Sentences with phrase «of plaque deposits»

Much like the buildup of plaque deposits in human veins and arteries, an accumulation of debt gained momentum exponentially until the economy crashed, wiping out bad debts — along with savings on the other side of the balance sheet.

Not exact matches

Pterostilbene is also beneficial for preventing plaque deposits in our arteries which causes narrowing and blockage of the arteries.
However, there are others who accumulate cholesterol in the blood, resulting in high serum cholesterol counts that lead to atherosclerosis, which is plaque deposits of cholesterol, fats, and other remains in the walls of medium - sized and large arteries.
Have a higher prevalence of stroke, atherosclerosis [a common form of arteriosclerosis in which fatty substances form a deposit of plaque on the inner lining of arterial walls], chronic obstructive pulmonary disease (COPD), Crohn's disease, lymphoma, metabolic syndrome [a collection of heart disease risk factors], cancer, and liver disease.
Although nanobacteria may not cause kidney stone disease, Coe notes additional circumstantial evidence: At least four teams have reported tiny spherical deposits in the calcified plaques that often appear in the kidneys of patients who suffer from kidney stones.
Previously, researchers have focused on the role of protein deposits called amyloid plaques that lodge in the brain of Alzheimer's affected people.
«Our study shows that both higher levels of HDL — good — and lower levels of LDL — bad — cholesterol in the bloodstream are associated with lower levels of amyloid plaque deposits in the brain,» said Bruce Reed, lead study author and associate director of the UC Davis Alzheimer's Disease Center.
When large quantities of LDL cholesterol circulate through the bloodstream, it can be deposited on artery walls, leading to a buildup of plaque and triggering inflammation.
A major contributor to these cardiovascular diseases is clogged blood vessels (atherosclerosis), which result from the buildup of fatty deposits or plaque.
More than 40 illnesses known as amyloid diseases — Alzheimer's, Parkinson's and rheumatoid arthritis are a few — are linked to the buildup of proteins after they have transformed from their normally folded, biologically active forms to abnormally folded, grouped deposits called fibrils or plaques.
One look at an image of an Alzheimer's afflicted brain is unflinching testimony to the disease's cruelty: It destroys of up to 30 percent of a brain's mass, carving out ravines and depositing piles of molecular junk, most visibly amyloid plaque.
Atherosclerosis — hardening of the arteries — is a potentially serious condition where arteries become clogged by a build - up of fatty deposits known as «plaques».
Studies in mice specially bred to have features of the disease found that DHA reduces beta - amyloid plaques, abnormal protein deposits in the brain that are a hallmark of Alzheimer's, although a clinical trial of DHA showed no impact on people with mild to moderate Alzheimer's disease.
Our ancestors going back thousands of years show signs of atherosclerosis, as suggested by modern research using CT to detect evidence of calcium deposits associated with atherosclerotic plaques in the arteries of mummies as old as 5,000 years.
Whether this strategy will stop Alzheimer's disease in its tracks is a big if, however, because there's still a debate over whether the b - amyloid deposited in plaques causes the brain damage seen in Alzheimer's disease, or instead is a symptom of some other underlying factor.
While approximately 80 percent of Alzheimer's disease patients also have cerebral amyloid angiopathy, or amyloid beta deposits in the brain's blood vessels that increase the risk for stroke and dementia, the predominant amyloid beta pathology is plaques.
Those plaques are dense deposits of protein and other cell material that attach themselves to and eventually kill neurons responsible for memory and decision making.
Immunotherapy is a promising strategy for the treatment of Alzheimer's that uses antibodies to stimulate the immune system to remove pieces of a protein called amyloid beta which accumulates in the brain (in deposits known as plaques) and is thought to be a major factor driving Alzheimer's neurodegenerative effects.
«The activity of the microglia is stimulated by dying brain cells, not by the deposits of amyloid proteins, called plaques, which also occur in Alzheimer's disease,» Haass notes.
The results were remarkably positive, with the reduction in BACE1 activity not only stalling the development of amyloid plaques in the mice, but actually removing the deposits that had already formed.
Measuring tau deposits using the novel radiotracer 18F - AV - 1451 (18F - T807), in conjunction with 11C - PiB to measure amyloid plaques and 18F - FDG to measure regional neurodegeneration, offers new insight into the neurodegenerative characteristics of Alzheimer's disease and shows that tau pathology may be an instrumental target for disease - modifying strategies.
At 10 months of age the mouse models were found have no amyloid plaque deposits at all.
atherosclerosis A form of heart disease where the vessels and arteries can be narrowed dangerously (threatening to clog completely) by the buildup of fatty deposits known as plaque.
They also work as garbage collectors, chewing up dead cells and molecular debris strewn among living cells — including clusters of a protein called A-beta, notorious for aggregating into gummy deposits called Alzheimer's plaques, the disease's hallmark anatomical feature.
The findings include evidence of how the protein fragments that make up the Alzheimer's plaque are deposited outside neurons and how loss of NEU1 possibly contributes to disease progression and spread.
The tiny particles are 1,000 times smaller than the tip of a human hair, and are designed to latch on to atherosclerotic plaques — hard deposits made from accumulated fat, cholesterol and calcium that build up on the walls of arteries and are prone to rupture, producing dangerous clots.
The underlying cause of many heart attacks is atherosclerosis, defined as build - up of deposits, or plaques, of cholesterol, calcium, and other substances in arteries.
The two types of cholesterol are low - density lipoprotein (LDL), or the bad cholesterol because it contributes to plaque, a hard deposit that can clog arteries, making them less flexible, and high - density lipoprotein (HDL), the good cholesterol which is able to reverse the effects of LDL cholesterol in the body.
The EDTA binds to these calcium deposits and flushes it out of the bloodstream, taking with it the arterial plaque.
Specifically, people who got plenty of physical activity and had a normal BMI had fewer plaque deposits and tangles than those who got less exercise and had higher BMIs.
The researchers were looking for two specific types of protein: deposits of beta - amyloid plaque and knotted threads of tau protein tangles.
In fact, the plaque deposits from bad bugs in the mouth are the same types of plaque found in arterial walls in heart disease sufferers!
Plaques form inside arteries in locations where the artery was, at some point in the past, acutely damaged by deposits of highly irritating pro-inflammatory fats that splatter on the insides of your arteries.
A build up of plaque in the arteries results from a damaged blood vessel having calcium deposited there so that you can remain alive.
Atherosclerosis, or the buildup of plaque in the arteries was originally considered to be cholesterol sludge depositing in the arterial walls.
Learn about the world's very best treatments for arteriosclerosis known as hardening of the arteries via plaque deposits.
Scientists have identified a basic pathologic process underlying Alzheimer's development that involves the formation of abnormal protein deposits in the brain known as beta - amyloid plaques, but they still aren't entirely sure what causes this to happen.
Basically, if you have significant internal inflammation, this plaque will be deposited as a healing agent regardless of whether you have high or low cholesterol.
TMAO is thought to encourage fatty plaque deposits to form within arteries (atherosclerosis), and therefore, the more TMAO you have in your blood the greater your risk of heart disease might be.
Over time, too much glucose in the blood can lead to increased plaque deposits on the insides of the blood vessel walls.
The skin on the shins gets thick with raised plaques or nodules due to deposits of altered material in the skin.
Even with regular brushing plaque and tartar are deposited on the tooth surface, out of sight, above and below the gumline,.
Deposits of plaque build - up on your pet's teeth.
Made with North Atlantic alga Ascophyllum nodosum, their line of food additive, treats, and bones for both cats and dogs prevent dental plaque from sticking to the teeth and soften already existing tartar deposits.
These products will wipe off plaque deposits from the surface of the tooth and, though they lack the ability to pick food particles out of the gum socket, they are probably the next best thing to brushing and, like brushing, these products are best used daily.
It begins with deposits of plaque which harden to form tartar.
Fourth, a POPD begins with the removal of supra - gingival deposits of plaque and calculus from the buccal, lingual and interproximal surfaces.
When the tooth encounters a piece of kibble, the morsel shatters and subsequently provides no abrasive effect to scrape plaque (invisible layer of bacteria), tartar (yellow - brown deposits from chronic bacterial growth), or other food debris from tooth surfaces.
STEP 4: Supra - gingival scaling — a POPD begins with the removal of supra - gingival deposits of plaque and calculus from the buccal, lingual and interproximal surfaces.
This plaque, in turn leads to deposits of rock - hard tartar along the gum line.
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