As a result, blood tests may be detecting the presence
of other
mycobacterial infections; they may also produce false positives when antibody levels remain elevated after cases
of TB have been treated.
Anna Huttenlocher, University
of Wisconsin, USA Neutrophils in the Tumor Microenvironment Neutrophils, Wounds, and Cancer Progression Stefan Kaufmann, Max Planck Institute, Germany Pathology and immune reactivity: understanding multidimensionality in pulmonary tuberculosis Constitutive BAK activation as a determinant
of drug sensitivity in malignant lymphohematopoietic cells Kathryn Moore, New York University, USA MicroRNA -33-dependent regulation
of macrophage metabolism directs immune cell polarization in atherosclerosis Lalita Ramakrishnan, University
of Cambridge, UK Myeloid Growth Factors Promote Resistance to
Mycobacterial Infection by Curtailing Granuloma Necrosis through Macrophage Replenishment Beth Stevens, Harvard University, USA Microglia: Dynamic Mediators
of Synapse Development and Plasticity Do glia drive synaptic and cognitive impairment in disease?
How a
mycobacterial lipid subverts host macrophages in tuberculosis and leprosy Lalita Ramakrishnan University
of Cambridge, UK 20th October 201704:15 pm Host: Pedro Alves, MPIIB Location: Max Planck Institute for
Infection Biology, Seminar room 1 +2 - Campus Charité Mitte
The cause
of this isn't always clear, but could be genetic or the result
of trauma, calcium deficiency, or a
mycobacterial infection.