Sentences with phrase «of endothelium»

It really is such a shame that you did not learn that it is the cholesterol in your body that works to REPAIR INFLAMMATION of the endothelium as a result of man - made chemicals, sugar, processed foods, vegetable oils (e.g. «I can't believe it's not butter»), and a plethora of other stressors in our modern world.

Basically it looks like it gives you an idea of how much of your endothelium do you have left.

Swelling of the endothelium increases vascular resistance by making the channel narrower, eventually affecting all organs.

They enhance the function of the endothelium, the interior lining of blood vessels, which plays a fundamental role in blood pressure.

Glutathione's effects ultimately support the health of the endothelium (vascular lining).

There are multiple factors that can cause inflammation and damage of the endothelium, the layer of cells that form the inside wall of every blood vessel in your body:

Research has shown that curcumin promotes healthy function of the endothelium, the thin layer of cells that creates an interface between circulating blood and the vessel wall.

Taking care of your endothelium is extremely important for your heart and your overall health and well being.

Just look up the relations between whey protein as a glutathione precursor and the prevention of heart disease via antioxidant protection of the endothelium (see Dr Mark Houston, for example).

[9] The endothelium is the blood and heart vessel lining, and dysfunction of the endothelium is linked to most kinds of cardiovascular diseases like high blood pressure, coronary artery disease, atherosclerosis, and chronic heart failure.

May also play a role in the formation of the endothelium that gives rise to the blood brain barrier.

Thus, a relative reduction of endothelium - derived relaxing factors with greater reliance on NO can be expressed.

Judah Folkman Award in Vascular Biology Lecture Insights into the Mechanobiology of the Endothelium Guillermo García - Cardeña, Brigham and Women's Hospital, Harvard Medical School

Role of Cyclooxygenase and Derived Reactive Oxygen Species in ϱ - Kinase - Mediated Impairment of Endothelium - Dependent Vasodilation and Blood Flow after Ischemia - Reperfusion of the Rat Kidney.

Self - recognition of the endothelium enables regulatory T - cell trafficking and defines the kinetics of immune regulation.

A 6 × 7 - mm rectangular piece of endothelium - Descemet membrane complex was obtained using a Grieshaber microsurgical knife and Kelman - McPherson forceps.

Multiple hematoxylin - eosin - stained sections showed the presence of endothelium with intact Descemet membrane and no stromal tissue.

One of the many functions of the endothelium is to help arteries dilate when necessary.

Cortisone helps to enhance the barrier function of the endothelium, preventing vascular leakage and further infiltration of the alveolar space by immune cells.

The dysfunction of endothelium is a common denominator of several cardiovascular diseases, particularly those associated with ischemic events.

The research indicates that early onset of menopausal symptoms is associated with dysfunction of the endothelium, which is the lining of blood vessels.

Red wine stimulates the formation of endothelium - dependent relaxation factors such as nitric oxide, which improve endothelial function in coronary arteries possibly because of the high phenol concentration in red wine.

In experiments conducted on human lung endothelial cells and in mice, the researchers showed that NS1 caused permeability of the endothelium, which lines the walls of blood and lymph vessels.

Not exact matches

Cell Metabolism: Activation of TRPV1 by Dietary Capsaicin Improves Endothelium - Dependent Vasorelaxation and Prevents Hypertension

Wine has been shown to be a promotor of nitric oxide (NO) release in the arterial endothelium under the influence of wine polyphenols and NO is associated with improved arterial wall elasticity, decrease arteriosclerosis, and blood pressure regulation.

With our human gut - on - a-chip, we can not only culture the normal gut microbiome for extended times, but we can also analyze contributions of pathogens, immune cells, and vascular and lymphatic endothelium, as well as model specific diseases to understand complex pathophysiological responses of the intestinal tract.»

The TEER measurement is used to quantify the flow of ions between electrodes and across the tissue - tissue interface made of an organ - specific epithelium and endothelium that is a core component of many of the Institute's human Organ Chips.

In the case of heart disease, Reaven says that high blood concentrations of insulin and glucose can damage the endothelium that lines coronary arteries and set the stage for the formation of plaques.

For the subset of more recent patients, the researchers assessed tumor cell behavior — in particular, cancer cells» ability to cross the endothelium (inner layer) of blood vessels.

Based on studies of other tissues, the genes very well could play an essential role in the health of the corneal endothelium, according to the study authors.

The endothelium is the innermost lining of all blood vessels that interacts with circulating blood.

Corneal sections from a person with Fuchs dystrophy show the presence of ATP1B1 in the corneal endothelium.

Affecting both eyes, Fuchs occurs when cells die in the endothelium, the innermost layer of the cornea, and the body fails to replace them.

As the endothelium layer dies, fluid accumulates in the central or stromal layer of the cornea leading to edema or swelling.

In this case, disruption of stable vascular endothelium disrupts this dormant niche and encourages the formation of a microenvironment that sparks micrometastatic outgrowth.»

The inner cell layer that lines vessels, known as the endothelium, is present in all the blood vessels of the body.

As part of this effort, Dr. Weinstein and his colleagues inserted a gene for a protein that turns green the cells that line the endothelium of selected embryonic veins and in the lymphatic system — the network of vessels through which immune cells travel in the body.

«Previously, we thought that all of the cells in the corneal endothelium were unable to divide, but we were surprised to find this small population of dormant stem cells capable of proliferating,» said principal investigator Ula Jurkunas, M.D., a corneal and refractive surgeon at Massachusetts Eye and Ear and Associate Professor of Ophthalmology at Harvard Medical School.

The corneal endothelium is responsible for pumping water throughout the cornea to keep vision clear, and when cells of the corneal endothelium die, they do not regenerate.

These studies, performed at Ohio University, are the first to identify the molecular mechanism of vitamin D3 - triggered restoration of the function of damaged endothelium in the cardiovasculature.

Researchers from Massachusetts Eye and Ear have, for the first time, identified rapidly proliferating cells (known as «neural crest - derived progenitor cells») in the corneal endothelium of specimens from normal corneas and from corneas with Fuchs» Endothelial Corneal Dystrophy (FECD), a condition in which the cells responsible for keeping the cornea clear die prematurely — often leading to blindness.

Therefore, the authors suggest that vitamin D3 may be of clinical importance in the restoration of dysfunctional cardiac endothelium after heart attack, capillary endothelium after brain ischemia (stroke), hypovolemia, vasculopathy, diabetes and atherosclerosis.

While these studies were performed using a cellular model of hypertension, the implication of vitamin D3 on dysfunctional endothelium is much broader.

After six weeks, researchers assessed how well the lining of blood vessels, or the endothelium, functioned, by measuring how much subjects» arteries dilated with increased blood flow.

In the process they induce inflammation; the cells also secrete other inflammatory chemicals, like interleukin - 6 and tumor necrosis factor - alpha, which are known to adhere to the endothelium of the blood vessels, an early event in atherosclerosis.

The researchers believe that MitoQ affects the endothelium, a thin layer of cells that lines our blood vessels.

In addition, when these transcription factors lose their function, terminal differentiation into the vascular endothelium (completion of differentiation) is completely suppressed, and genes that are key to differentiation into vascular endothelial cells as well as transcription factors that maintain the undifferentiated state are adversely induced.

Endothelial cells form the innermost layer of the blood vessels - the endothelium - and have the function of containing blood inside the vessels while allowing the passage of nutrients to tissues.

Furthermore, an examination of the histone code revealed that the regulatory genomic region of the transcription factors (ETS / GATA / SOX) was found to have gradually switched from a «brake histone mark,» which suppresses transcription, to an «accelerator histone mark,» which activates transcription, while in the process of differentiating into the vascular endothelium.

In drug development, particular attention is paid to the expression and function of membrane transporters expressed in epithelia of the intestine, liver and kidney, and in the endothelium of the blood - brain and blood - testis barriers.