Alpha activity is associated with attention and processing of sensory stimuli, and this pattern of high EEG alpha reflects a pattern
of cortical maturity.
These include a test of vision; a measure of cognition, language, motor skills, behavior, attention and emotional reactivity; a test
of cortical auditory processing and a measure of sleep patterns and daytime physical activity.
Thus, positive beliefs associated with optimism, a sense of personal control, and self - esteem appear to help people manage stress by virtue
of cortical regulation of amygdala activity and consequent lower downstream neuroendocrine responses.
Modulation
of cortical - limbic pathways in major depression: Treatment - specific effects of cognitive behavior therapy.
We observed greater activation in men, compared to women, in a wide array
of cortical and sub-cortical areas, during stop success (SS) as compared to stop error (SE).
In summary, when considering associations between experience and brain morphometry, cortical thickness and surface area should be assessed separately, rather than reporting on the composite metric
of cortical volume (Winkler et al., 2010; Raznahan et al., 2011).
The infant is a «subcortical creature... [who] lacks the means for modulation of behavior which is made possible by the development
of cortical control» (Diamond, Balvin and Diamond, 1963, p. 305).
If now a new and different stimulus reaches another set
of cortical arrangements, the threshold will be much higher than would normally be required for excitatory processes to develop into conscious processes and this new stimulus will have no observable effect.
Because of the effective use of the ketogenic diet in reducing seizures (see above), it has been suggested that it may also improve the clinical status in brain injury, especially by reducing the incidence of long - term consequences, such as epilepsy.79 Positive effects of a ketogenic diet have also been reported in reducing the cortical contusion volume in an age - dependent manner in an animal model
of cortical injury, which is related to the maturation - dependent variability in brain ketone metabolism.92 These findings were also supported by the demonstration that a ketogenic diet reduced post-traumatic cognitive and motor function impairment, at least in a rat model.93
Age - dependent reduction
of cortical contusion volume by ketones after traumatic brain injury.
And studies using functional MRI measures have shown that the amount
of cortical brain area used is greater during eccentric contractions than in concentric contractions (Yao et al. 2014), while functional connectivity between the primary motor cortex and other parts of the brain is reduced (Yao et al. 2016).
There is a dramatic increase
of cortical surface area during late fetal development as a result
of cortical folding, while there is a dynamic change
of cortical thickness during the entire life span as a result of development and disease.
Much of these studies have relied on voxel - based morphometry, providing estimates
of the cortical volume of the brain.
The most significant outcome of the research was that cortical abnormalities which take place in individuals with migraine are a consequence of the balance between an intrinsic predisposition, as indicated by modification
of cortical surface area, and processes related to disease, as indicated by abnormalities
of cortical thickness.
Surface area abnormalities and cortical thickness were assessed in individuals with migraine, 2 components
of cortical volume providing complementary and different bits of information.
Recently, we extended our imaging methods to the scattering tissue domain and demonstrated fast volumetric calcium imaging across the majority
of a cortical column in the mouse.
Overall, these results demonstrate an endophenotypic impairment in the synchronization
of cortical areas within the parieto - premotor network during somatosensory processing and motor planning in WC patients
Cellular biology
of cortical neurons.
Development
of cortical neural circuits, in particular on activity - dependent remodeling and how to correct mis - wiring in neurological disorders.
Using multichannel electroencephalography (EEG), we investigated temporal dynamics
of the cortical response to transcranial magnetic stimulation (TMS).
These results indicate that cortical regions within rsFC - defined systems exhibit similar thinning patterns across the lifespan, and that structural covariance
of cortical thinning may delineate local cortical distinctions.
Neural synchrony and the development
of cortical networks.
A large number
of these cortical malformations remain unexplained, both because causative mutations have yet to be identified and because the function of some of the identified genes remains poorly characterized.
Differentiation from human pluripotent stem cells
of cortical neurons of the superficial layers amenable to psychiatric disease modeling and high - throughput drug screening.
Rapid development
of cortical auditory evoked potentials after early cochlear implantation.
Given this functional distinction, it is possible that patterns
of cortical thinning exhibit more specific processing - related distinctions of the brain.
The aging brain: morphomolecular senescence
of cortical circuits.
Conclusions: This is the first evidence
of cortical calcification in patients with an APP mutation other than the Iowa mutation.
Results in normal subjects were tested for reproducibility in an independent cohort of 13 patients with depression.Differences in functional connectivity were related to previously reported differences in clinical efficacy across a distributed set
of cortical and limbic regions.
Evidence for similarities and differences in rates of thinning across cortical regions have been made at the level
of cortical lobes and gyral & sulcal landmarks.
Recent functional imaging studies have revealed coactivation in a distributed network
of cortical regions that characterizes the resting state, or default mode, of the human brain.
His current projects include elucidation
of the cortical representation of oculomotor proprioception, using saccadic adaptation to understand the coordinate system of neurons in the lateral intraparietal area, the role of prestriate cortex in visual search, and the role of inhibition in the response of parietal neurons.
Linear metrics (cerebral lobe dimensions and sulcal lengths) and principal component scores
of cortical shape variation in 206 chimpanzees and 218 human brains.
Wide - field fluorescence microscopy combined with genetically encoded fluorescent reporters of neuronal activity allow visualization
of cortical remapping over large areas during rehabilitative training.
The brain's inhibitory systems are essential for controlling the pattern of activity in the cortex, which has important implications for the mechanisms
of cortical operation, according to a Yale School of Medicine study in Neuron.
Electroencephalography / electromyography monitoring recorded no pathologic increase
of cortical excitability or epileptic activity.
These researchers stated that further studies should examine if the AD / FTD - specific pattern
of cortical dysfunction, classified according to the SICI - ICF / SAI ratio identified here, could also provide relevant prognostic information.
Vertex - and atlas - based comparisons in measures
of cortical thickness, gyrification and white matter volume between humans and chimpanzees.
Results on other measures
of cortical reactivity, (e.g., intra-cortical inhibition [ICI]-RRB-, are more divergent.
Title: Role of thalamic projection in NMDA receptor - induced disruption
of cortical slow oscillation and short - term plasticity Author: T. Kiss et al..
Barnes A.P., Lilley B., Pan, A. Plummer L, Powell A., Raines, A, Sanes J.R., Polleux F. (2007) LKB1 and SAD kinases define a pathway required for the polarization
of cortical neurons.
Bortone D. and Polleux F. (2009) KCC2 expression promotes the termination
of cortical interneuron migration in a voltage - sensitive calcium - dependent manner.
Longitudinal MRI study
of cortical thickness, perfusion, and metabolite levels in major depressive disorder.
Over the past three decades, the field has made important progress in the identification of the molecular mechanisms regulating neurogenesis and neuronal migration of these various classes
of cortical neurons.
Neural markers of familial risk for depression: An investigation
of cortical thickness abnormalities in healthy adolescent daughters of mothers with recurrent depression.
Interestingly, we find that that the slowest relaxation timescale of the cortex is similar to turnover times
of cortical cross-linkers.
Early in his career, he discovered that previously enigmatic support cells, known as radial glia, serve as guides for the migration
of cortical neurons in the developing brain, and showed how this process is critical for the organization of the multi-layered structure of the cerebral cortex.
Using correlated 2 - photon and electron microscopy, these have shown how adult neuronal connectivity is far from hard - wired, with dendritic spines playing a significant role in the rapid alterations in the wiring
of cortical circuits.
Regional increases
of cortical thickness in untreated, first - episode major depressive disorder
Polleux F., Giger R.J., Ginty, D.D. Kolodkin A.L. and Ghosh A. (1998) Patterning
of cortical efferent projections by semaphorin - neuropilin interactions.