Sentences with phrase «of cholesterol particles»

In fact, the diet should be approximately 70 % of calories from unadulturated fats like low carb nuts (pecans and macadamias are great, almonds ok and peanuts and cashews are considered higher carb on the nut scale), avocado, grass fed butter, coconut oil, olive oil; and the remainng 15/15 for protein and non-starchy vegetable carbs, especially nutrient dense leafy greens It is carbohydrates or high protein leading to gluconeogenesis in the diet that make concurrent consumption of fats a cardiovascular risk, but in a properly carb - restricted and moderate protein diet, and in the absence of systemic inflammation (hsCRP, ESR), one should not worry about increases in cholesterol, but focus on the size of the cholesterol particles (bigger is better) Dr. Peter Attia explains this complex topic well.

We run the NMR lipid test which looks at the size and number of the cholesterol particles in your blood, rather than just the amount of cholesterol.

I saw «The Last Heart Attack» and I was wondering what you thought about what Dr. Arthur Agatston said in the show, as he's the inventor of the South Beach Diet, I'm wondering how credible his idea that the size of cholesterol particles and your cholesterol number doesn't matter.

Not exact matches

To mimic this versatile substance, Joke Bouwstra and Robert Rissman at Leiden University in the Netherlands mixed a range of fatty compounds including lanolin, fatty acids, ceramides and cholesterol with particles made of a water - storing hydrogel (International Journal of Pharmaceutics, DOI: 10.1016 / j.ijpharm.2009.01.013).

In individuals with high HDL - cholesterol levels in the circulation, the lipid composition of the HDL particle was more beneficial regarding heart disease risk.

LDL particles transport cholesterol to the organs of the body from the blood stream.

In individuals, whose HDL - cholesterol levels were low, the quality of HDL particles was also impaired; they contained smaller amounts of lipid molecules which are known to be antioxidant and thus protective to arteries.

It is likely that molecules other than cholesterol — the lipid and protein molecules on the surface of the HDL particle — are responsible for the protective effects of HDL particles against heart disease.

Men are also reported to have smaller low - density lipoprotein particles than women, whereas men and postmenopausal women have higher levels of serum cholesterol and triglyceride sub-classes compared with premenopausal women.

Her paradoxical case has helped motivate a team of scientists to show how high «good cholesterol» can sometimes be a signal not of heart health, but of the opposite: a cholesterol system unable to siphon the fatty particles from circulation.

Statins, prescribed to lower cholesterol and reduce risks of heart attacks and strokes, seem to diminish inflammation that occurs after people breathe airborne particles

For example, a chronic inflammatory disease such as gout or arteriosclerosis may be triggered by a very specific interaction of a particle (uric acid crystals, cholesterol crystals, amyloid plaque,....)

Another area focuses on the HDL particle (carrier of good cholesterol in the blood).

Now a team of scientists, led by Mary Jo LaDu in the department of pathology at the University of Chicago Medical Center, has shown that the apoE - containing fat transporters found in brain cells are very different from the apoE particles found elsewhere in the body and contain most of their cholesterol in a different form.

The differences between the lipoproteins secreted by astrocytes and those found in the CSF suggest that the astrocytes produce particles that help the brain rid itself of excess cholesterol in addition to helping deliver membrane components to the nerve cells.

The tiny particles are 1,000 times smaller than the tip of a human hair, and are designed to latch on to atherosclerotic plaques — hard deposits made from accumulated fat, cholesterol and calcium that build up on the walls of arteries and are prone to rupture, producing dangerous clots.

According to another study, apple cider vinegar contains the antioxidant chlorogenic acid, which protects the particles of the LDL cholesterol from becoming oxidized, thus reducing the risk of heart disease.

The LDL - particle number and the small dense LDL - particles fell toward normal with the addition of the avocado to the diet — both favorable results for people with high cholesterol.

According to the Cooper Institute, a good way to determine what kind of LDL particles you have is to find your ratio of triglycerides to HDL cholesterol.

It also transports cholesterol from extra-hepatic sites, including the arterial wall, to the liver for excretion via reverse cholesterol transport.77 Results from IER (60 - 85 % ER / alternate days) trials have thus far been inconsistent, with some reporting decreases 40, and others increases 37, 39, but with the majority showing no effects on HDL cholesterol levels.38, 42, 43, 45, 49, 51, 52Inconsistencies may have arisen due to the biphasic response of HDL documented by studies of ER induced weight - loss, whereby levels typically decrease during active weight - loss then either return to baseline or (less commonly) rise following attainment of weight stability.78 Complementary increases in HDL levels and particle size have been shown when endurance exercise have been combined with IER 42, 46, 47 however further discussion goes beyond the scope of this review.

So, for those you listening in, just to kinda bring this full circle here briefly, high cortisol, in the case of high cortisol, one of the things that Dr. Bryan recommended was to look at things like your cholesterol particles and your HDL, to look at things like your testosterone, to look at things like the ACTH that we talked about, and some of these other variables that can affect ACTH, and then of course, something like an oxidative stress panel.

Another thing about cholesterol: even though higher LDL numbers are seen as risky, the type of LDL particles you have shuttling through your arteries is most important.

It seems fairly clear that those on keto and very - low - carb diets who get most of their energy from fat — including their own fat stores — tend to have more LDL particles circulating in their bloodstream, delivering triglycerides to cells and transferring cholesterol to other lipoproteins.

This way you can raise your HDL cholesterol («good» cholesterol), lower your triglycerides and reduce levels of small particle LDL cholesterol («bad» cholesterol).

Increased enterohepatic circulation on high fat means that cholesterol is kept «in play» - bounced back into the bloodstream in ApoB particles - while low enterohepatic circulation, in people with with higher synthesis rates, during weight loss - when cholesterol is being dumped by shrinking cells - means that cholesterol can pile up in the gall bladder faster than it can be conjugated to bile salts and bile acids and faster than it can be extracted by the weak stimulus of low fat food.

In a 2010 study published in the American Journal of Clinical Nutrition, the researchers found that when you replace saturated fat with a higher carbohydrate intake, you increase insulin resistance and obesity, and increase your chances of getting heart disease by increasing triglycerides and small LDL particles, and reducing beneficial HDL cholesterol.

We are just starting to understand how the body carries cholesterol to and from cells in the form of LDL, VDL, and HDL particles.

The type of cholesterol (LDL particle size) is the more important factor in regards to cholesterol consumption.

The smaller the particles, and the more of them, the more likely you are to have heart disease and plaque, even if the total cholesterol level is low.

It is important to understand that cardiovascular disease is not just an illnesses that is associated with having high blood fats (high LDL cholesterol and an abnormal distribution of LDL particle size), but it is also an inflammatory condition.

«The results indicate that when the cholesterol and particle measures of LDL disagree, the clinical and subclinical outcomes track with LDL - P more so than with LDL - C» http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3070150/

The idea that small LDL particles are the only bad ones and large LDL is benign is just something made up by people selling low carb diets as a way to excuse one of the biggest flaws of the diet, which is that it raises your LDL cholesterol and consequently your risk of developing heart disease.

LDL or «bad cholesterol» measures the amount of cholesterol in the LDL particles but not the number or size of the particles.

Increased glycated ApoE particles have been detected in the cerebrospinal fluid (CSF) of AD patients.34, 35 The physiological insult of glycated ApoE is that ApoE helps transport LDL particles (and their critical cholesterol and fatty acid passengers) across the blood brain barrier.

And, naturally, the drug companies don't want you to know that part of the science because it would severely limit the number of people going on cholesterol - lowering drugs, since statins do not modulate the size of the particles.

Glucose / HBA1C Free - ranging glucose molecules in your bloodstream can adhere to cholesterol particles and cause those particles to remain in the bloodstream for long periods of time, since your liver can't properly process cholesterol when it has a glucose molecule attached to it.

Siri Tarino stated, «However, replacement of saturated fat by carbohydrates, particularly refined carbohydrates and added sugars, increases levels of triglyceride and small LDL particles and reduces high - density lipoprotein cholesterol, effects that are of particular concern in the context of the increased prevalence of obesity and insulin resistance.

What matters is whether the cholesterol and fat residing in those LDL particles have been oxidized... «Cholesterol has nothing to do with heart disease, except if it's oxidized,» Dr. Kummerow said... [He] contends that the high temperatures used in commercial frying cause inherently unstable polyunsaturated oils to oxidize, and that these oxidized fatty acids become a destructive part of LDLcholesterol and fat residing in those LDL particles have been oxidized... «Cholesterol has nothing to do with heart disease, except if it's oxidized,» Dr. Kummerow said... [He] contends that the high temperatures used in commercial frying cause inherently unstable polyunsaturated oils to oxidize, and that these oxidized fatty acids become a destructive part of LDLCholesterol has nothing to do with heart disease, except if it's oxidized,» Dr. Kummerow said... [He] contends that the high temperatures used in commercial frying cause inherently unstable polyunsaturated oils to oxidize, and that these oxidized fatty acids become a destructive part of LDL particles.

Studies have linked them to improvements in LDL cholesterol and particle levels, lower blood pressure, and reduction of inflammatory markers (23, 24, 25, 26, 27).

Furthermore, positive associations between TSH and LDL as well as total cholesterol levels have been found in cross-sectional studies in euthyroid healthy subjects, and the strength of these associations seems to depend on an individual's insulin sensitivity.We therefore hypothesize that the KD has diminished the production of T3 from T4, thereby reducing the number of LDL receptors and thus reducing LDL particle clearance which might be further impaired due to the missing stimulating effect of insulin on LDL uptake into cells.

According to research found in Atherosclerosis, Thrombosis, and Vascular Biology, large LDL particles were not found to be associated with an increased risk of ischemic heart disease in men, and that the cardiovascular risk LDL cholesterol does pose is related to levels of its small particles.

What we've learned since then, when it comes to cholesterol health and heart disease risk, is that the relative size and number of LDL particles in the bloodstream matter.

As they circulate in the bloodstream they release little particles of fat and cholesterol that are utilized by the cells or stored as body fat.

The results from this trial clearly indicate that the replacement of refined carbohydrates with EPRO and UFA increased insulin sensitivity, LDL peak particle size, and lowered fasting TG and VLDL cholesterol concentrations in men and women with an elevated TG concentrations.

Some say its a numbers game, the more LDL particles crashing the walls of the arteries... some say the LDL is only a sign of vascular damage, but if my LDL goes down on a low fat diet, then that is bs, some say that it may be a problem metabolizing LDL, intake / production is outpacing LDL receptor activity, some say its not LDL but LDL that stays in the blood too long and oxidizes, some say about 20 % carbs (I was less than 5 %) will produce just enough insulin to help metabolize cholesterol, but the hard core low carb guys, say the whole cholesterol thing is a scam and cholesterol under 500, without insulin resistance is nothing to worry about.

The results of this study also show that theEPRO / UFA condition reduced TD and VLDL cholesterol concentrations and increased LDL particle size compared with the refined - carbohydrate condition, indicating improved cardiovascular health.

Lipoprotein lipid profile disturbances associated with an elevated TG level include low High Density Lipoprotein (HDL) cholesterol and HDL particle (HDL - P), and a predominance of small dense Low Density Lipoprotein (LDL) and LDL particle (LDL - P).

We also have the testing to evaluate the FUNCTION of your LDL particles, not just the total amount of LDL cholesterol.

The study also reported improvements in other cardiovascular and metabolic variables such as triglycerides, LDL - cholesterol particle size, and C - reactive protein, but it did not correct for multiple comparisons (a common scientific design problem of many fasting studies)(36).

Soluble fiber can reduce both «bad» LDL and overall cholesterol, perhaps by binding with cholesterol particles in your digestive system and moving them out of the body before they're absorbed.