Sentences with phrase «of host cells»

Tiny, round Cryptosporidium organisms line the outside of host cells.

The LumiFluor reporter constructs were designed to be packaged into a retrovirus for infection of host cells.

Bacterial pathogens are well known to cause tissue damage by colonization, induction of intense inflammation, invasion of host cells, and production of toxins [59].

A particularly efficient and deadly type of toxin punches holes in the membrane of host cells, and thereby kills them.

One of these genes codes for a protein that the team dubbed claudin - like apicomplexan microneme protein (CLAMP), which has a strong effect on the parasite's invasion of host cells.

The researchers suppressed the lipid droplets of the host cells, the bacterium's preferred food source, and found that the bacterium has a back - up plan that allows it to compensate for this shortage by drawing on the lipids within the host's membranes.

Like all viruses, rhinovirus C uses the molecular machinery of host cells to replicate and become infectious.

A study of the way malaria parasites behave when they live in human red blood cells has revealed that they can rapidly change the proteins on the surface of their host cells during the course of a single infection in order to hide from the immune system.

This is thought to prevent the viral envelope structure from changing its shape to enable infection of host cells.

But if we changed the code of the host cells, it would thwart the virus's ability to replicate, and so make the host immune.

By encasing itself in a protective bubble made of fat, pR1SE could hop out of its host cell — maybe even looking for other cells to occupy.

In the case of teh bacterial flagellum, removal of some of the «well matched parts» turns it into a type III secretory system which allows gram negative bacteria to translocate proteins directly into the cytoplasm of a host cell.

That was surprising, he explains, because «fibrillarin resides deep within the nucleolus of the host cell... [where it] methylates ribosomal RNA molecules, which then go on to form ribosomes,» but its full function may not be completely understood.

The viral plasma membrane is mostly co-opted from that of the host cell as newly formed viruses bud from infected cells.

They will then reproduce as directed by their own genes but using the resources of the host cell.

The new study shows that the synthetic compound is capable of inhibiting the activities of several DNA - processing enzymes, including the «integrase» used by the Human Immunodeficiency Virus (HIV) to insert its genome into that of its host cell.

When EBOV makes copies of itself inside cells, it does so by taking over and hijacking parts of that host cell's basic machinery to make its own proteins.

«What I find most fascinating is that it suggests that viruses can function with a sort of primitive «hive mentality» to ensure efficient use of host cell resources, akin to the way worker bees tell others where to locate the best food sources.»

The swivelling component takes the necessary piece of host cell RNA and directs it into a slot leading to the machine's heart, where it triggers the production of viral messenger RNA.

This process enables the virus to take advantage of the host cell's protein translation machinery for its own purposes.

In the future, these findings may form the basis for the development of new methods for treating viral infections, as the majority of all virus faces a similar challenge, namely to have to selectively replicate its own genetic material in competition with the genetic material of the host cell.

An international team of researchers from Denmark and Russia used a series of biochemical and structural biology techniques to investigate how the Qβ bacteriophage, which infects the common coli bacteria, utilises several of its host cell's proteins while replicating its genetic material.

Thanks to the protected environment of the host cell, these organisms tend to evolve rapidly, with the smallest mutating the fastest.

«These viruses are hijacking many of the host cell translation steps or pathways to favor the production of new virus progeny,» says Ruggieri.

Viruses are generally considered nonliving because of their small size, simplicity, and inability to replicate outside of a host cell.

As it invades a red blood cell, the malaria parasite takes part of the host cell's membrane to build a protective compartment.

Intracellular pathogens adopt various strategies to circumvent the defences of the host cell and to proliferate intracellularly.

d) Mechanisms of host cell actin remodeling.

b) Mechanism of host cell binding by the giant adhesin SiiE.

The two additional membranes are thought to correspond to the plasma membrane of the engulfed alga and the phagosomal membrane of the host cell.

Salmonella uses the SPI4 - T1SS secreted giant adhesin SiiE for temporal binding to the apical side of host cell.

It inserts its genes into the DNA of the host cell, as it normally does.

The first step in both direct and cell - based gene therapy is packaging a therapeutic transgene into a delivery vehicle (e.g., a viral vector), followed by expansion of its host cell lines to produce high - enough vector concentrations.

This helped reveal that the virus has the ability to control whether it is active or latent, independent of the host cell.»

Modulation of Host Cell Gene Expression through Activation of STAT Transcription Factors by Pasteurella multocida Toxin

Like most viruses, Ebola requires the help of a host cell to survive and replicate.

Ebola virus swarms the surface of a host cell in this electron micrograph.

Not exact matches

Instead of remaining trapped inside its host cell like most plasmids, the plasmid (which they named pR1SE) was able to break free.

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Many of these mutants are likely non-viable; however, many will have subtle changes to their functional components, including the HA gene which allows the virus to bind host cells, and NA gene which allows the virus to escape from host cells.

After her encounter in the garden with what she calls elsewhere a «slow - moving sinister aggregation of cells... like a cancer looking for a host,» wherever she goes and whatever she does, what she has seen accompanies her: «a body inside my body... budding and malign.»

I care about the life people like you are attempting to force in to this world, you care about what appeases your god - big difference when you really don't care or consider the child only what your imaginary friend god wants and yet your god cause abortions all the time... you care about a clump of cells, not the actual life of the child and that's much worse than me supporting the rights of a woman to have control over her body, especially if the clump of cells couldn't survive outside of the host.

«For pro-life folk, imagine a country where it was illegal to remove a tumor, even if the tumor might kill the host, because the tumor is its own collection of living cells.

The cell nuclei are removed from both sets of embryonic cells, as shown in the diagram, the donor's nuclei and the remains of the parents» embryo are destroyed and the parents» nuclei are then inserted into the donor or «host» embryo, still containing its healthy mitochondria.

Unfortunately, at this formative stage in their lives one viewpoint is pushed to the fore on campus, and that's the opinion that euthanasia, abortion, embryonic stem cell research and a host of other practices which strip humans of their most fundamental right are good things.

Where is the clear line in a progression from (1) using animal insulin to treat diabetes, to (2) using gene remodeling techniques to grow insulin in a host bacterium that will reproduce rapidly and from which a plentiful supply of insulin can be harvested, to (3) genetic surgery to replace the defective gene in a person diagnosed as diabetic, to (4) genetic surgery immediately after fertilization in order to replace the defective gene and alter the germ cells which would otherwise have transmitted the disease to one's offspring?

First seen as poisons, then as life - forms, then biological chemicals, viruses today are thought of as being in a gray area between living and nonliving: they can not replicate on their own but can do so in truly living cells and can also affect the behavior of their hosts profoundly.

Specifically, when capsaicin frequently binds to receptors within the human central nervous system's TRPV1 channel (the sensory receptor system for pain and heat detection), these receptors deplete and this depletion results in a whole host of benefits for the central nervous system at large, including terminating cancer cells, increasing the metabolic rate and digestive efficiency, increasing circulatory blood flow, and combatting inflammation, and making you feel better about the world.

In an FWF - funded project, scientists at the University of Graz modified and reinforced a host defence peptide from breast milk so as to enable it to specifically detect cancer cells.

Bracknell, UK, 3 November 2014 — Diversified technology company 3M has announced availability of a unique single - use platform that removes host cell DNA (HcDNA) and proteins (HCP) in various applications within the bioprocessing industry.