Athetoid cerebral palsy is comprised
of hypertonia and hypotonia, making it difficult for the individual to remain in the same position, walk or sit.
Not exact matches
Although many
of the components
of this syndrome, such as cognitive, sensory, and motor disabilities, are shared by other congenital infections, 5 features differentiate CZS from other congenital infections: (1) severe microcephaly with partially collapsed skull; (2) thin cerebral cortices with subcortical calcifications; (3) macular scarring and focal pigmentary retinal mottling; (4) congenital contractures; and (5) marked early
hypertonia with symptoms
of extrapyramidal involvement (Table).
The most common form
of cerebral palsy, spastic cerebral palsy accounts for about 70 to 80 percent
of all cases.1 Affected children typically exhibit
hypertonia and awkward movements in some or all areas
of the body.
If a baby appears «stiff,» this may indicate
hypertonia (increased muscle tone), which is the hallmark
of spastic cerebral palsy.
The list
of problems includes respiratory distress, cyanosis, apnea, seizures, temperature instability, feeding difficulty, vomiting, hypoglycemia, hypotonia,
hypertonia, hyperreflexia, tremor, jitteriness, irritability, and constant crying.