Vitamin C has been shown to reduce markers
of oxidative stress after exercise 9.
Not exact matches
4 - The Redox is at the heart
of it all, as lipid peroxidation load drops redox balance is greatly affected, meaning a cell redox state
of little
oxidative stress and inflammatory gene activation (by absence
of lipid peroxidation
after lipid remodeling).
Certain particle compounds may directly generate ROS in vivo because
of their surface chemistry (eg, metals, organic compounds, and semiquinones) or
after bioactivation by cytochrome P450 systems (eg, polycyclic aromatic hydrocarbon conversion to quinones).6, 290 a, 290 b A particle surface or anions present on otherwise more inert particles may disrupt iron homeostasis in the lung and thereby also generate ROS via Fenton reactions.291 Other PM constituents may do so indirectly by the upregulation
of endogenous cellular sources (eg, nicotinamide adenine dinucleotide phosphate [NADPH]-RRB- oxidase) 292,293 or by perturbing organelle function (eg, mitochondria) by taken - up PM components.261 Particle stimulation
of irritant and afferent ANS fibers may also play a role in local and systemic
oxidative stress formation.294 Given the rich antioxidant defenses in the lung fluid, secondarily generated oxidization products
of endogenous molecules (eg, oxidized phospholipids, proteins) or a reduction in endogenous antioxidants per se may be responsible at least in part for the state
of oxidative stress in the lungs (along with instigating the subsequent cellular responses) rather than ROS derived directly from PM and its constituents.
Oxidative stress during and
after a workout «Now researchers have extended this study by looking at the effect
of antioxidants in trained female runners,» says Crowe.
Studies have found that it restores normal patterns
of eating and sleeping
after stress; lowers mental and physical fatigue; and protects against
oxidative stress, heat
stress, radiation and exposure to toxic chemicals.
Research has shown that the addition
of bovine colostrum can reduce
oxidative stress on and damage to muscles
after exercise.
Stress resistance has not been assessed however and so the biological relevance of this finding is currently unknown.32 Several IER trials (75 - 85 % ER on restricted days) in overweight / obese populations have reported reductions in various markers of oxidative stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Dire
Stress resistance has not been assessed however and so the biological relevance
of this finding is currently unknown.32 Several IER trials (75 - 85 % ER on restricted days) in overweight / obese populations have reported reductions in various markers
of oxidative stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Dire
stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison
of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels
of fast - acting advanced oxidation protein products (AOPP)
after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels
of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation
of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups
after three months.48 Summary and Future Research Directions
Immediately before or
after a bout
of physical exercise, consumption
of 10 - 30 grams
of cysteine - rich whey protein (depending on body weight and exercise intensity) is a better option since this delivers amino acids preferentially to skeletal muscle while also providing cysteine to lessen
oxidative stress.15
Effect
of blueberry ingestion on natural killer cell counts,
oxidative stress, and inflammation prior to and
after 2.5 h
of running.
All this culminated in a study where 84 grams
of almonds reduced overall levels
of oxidative stress in humans by 23 - 24 %
after four weeks.
«Soreness
after a workout is caused by a combination
of inflammation, muscle damage and
oxidative stress,» explains Malachy P. McHugh, Ph.D., Director
of Research at the Nicholas Institute
of Sports Medicine and Athletic Trauma (NISMAT) and New York Rangers Hockey Team consultant.
Research has shown that supplementation
of taurine increases muscular performance, exercise capacity, reduces lactate acid concentration during exercise, muscle damage and
oxidative stress after exercise *.
A new study demonstrates the impact
of Montmorency tart cherries on inflammation and
oxidative stress after high - intensity cycling.
«We have been investigating Montmorency tart cherries for several years because they're a unique fruit with a high concentration
of anthocyanins,» said co-author Howatson, whose previous studies have demonstrated the ability
of Montmorency tart cherry juice to reduce certain inflammation responses and
oxidative stress after high - intensity exercise.
Soreness
after a workout is caused by a combination
of inflammation, muscle damage and
oxidative stress — and researchers suggest the natural compounds in Montmorency tart cherries, including anthocyanins, may aid in faster recovery, decreased muscle pain, and less muscle damage when consumed prior to working out, as well as
after.1, 2,3
Furthermore, pump workers, who are often exposed to benzene; a chemical which induces
oxidative -
stress related toxicity, lessened their benzene - induced toxicity and saw significant improvements in their antioxidant status,
after they had to drink six cups
of green tea per day for over six months.
They analysed the bloodstreams
of each patient
after both 15 days and 30 days, and found that the level
of oxidative stress in their bodies had fallen substantially.
A multi-center study
of German breast cancer patients showed that IV therapy alongside chemo and radiation keeps vitamin C levels where they should be and counteracts the
oxidative stress that cancer treatments cause, reducing side effects and enhancing quality
of life during and
after treatment.