the mechanisms that determine the lifespan of the whole organism,» Apfeld says.

In this case, he was testing the idea that a buildup of cellular damage caused by oxidation — technically, the chemical removal of electrons from a molecule by highly reactive compounds, such as free radicals — is the main mechanism behind aging.

In another application, the sensor was used to study how cells respond to the introduction of extracellular hydrogen peroxide, which produces a wave of oxidation as it moves through the cellular structures.

We are looking for candidates interested in combining experimental and theoretical approaches to study the regulation of protein oxidation at molecular and cellular levels.

The Keap1 - Nrf2 system has evolved to respond to intracellular oxidative stress; in particular the generation of reactive electrophiles produced from oxidation of endogenous cellular constituents as well as xenobiotics [2]--[4].

A comforting mug of rooibos helps combat harmful oxidation — a process linked to cellular aging and disease — thanks to its generous amount of free radicals.

Stress resistance has not been assessed however and so the biological relevance of this finding is currently unknown.32 Several IER trials (75 - 85 % ER on restricted days) in overweight / obese populations have reported reductions in various markers of oxidative stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directions

Is a key component of various proteins, as well as enzymes that catalyze cellular oxidation reactions

When we are young and have high levels of NAD +, it is mostly consumed as part of normal cellular respiration process — that is, oxidation of ATP for energy.

As an antioxidant, it helps to protect mitochondrial and cellular membranes and to inhibit the oxidation of LDL cholesterol.

Similar to the physical and cellular burn of exercise that creates some oxidizing free radicals, the «stress» from cruciferous vegetables stimulates our mitochondria to produce homemade antioxidants to counterbalance the oxidation and protect our vital DNA.28 Even cancer cells have caught on to this central role of our mitochondria — the bastards can amplify their levels of some of these same pathways — further stressing their importance for survival.25 However, our cells still seem to have the upper hand, as multiple studies show that the organosulfur chemicals in many of these anticancer vegetables can severely stress cancer cells, leading to oxidative damage, and ultimately, death.29

Research has shown a link between mercury fillings and leaky gut syndrome, malabsorption of essential minerals and essential fatty acids, blocked cellular enzymatic processes related to the ATPASE energy function and sulfur oxidation.

This process of oxidation disrupts cellular chemistry eventually causing cells to die.

Most scientists agree that this cellular oxidation is the primary cause of premature aging.

Over time this leads to nutritional deficiency, cellular oxidation, and a dangerous build up of toxins.