Sentences with phrase «of hypertrophic»

Symptoms associated with restrictive cardiomyopathy will often times be very similar to those of hypertrophic cardiomyopathy and in some cases not present any symptoms at all.
When it comes to purchasing life insurance with a diagnosis of hypertrophic cardiomyopathy, you'll generally find that you should be able to qualify for a Table Rating somewhere between Table D and Table J.
(ref) The condition is a form of hypertrophic cardiomyopathy.
Hyperthyroidism is not the only cause of hypertrophic cardiomyopathy.
The use of nutritional supplements can produce an imbalance in the calcium to phosphorus ratio, which has been implicated as one potential cause of the development of hypertrophic dystrophy.
I mentioned earlier that veterinarians believe that the underlying cause of hypertrophic cardiomyopathy (and perhaps the other cardiomyopathies as well) is a genetic defect in genes that direct how the heart muscle is constructed.
Certain breeds or families of dogs appear to be more susceptible to adverse vaccine reactions, particularly post-vaccinal seizures, high fevers, and painful episodes of hypertrophic osteodystrophy (HOD)(Dodds, 1997, 1999, 2001).
American and British shorthair — a less serious form of hypertrophic cardiomyopathy is seen in these breeds.
A cardiac examination allows us to follow a thorough investigative protocol to determine the presence and extent of hypertrophic cardiomyopathy in cats.
Persians (domestic long hair)-- this breed also has a high incidence of hypertrophic cardiomyopathy.
At one end of the severity spectrum, early chronic valvular disease, some forms of hypertrophic cardiomyopathy and some mild congenital heart defects may have little effect on an animal's lifestyle or life expectancy; at the other end, severe heart failure can interfere with even simple activities and prove rapidly fatal.
Most hyperthyroid cats will have some degree of hypertrophic cardiomyopathy (which is a condition where the heart muscle becomes thickened).
They found numerous cases of infections and metabolic diseases, and some skeletons showed signs of Hypertrophic Pulmonary Osteopathy and therefore potentially tuberculosis.
Osteological and Biomolecular Evidence of a 7000 - Year - Old Case of Hypertrophic Pulmonary Osteopathy Secondary to Tuberculosis from Neolithic Hungary.
Constitutive phosphorylation of cardiac myosin regulatory light chain prevents development of hypertrophic cardiomyopathy in mice.

Not exact matches

African Americans are being misdiagnosed with the heart condition (hypertrophic cardiomyopathy) that caused the sudden death of basketball player Hank Gathers (pictured left with teammate Bo Kimble) in 1990.
He has «apical hypertrophic cardiomyopathy» — essentially an enlargement of the heart which is caused by weak heart muscles.
An electrocardiogram revealed an abnormality, and follow - up tests in June confirmed that Carle has hypertrophic cardiomyopathy (HCM), a genetic disease that is the most common cause of sudden death in young athletes.
having other family members with a «history of premature death (sudden or otherwise), or significant disability from cardiovascular disease in close relative (s) younger than 50 years old or specific knowledge of the occurrence of certain conditions (eg, hypertrophic cardiomyopathy, dilated cardiomyopathy, long QT syndrome, Marfan syndrome, or clinically important arrhythmias)».
Carrying just one mutant copy of the gene causes an inherited heart problem called hypertrophic cardiomyopathy (SN: 9/17/16, p. 8).
Researchers found that the ring - shaped vortex helps to allocate about 15 percent of the blood flow within the left ventricle in healthy patients; roughly 20 percent in patients with non-ischemic dilated cardiomyopathy; but only about 5 percent for patients with hypertrophic cardiomyopathy.
In their study Mitalipov and colleagues edited out the MYBPC3 mutation associated with hypertrophic cardiomyopathy (HCM), a disease of the heart muscle that affects about one person in 500.
For this study, the team of cardiologists recruited 60 subjects, including 20 patients with non-ischemic dilated cardiomyopathy; 20 patients with hypertrophic cardiomyopathy; and 20 healthy subjects as a control group.
Their research, published in the Journal of Clinical Investigation, compared the differences between hypertrophic heart growth in rats as a result of exercise — which is beneficial — and heart growth induced by pathology — in this case, increased load.
A strong heart may be healthy, but too much heart muscle can be fatal: A leading cause of sudden death in young people — particularly in world - class athletes — is a condition called hypertrophic cardiomyopathy, or enlarged heart.
Hypertrophic cardiomyopathy (HCM) is the most common cause of sudden death in otherwise healthy young athletes, and affects approximately 1 in 500 people overall.
Seeking a genetic clue, a team led by David Bonthron, a clinical geneticist at the University of Leeds, U.K., went looking for the DNA glitch behind primary hypertrophic osteoarthropathy (PHO).
For extended modules, many different combinations of perturbed genes could incapacitate the module, as a result of which mutations in different genes will appear to lead to the same phenotype (e.g., hypertrophic cardiomyopathy).
Another lesson learned from hypertrophic cardiomyopathy is knowing the sarcomeric protein involved and the specific mutation does not invariably provide prognostic information about the course of the disease, including the risk of sudden cardiac death.
We also aim, in the model of cardiomyocyte, decipher implication of mutations identified in cardiomyopathies such as inherited hypertrophic cardiomyopathies (HCM), Atrial fibrillation (AF) or Arrhythmogenic right ventricular cardiomyopathy (ARVC).
Characterized other proteins and pathways that modulate proliferation and differentiation of myogenic stem cells, hypertrophic growth of the heart, mitochondrial biogenesis and fiber type — specific gene expression in skeletal muscles.
Left ventricular outflow tract gradient is associated with reduced capillary density in hypertrophic cardiomyopathy irrespective of genotype.
Poster Presentations Allison Schreiber — «CHD7 Variants in Two Families with Evidence of Parent - to - Child Transmission of CHARGE Syndrome» Patty Arscott — «Genetic Counseling Dilemma: Multiple Gene Variants Identified Through Genetic Testing for Hypertrophic Cardiomyopathy» Christina Rigelsky — «Clinical Features of Three Patients with Vascular Ehlers - Danlos Syndrome as the Result of COL3A1 Functional Haploinsufficiency with Stable mRNA but Unstable Protein» Rebecca Cook — «Assessing the Utility of Four Educational Videos Concerning Alpha - 1 Antitrypsin Deficiency» and «Guidelines for Advocacy Organizations Considering Establishing a Registry or Biobank» Educational Breakout Session Presentations Meg Doerr — «Using Information Technology to Facilitate the Cancer Genetic Counseling Process»
A mouse model of familial hypertrophic cardiomyopathy (FHC) is providing scientists with valuable information about whether genes or environment trigger sudden death.
It is not only the earliest occurrence of fully - developed hypertrophic pulmonary osteopathy on an adult skeleton to date, but also clearly establishes the presence of tuberculosis in Europe 7,000 years ago.»
We are recruiting and enrolling 10 primary care physicians (PCPs) and 100 of their generally healthy middle - aged patients ages 40 - 65 to evaluate the use of General Genomic Medicine, and 10 cardiologists and 100 of their patients with hypertrophic or dilated cardiomyopathy to evaluate the use of Disease - Specific Genomic Medicine.
Even if the stem cells are induced to differentiate into cartilage cells, they spontaneously mature into a so - called «hypertrophic» state, ultimately leading to the formation of bone tissue; this is similar to the cartilaginous tissue temporarily formed after a fracture.
Dr. Seidman has investigated the molecular etiology of human diseases, with a focus on congenital heart disease, familial hypertrophic cardiomyopathy, and familial dilated cardiomyopathy.
The mice had grossly evident hypertrophic cardiomyopathy compared to Idua + / - control mice, which is consistent with other assessments of cardiovascular phenotype [23].
Hereditary hypertrophic cardiomyopathy occurs in about one out of every 500 adults, and is passed along when a person winds up with one good copy and one mutated copy of a gene called MYBPC3, the researchers said.
People with hypertrophic cardiomyopathy are at increased risk of heart failure and sudden heart death.
Studies have shown that such rep patterns coupled with loads that are at least 65 % of your 1RM optimize the hypertrophic response.
In this instance, leading scientist Shoukhrat Mitalipov, with help from researchers from the United States and South Korea, used CRISPR to delete a genetic mutation for «HCM,» or hypertrophic cardiomyopathy, in the sperm of a man with the condition.
Still, since the volume of training was greater for the hypertrophic regime, the stronger anabolic response could be caused by the greater total volume and not the longer TUT.
The best attempt is a recent study by Hulmi et al. which reported that the anabolic response after a hypertrophic (5 sets of 10 reps) resistance training regime was greater than the one following a maximal strength (15 sets of 1 rep) resistance training regime.
One of them, that explored hypertrophic adaptations between different concentric and eccentric tempos under traditional training methods, split 28 women with previous training experience into two groups: one performed lower body exercises with two - second duration of the concentric portion and a six - second duration of the eccentric portion of the movement, while the second group trained on the reverse way — with a six - second duration of the concentric and two - second duration of the eccentric portion.
There's been a lot of debate whether increasing the time under tensions triggers greater muscle gains, but the majority of studies support this claim under various discoveries: • The cross-section area of the slow - twitch type muscle fibers is greatly increased, which leads to maximal stimulation of these type I fibers since they possess a high level of fatigue threshold, which also promotes an increased hypertrophic response.
As with all other types of training, employing periodization will trigger greater hypertrophic and strength gains in the long term.
One theory proposes that once the storage capacity of subcutaneous adipose tissue (SAT) depots is exceeded under conditions of energy excess, either as a result of impaired expandability and / or excessive hypertrophic growth, fat deposition within visceral depots and non-adipose tissues including the liver, skeletal muscle and pancreas can ensue.93 This can subsequently lead to the development of systemic IR and a series of associated cardiometabolic disorders including dyslipidaemia, dysglycaemia, hyperinsulinaemia and hypertension.3 Expression of pro-inflammatory mediators including interleukins 1 (IL - 1), 6 (IL - 6), tumour necrosis factor alpha (TNF - α) and resistin, are also increased which can further potentiate IR and promote atherosclerosis.
Due to an absence of data, it is not clear whether training muscle groups more than 3 days per week might enhance the hypertrophic response.
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