The chief complaints by owners
of dogs with cataracts are cloudy, white - ish or blue - grey pupils (or spots in the pupil) and impaired vision.
When an ophthalmoscope is used to examine the eye
of a dog with cataracts, the retina at the back of the eye can not be seen clearly.
Not exact matches
One reason the FDA has moved so cautiously is that researchers have detected
cataracts in the eyes
of monkeys,
dogs and rats that were treated
with DMSO, and there seems to be evidence that its use has damaged fetuses in hamsters.
These ways are
with: Diet — eat more fruits and vegetables daily, including: foods rich in Vitamins A (leafy green vegetables), C (peppers, citrus fruits, berries, tropical fruits, broccoli and tomatoes), and E (almonds, spinach, wheat germ and sweet potato), Zinc (grass - fed beef, kefir, yogurt, chickpeas and pumpkin seeds); Lutein and zeaxanthin (spinach, kale and broccoli, and eggs), fish and omega 3 — eating fish 3 times a week is in total co-relation to
cataract health and can lower the risk
of cataracts; Supplements (it's preferable to get your nutrients from food, but it's not always possible) such as bilberry which is used traditionally to help protect against
cataracts, glaucoma, and macular degeneration; Sun protection — make sure to wear eye protection whenever out in the sun to help reduce the risk
of eye health issues; Lifestyle modifications — smoking and drinking are known health risks, but also for the eyes; and the possible upcoming Eye Drop intervention — drops containing Lanosterol have been tested on 3
dogs that cleared their vision after 6 weeks
of using these drops — unfortunately, it's not yet available for human use at this time.
The success rate
of uncomplicated
cataract surgery
with intraocular lens implantation in
dogs is approximately 95 % or, in other words, 95 out
of 100 eyes will have functional vision after surgery.
There has been no evidence that
cataracts correlate
with coat color in any breed
of dog.
A
dog with posterior punctate
cataracts might be in the early stages
of hereditary disease and should have the DNA test for the HSF4
cataract mutation.
From left to right, these photos show an actual clear
dog lens, a mature
cataract, and a
dog eye following
cataract surgery
with placement
of a clear artificial replacement lens.
Dogs with two normal copies
of the gene will not develop this type
of cataract.
The mode
of inheritance for most Aussie
cataracts is dominant
with incomplete penetrance, meaning not every
dog with the mutation will develop
cataracts though 70 %
of those
with cataracts have it.
Dogs with one or two copies
of the mutation have a heightened risk for developing
cataracts.
The majority
of dogs with diabetes mellitus will develop
cataracts within 5 - 6 months from the time
of diagnosis
of the disease.
Some hereditary
cataracts generally appear at an early age, and while they may or may not interfere
with the
dog's vision, some do progress into loss
of vision.
There are many types
of genetic eye conditions, including
cataracts, retinal dysplasia and retinal degeneration which occur
with a higher incidence in purebred
dogs.
Usually when we think
of cataracts we commonly associate them
with Geriatric
Dogs.
The risk
of this complication has been decreased by placing a prosthetic lens (a formerly uncommon but now fairly standard procedure) but
dogs who start off
with hypermature (dissolving)
cataracts or have an unusually long surgery time tend to have an increased risk for this complication.
Of the 11 dogs with problems, cataracts were found in all cases, nine of which were bilateral and two of which were unilatera
Of the 11
dogs with problems,
cataracts were found in all cases, nine
of which were bilateral and two of which were unilatera
of which were bilateral and two
of which were unilatera
of which were unilateral.
Kadon P.F., et al., «Topical KINOSTAT ™ ameliorates the clinical progression
of cataracts in
dogs with diabetes mellitus» Vet Ophthalmol Nov. 2010; 13 (6): 363 - 8.
For many years we have «All» had our Flatcoats tested for PRA and
Cataract,
with pass certificates; we now face an uncertain future, it is a worry for us all, until several generations
of our
dogs are tested and more is known.
However, it is also true (in Dr. McCalla's view) that restoring a blind
dog's vision
with cataract surgery is one
of the most satisfying parts
of being a veterinary ophthalmologist, and surgery can give a
dog a wonderful new lease on life.
It is very possible, given the disparity between the numbers
of dogs with the mutation and the frequency
of cataracts that HSF4 positive / no
cataracts families might be possible, but without further study we can not be sure.
Not every small
cataract will advance, which is why the American College
of Veterinary Ophthalmologists gives
dogs with punctate
cataracts a passing exam report.
Dogs with these generalized
cataracts are unable to distinguish anything but extremes
of light and dark.
The HSF4 test, which targets the mutation associated
with 70 %
of our breed's inherited
cataracts, was released in 2008 and has been widely used but will have had little impact on the
dogs in this survey because
of the birth date range.
The degree
of risk varies in genes like this, but
with this particular mutation it is very high: A
dog with it is 17 times more likely to get
cataracts than one that doesn't have it.
Hyaloid arteries are not considered a hereditary problem, however their association
with cataracts and the frequency
with which they occurred among
dogs which were part
of the CEA study in the early 1990s is reason for concern.
Dogs with the mutation and at least four years of age (to have some assurance that they won't have cataracts) should be bred only to clear tested dogs and preference should be given to clear tested offspring to carry on w
Dogs with the mutation and at least four years
of age (to have some assurance that they won't have
cataracts) should be bred only to clear tested
dogs and preference should be given to clear tested offspring to carry on w
dogs and preference should be given to clear tested offspring to carry on
with.
-- First - step relatives (parents, offspring, full and half siblings)
of an HSFS4 - clear
dog diagnosed
with hereditary
cataracts should be bred only to HSF4 - clear mates who have no family history
of non-HSF4
cataracts.
A
dog might have a
cataract even though it does not have a mutation
of HSF4, If a
dog has just one copy
of the Aussie mutation can develop
cataracts, and — in what must seem a perverse whim
of the DNA gods — not every
dog that has the mutation has or will get
cataracts, nor every
dog with cataracts have the mutation.
Dogs affected
with this condition will exhibit some combination
of the following: Microphthalmia, eccentric pupils, coloboma or other irregularities
of the iris, lens luxation,
cataract, retinal dysplasia or detachment, persistent pupillary membrane, equatorial staphyloma or lack
of a tapetum.
We should continue to support their effort by submitting samples from
dogs affected
with any type
of cataract.
Based on the author's correspondence and conversations
with Aussie breeders since the release
of the test, their one biggest conundrum is the fact that not all
dogs with the mutation actually have
cataracts.
The mutation was associated
with all types
of cataracts, but the majority
of dogs had bilatera
The mutation is dominant, so
dogs with even one copy are at risk
of developing
cataracts at some point in their lives.
Finally,
dogs with progressive
cataracts, that are not candidates for surgery, can learn,
with help from their owners, to cope and compensate for the loss
of vision
with their other keen senses, such as smell.
Most
cataracts develop
with age, but shouldn't be confused
with nuclear sclerosis, a normal change
of the lens in pets over 7 years
of age, which causes the lens to appear somewhat whiter or grayer but does not seem to impair the
dog's vision.
Did you know that the majority
of dogs with diabetes eventually go blind from
cataracts?
Although there is always a risk, the success rate
of cataract surgery for
dogs is extremely high
with proper postoperative care.
We feel that
dogs at 10 years
of age
with no
cataract formation would make a perfect control for our study.
If your
dog is diagnosed
with diabetes mellitus, talk to your veterinarian about monitoring your
dog's eyes for development
of cataracts and prevention
of ocular complications.
Did you know that approximately 80 %
of diabetic
dogs will develop
cataracts within 16 months
of diagnosis,
with the majority developing significant
cataract within 5 - 6 months?
I don't mean the
dogs that are completely blind, but the ones
with the haze
of cataracts over their eyes, or just the more senior ones
with a shorter range
of vision.
In fact, 75 %
of dogs develop
cataracts and blindness in both eyes within 9 months
of being diagnosed
with diabetes.
Like
with human
cataracts,
dogs develop a cloudy white or gray film over the center
of their eyes, just behind the retina.
The problem can occur at an early age, and while
cataracts may or may not interfere
with the
dog's vision, some do progress into severe or total loss
of vision.
Cataracts are opacities within the lens, and usually both eyes of dogs with PRA develop c
Cataracts are opacities within the lens, and usually both eyes
of dogs with PRA develop
cataractscataracts.
In addition to supporting retinal health in
dogs with PRA, specific antioxidant supplementation helps reduce the severity and slow the progression
of toxic
cataracts.
Dogs with PRA usually develop
cataracts that are termed «toxic
cataracts», in the mid to late stages
of PRA.
It is the belief
of Dr. McCalla, however, that a
dog with PRA that is on daily specific antioxidant supplementation supplementation AND still has some viable retinal function at the time that the
cataracts progress, can undergo
cataract surgery.
In fact, 75 %
of dogs develop
cataracts and blindness in both eyes within one year
of being diagnosed
with diabetes.