Sentences with phrase «of mouse eyes»

While Yuh - Nung studied sensory transduction in a fungus, Lily set forth to localize the visual pigment rhodopsin in the retina of mouse eyes.

Not exact matches

That was the message that Hyung - Jin Shin sent out today in Seoul, South Korea, with the help of Samsung's «eye mouse,» or Eyecan +, The Verge reports.

Using the gene - editing tool CRISPR - Cas9 to turn off certain genes in a mouse zygote as well as other new techniques to enrich the pluripotent stem cells of a rat, the group managed to grow various rat organs (a pancreas, heart, and eyes) in a mouse embryo.

as he follows the tracks of the skunk in the January snow; wondering where the skunk is beading and why; speculating on the different meanings of a winter thaw for the mouse whose snow burrow has collapsed and the owl who has just made dinner on the mouse; trying to understand the honking of the geese as they circle the pond; and wondering what the world must look like to a muskrat eye - deep in the swamp.

When tissue from the jaw region of a chick embryo is wrapped in tissue from a mouse embryo from the region where teeth are formed and then incubated in the eye of an adult mouse, the chick develops teeth.

A study published in the October, 1999 issue of the Archives of Environmental Health found that laboratory mice exposed to various brands of disposable diapers suffered increased eye, nose, and throat irritation, including bronchoconstriction similar to that of an asthma attack.

The shape of the plate kind of reminds me of Mickey Mouse and the utensils have little eyes.

Another study published in 1999 by Anderson Laboratories found that lab mice exposed to various brands of disposable diapers experienced asthma - like symptoms, as well as eye, nose and throat irritation.

Other rows are eyes from three different mouse models of diabetes.

The researchers found that these electric currents were much weaker in eyes from all three strains of diabetic mice than in healthy mice.

One of the research lines that John, an investigator of the Howard Hughes Medical Institute (HHMI) and a professor at The Jackson Laboratory in Bar Harbor, Maine, currently has going is the development of a wireless sensor so tiny it can be implanted into the eye of a mouse.

In the new work, published June 10 in the journal Scientific Reports, Zhao, Reid and colleagues used a highly sensitive probe to measure electrical fields in the corneas of isolated eyes from three different lab mouse models with different types of diabetes: genetic, drug - induced and in mice fed a high - fat diet.

The study coupled gene therapy that excited visual neurons in the eyes with stimulation — a swirling black - and - white grid placed in front of the mice.

Both strains of mice were extremely fat, a trait that was passed on to their offspring in a simple, dominant pattern of inheritance, like eye color.

Mice with this eye disease, which damages the optic nerve and causes vision loss, have higher levels of the immune molecule, which accumulates at retinal synapses before the neurons die.

The largest effect was on the number of times the mice went in and out of a sleep phase called paradoxical sleep, which resembles REM (rapid eye movement) sleep in humans, when dreams occur and memories are strengthened.

By peering into the eyes of mice and tracking their ocular movements, researchers made an unexpected discovery: the visual cortex — a region of the brain known to process sensory information — plays a key role in promoting the plasticity of innate, spontaneous eye movements.

Only when Foster surgically removed their entire eyes did the blind mice drift out of sync.

Scanziani and his colleagues sought to understand the origins of this adaptive plasticity by studying the eye movements in mice before and after disabling their vestibular ocular reflex.

Mice and humans are so closely related that it seemed likely we have the same basic collection of ipRGCs in our eyes, carrying out the same tasks.

When we took the mouse version of this gene — the same gene we find in the human — and put it in the fly and tweaked it, we induced fly eye tissue.

First she deprived juveniles of vision in one eye so that the corresponding brain cells failed to make connections; once the mice reached maturity, they were put in a water maze that required them to recognize a pattern of fine lines to find a floating platform.

If one eye is deprived of sight, they rapidly rewire their brains to compensate, then beat normal one - eyed mice on tests of visual acuity.

Anatomical examination of human and mouse eyes was used to determine the effect of the laser on the sensitive light - detecting retina.

A tumor treated with verteporfin (right) is much smaller and much of the structure of the mouse's eye is visible.

These glowing layers reveal the complex structure of the retina — the photoreceptive part of the eye — in a mouse at one month old.

Along the way, the researchers also kept an eye on ticks, as white - footed mice are a reservoir of the Lyme disease spirochete, which they transmit to tick larvae.

By pairing a receptor that targets neurons with a molecule that degrades the main component of Alzheimer's plaques, the biologists were able to substantially dissolve these plaques in mice brains and human brain tissue, offering a potential mechanism for treating the debilitating disease, as well as other conditions that involve either the brain or the eyes.

Now scientists at the Massachusetts Institute of Technology (MIT) have taken a first step toward designing such a treatment: They have identified a protein that stimulates regrowth of severed eye - brain connections in mice, according to a report in tomorrow's issue of Nature.

Inhibiting the same area of the brain with similar techniques increased the amount of time mice slept, especially non-REM (rapid eye movement) sleep, although sleep was not instant upon drug administration, as has been shown for other parts of the arousal system, Pedersen says.

When the eyes of her mice looked normal, Xu Wang was certain she had done something wrong.

The basic experimental approach is to record from neurons of the visual cortex of an animal - in this case a mouse - some time after one of its eyes has been sutured shut.

By contrast, several other strains of bacteria inoculated onto the eyes of JAX mice disappeared without inducing local immunity.

Treating the potentially blinding haze of a scar on the cornea might be as straightforward as growing stem cells from a tiny biopsy of the patient's undamaged eye and then placing them on the injury site, according to mouse model experiments conducted by researchers at the University of Pittsburgh School of Medicine.

In a four - year study conducted on the mouse model in advanced breast cancer metastasis in the eye's anterior chamber, Petty and colleagues found that the new nanoparticle not only killed tumor cells in the eye, but also extended the survival of experimental mice bearing 4T1 tumors, a cell line that is extremely difficult to kill.

NERVE PROTECTORS The glowing cells in this micrograph of a mouse's optic nerve help shield electrical signals passing between eyes and brain.

In normal mice with working photoreceptors (PR driven), stimulating the retina produces a variety of responses in retinal ganglion cells, the output of the eye.

They found that the mice can develop damage to the optic nerve despite normal pressure in the eye following KPro surgery and identified TNFa and IL - 1 as inflammatory factors involved in this process, with high levels of TNFa mediating the damage to the optic nerve.

«We used a mouse model of the KPro to, first of all, identify the inflammatory factors that cause damage to the eye, and then we also quantified the amount of nerve cell death in the back of the eye that mediates the optic neuropathy, and, lastly, we looked at blocking these factors with antibodies,» said Reza Dana, M.D., M.Sc., MPH, Director of the Cornea and Refractive Surgery Service at Massachusetts Eye and Ear and the Claes H. Dohlman Professor of Ophthalmology at Harvard Medical School.

To precisely map how glucose and lactate move around in the eye, Hurley and colleagues grew human RPE in a lab dish and studied its biochemistry along with that of isolated mouse retinas.

This rainbow pinwheel of mouse placentas isn't just an eye - catching, award - winning image.

The software can determine whether the people are actually at their desks as well as very nuanced metrics of their computer usage, from number of words typed per minute and mouse clicks to where their eyes go on the screen.

Researchers from Massachusetts Eye and Ear / Harvard Medical School have identified inflammatory factors that cause optic neuropathy in the back of the eye following implantation of a keratoprosthesis (KPro)-- similar to what glaucoma patients experience, without the rise of pressure in the eye — and have shown that blocking one of those factors, tumor necrosis factor alpha (TNFa), successfully halts the development of optic nerve damage in a mouse model.

«Importantly, the investigation also demonstrates that newly generated cells in the mouse retina not only look and behave like neurons, they also wire correctly to the existing neural circuitry at the back of the eye.»

The computer speaks a word describing one of the pictures and then captures the child's eye movement every three milliseconds until the child uses a computer mouse to click on one of the pictures.

Working with mice, a multicenter team of researchers has found a new way to reduce the abnormal blood vessel growth and leakage in the eye that accompany some eye diseases.

The finding, published in The Journal of Clinical Investigation, validates a similar discovery made by the scientists in mice two years ago and suggests a target for future therapies to treat the devastating eye disease that currently has no cure.

Studying mouse models of glaucoma, Ban, Apte and their colleagues identified a molecule in the eye called growth differentiation factor 15 (GDF15), noting that the levels of the molecule increased as the animals aged and developed optic nerve damage.

A compound that reverses the molecular cause of cataract formation improves eye lens transparency in mice.

The study, published in Cell Reports, involved giving cancer drugs to mice and inducing uveitis, an incurable autoimmune eye condition responsible for 1 in every 10 cases of visual impairment in the UK.