Sentences with phrase «of human cancer»
In particular, common references to PAHs in relation to human cancer risk have been loose and inconsistent with the scientific understanding of human cancer risk from this class of compounds.
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The complexity of human cancer etiology is particularly challenging for those types of cancer with long latency, which are associated with exposure to ubiquitous environmental carcinogens 34.
Historically, to help develop and understand the potential of human cancer treatments the gold -LSB-...]
«Because many types of tumors that affect people also occur naturally in dogs, focused ultrasound could not only augment the traditional approach to cancer in dogs but also advance our understanding of human cancer.»
But marijuana can still kill many types of human cancer cells.
It is interesting to note that through a test tube study, potent onion extract proved to destroy 95 % of human cancer cells, while extract from a sweet onion only killed 10 %!
Myrrh essential oil was able to reduce the replication of human cancer cells.
Dr. Michael Greger has also conveyed numerous times through his videos that animal products promote the growth of human cancer cells.
For example, in this apple study, they also measured the ability of apple extracts, from both peeled and unpeeled apples, to suppress the growth of human cancer cells growing in a petri dish, compared to control.
In an in vitro study, vanillin induced apoptosis and arrested the cell cycle of human colorectal cancer cells, and some researchers think vanillin could reduce the metastatic potential (ability to spread to other parts of the body) of human cancer cells in vivo, too.
Analysis of the human cancer glycome identifies a novel group of tumor - associated N - acetylglucosamine glycan antigens.
The higher the apple concentration, the more the growth rates of the human cancer cells dropped, compared to control.
In a Cancer Research study it was discovered that about 80 % of the human cancer cells grown in mice have died after they were treated with capsaicin.
Development of an orally - administrative MELK - targeting inhibitor that suppresses the growth of various types of human cancer
The next step in this ongoing research project involved translating the findings from basic research into applied clinical research with experimental treatment of human cancer patients.
The Nobel Prize laureate Otto Warburg who pioneered modern cancer metabolism research once said that «The cure of human cancer will be the resultant of biochemistry of cancer and of biochemistry of man ``.
An internationally recognized authority on the genetic basis of human cancer, Dr. Robert A. Weinberg is a founding member of the Whitehead Institute for Biomedical Research and the Daniel K. Ludwig Professor for Cancer Research at the Massachusetts Institute of Technology (MIT).
The Translational Research and Interventional Oncology Research Program (TR) promotes, develops and exploits mechanism - based research for improved detection and therapy of human cancer.
In particular, NOD / SCID / IL2rg -LRB-- / --RRB- mice can support the growth of various types of human cancer cells.
COSMIC, the Catalogue of Somatic Mutations in Cancer (http://cancer.sanger.ac.uk) is a high - resolution resource for exploring targets and trends in the genetics of human cancer.
«Lung adenocarcinoma is the leading cause of human cancer death.
Antisense - Mediated Suppression of Human Heparanase Gene Expression Inhibits Pleural Dissemination of Human Cancer Cells, Cancer Research, 61: 7855 - 7860, 2001.
Inhibition of growth, production of insulin - like growth factor - II (IGFII), and expression of IGF - II mRNA of human cancer cell lines by antagonistic analogs of growth hormone - releasing hormone in vitro.
Feinberg AP, Ohlsson R, and Henikoff S The epigenetic progenitor origin of human cancer.
Ludmil Alexandrov, awarded for his essay in the category Genomics and Proteomics: «Understanding the Origins of Human Cancer: Mutational Signatures»
«Mouse models of human cancer have taught us a great deal about the basic principles of cancer biology,» says Inder Verma, Ph.D., a professor in the Laboratory of Genetics.
And we can also use what we know about the genetics of the human cancer to make mutations in normal cells, and see how different mutations drive invasion.»
Methoxyethylamino - numonafide is an efficacious and minimally toxic amonafide derivative in murine models of human cancer.
We address these fundamental biological questions at the organismal level, utilizing relevant models of human cancer, and at a cellular and detailed molecular structural level.
Society for Natural Immunity (SNI) Symposium Towards Treatment of Human Cancer with NK Cells Saturday, May 5, 12:30 PM — 2:30 PM, Room 12AB Chairs: Hans - Gustaf Ljunggren, Karolinska Inst.
NCI's efforts to develop new laboratory models of human cancer includes vastly increasing the number of human cancer cell lines (grown as two - dimensional and three - dimensional cultures) and patient - derived tumor xenografts.
Michael Pfreundschuh and colleagues Ugur Sahin and Ozlem Türeci at the University of Saarland develop SEREX (SErological analysis of Recombinant cDNA Expression), providing a powerful method to analyze the humoral immune response to intracellular cancer antigens and ushering in the third phase of cancer serology, bringing with it the prospect of providing a comprehensive view of the immune recognition of human cancer.
But while most PDX mice are used as general models of human cancer in the laboratory, others seek to use them as Fiebig originally hoped — as avatars to guide customized patient care.
coordinate and conduct research on the causes of human cancer and the mechanisms of carcinogenesis;
For more than 25 years, scientists examining cultures of human cancer cells have occasionally spotted cells tucked within other cells.
Furthermore, their study reveals novel physical as well as functional links to phosphatase - based regulation of human cancer.
A year later, Carlo Croce, now director of the Human Cancer Genetics Program at Ohio State University, reported that chronic lymphocytic leukemia (CLL), the most common form of the disease, was caused by deletion of two microRNA genes.
In addition, the researchers examined neuroligin - 3 data from The Cancer Genome Atlas, a large public database of human cancer genetics.
«We've seen this in six different types of human cancer cell lines in the lab,» Choudhury said.
Slashing the amount of HSF1 in several lines of human cancer cells diminished growth and survival.
The use of bone marrow - derived stem cells is well established in the treatment of human cancer patients, and veterinary applications for bone marrow - and adipose - derived stem cells are being evaluated.
I would bet that by 2050 we'll have found that more than 80 percent of all human cancer is caused by infection.
PE: If I were going to put my money on it, I would bet that by 2050 — hopefully earlier — we'll have found that more than 80 percent of all human cancer is caused by infection.
The researchers have shown that it is possible to produce chromosome modifications in human cells that are genetically identical to those observed in leukemia and other types of human cancer.
Moreno also found a homologue of a human cancer gene involved in cell competition.
In their latest study, they tested compounds against cells from nine different types of human cancer, including common types affecting blood, colon, breast, prostate, ovaries, kidneys, and lungs.
The process of integrating naturally occurring cancers in dogs into the general studies of human cancer biology and therapy is known as comparative oncology.
Tumor virology has survived its failure to find abundant viral agents of human cancer.
Studies have shown that more than 50 % of all human cancers carry defects in the p53 gene, and almost all other cancers with a normal p53 function carry other defects which indirectly impair the cancer - fighting function of p53.
But she notes that even if the rodent finding is mirrored in people, only a subset of human cancers would be influenced by the drugs — those not already abundant in NRF2.