Much like the buildup
of plaque deposits in human veins and arteries, an accumulation of debt gained momentum exponentially until the economy crashed, wiping out bad debts — along with savings on the other side of the balance sheet.
Not exact matches
Pterostilbene is also beneficial for preventing
plaque deposits in our arteries which causes narrowing and blockage
of the arteries.
However, there are others who accumulate cholesterol in the blood, resulting in high serum cholesterol counts that lead to atherosclerosis, which is
plaque deposits of cholesterol, fats, and other remains in the walls
of medium - sized and large arteries.
Have a higher prevalence
of stroke, atherosclerosis [a common form
of arteriosclerosis in which fatty substances form a
deposit of plaque on the inner lining
of arterial walls], chronic obstructive pulmonary disease (COPD), Crohn's disease, lymphoma, metabolic syndrome [a collection
of heart disease risk factors], cancer, and liver disease.
Although nanobacteria may not cause kidney stone disease, Coe notes additional circumstantial evidence: At least four teams have reported tiny spherical
deposits in the calcified
plaques that often appear in the kidneys
of patients who suffer from kidney stones.
Previously, researchers have focused on the role
of protein
deposits called amyloid
plaques that lodge in the brain
of Alzheimer's affected people.
«Our study shows that both higher levels
of HDL — good — and lower levels
of LDL — bad — cholesterol in the bloodstream are associated with lower levels
of amyloid
plaque deposits in the brain,» said Bruce Reed, lead study author and associate director
of the UC Davis Alzheimer's Disease Center.
When large quantities
of LDL cholesterol circulate through the bloodstream, it can be
deposited on artery walls, leading to a buildup
of plaque and triggering inflammation.
A major contributor to these cardiovascular diseases is clogged blood vessels (atherosclerosis), which result from the buildup
of fatty
deposits or
plaque.
More than 40 illnesses known as amyloid diseases — Alzheimer's, Parkinson's and rheumatoid arthritis are a few — are linked to the buildup
of proteins after they have transformed from their normally folded, biologically active forms to abnormally folded, grouped
deposits called fibrils or
plaques.
One look at an image
of an Alzheimer's afflicted brain is unflinching testimony to the disease's cruelty: It destroys
of up to 30 percent
of a brain's mass, carving out ravines and
depositing piles
of molecular junk, most visibly amyloid
plaque.
Atherosclerosis — hardening
of the arteries — is a potentially serious condition where arteries become clogged by a build - up
of fatty
deposits known as «
plaques».
Studies in mice specially bred to have features
of the disease found that DHA reduces beta - amyloid
plaques, abnormal protein
deposits in the brain that are a hallmark
of Alzheimer's, although a clinical trial
of DHA showed no impact on people with mild to moderate Alzheimer's disease.
Our ancestors going back thousands
of years show signs
of atherosclerosis, as suggested by modern research using CT to detect evidence
of calcium
deposits associated with atherosclerotic
plaques in the arteries
of mummies as old as 5,000 years.
Whether this strategy will stop Alzheimer's disease in its tracks is a big if, however, because there's still a debate over whether the b - amyloid
deposited in
plaques causes the brain damage seen in Alzheimer's disease, or instead is a symptom
of some other underlying factor.
While approximately 80 percent
of Alzheimer's disease patients also have cerebral amyloid angiopathy, or amyloid beta
deposits in the brain's blood vessels that increase the risk for stroke and dementia, the predominant amyloid beta pathology is
plaques.
Those
plaques are dense
deposits of protein and other cell material that attach themselves to and eventually kill neurons responsible for memory and decision making.
Immunotherapy is a promising strategy for the treatment
of Alzheimer's that uses antibodies to stimulate the immune system to remove pieces
of a protein called amyloid beta which accumulates in the brain (in
deposits known as
plaques) and is thought to be a major factor driving Alzheimer's neurodegenerative effects.
«The activity
of the microglia is stimulated by dying brain cells, not by the
deposits of amyloid proteins, called
plaques, which also occur in Alzheimer's disease,» Haass notes.
The results were remarkably positive, with the reduction in BACE1 activity not only stalling the development
of amyloid
plaques in the mice, but actually removing the
deposits that had already formed.
Measuring tau
deposits using the novel radiotracer 18F - AV - 1451 (18F - T807), in conjunction with 11C - PiB to measure amyloid
plaques and 18F - FDG to measure regional neurodegeneration, offers new insight into the neurodegenerative characteristics
of Alzheimer's disease and shows that tau pathology may be an instrumental target for disease - modifying strategies.
At 10 months
of age the mouse models were found have no amyloid
plaque deposits at all.
atherosclerosis A form
of heart disease where the vessels and arteries can be narrowed dangerously (threatening to clog completely) by the buildup
of fatty
deposits known as
plaque.
They also work as garbage collectors, chewing up dead cells and molecular debris strewn among living cells — including clusters
of a protein called A-beta, notorious for aggregating into gummy
deposits called Alzheimer's
plaques, the disease's hallmark anatomical feature.
The findings include evidence
of how the protein fragments that make up the Alzheimer's
plaque are
deposited outside neurons and how loss
of NEU1 possibly contributes to disease progression and spread.
The tiny particles are 1,000 times smaller than the tip
of a human hair, and are designed to latch on to atherosclerotic
plaques — hard
deposits made from accumulated fat, cholesterol and calcium that build up on the walls
of arteries and are prone to rupture, producing dangerous clots.
The underlying cause
of many heart attacks is atherosclerosis, defined as build - up
of deposits, or
plaques,
of cholesterol, calcium, and other substances in arteries.
The two types
of cholesterol are low - density lipoprotein (LDL), or the bad cholesterol because it contributes to
plaque, a hard
deposit that can clog arteries, making them less flexible, and high - density lipoprotein (HDL), the good cholesterol which is able to reverse the effects
of LDL cholesterol in the body.
The EDTA binds to these calcium
deposits and flushes it out
of the bloodstream, taking with it the arterial
plaque.
Specifically, people who got plenty
of physical activity and had a normal BMI had fewer
plaque deposits and tangles than those who got less exercise and had higher BMIs.
The researchers were looking for two specific types
of protein:
deposits of beta - amyloid
plaque and knotted threads
of tau protein tangles.
In fact, the
plaque deposits from bad bugs in the mouth are the same types
of plaque found in arterial walls in heart disease sufferers!
Plaques form inside arteries in locations where the artery was, at some point in the past, acutely damaged by
deposits of highly irritating pro-inflammatory fats that splatter on the insides
of your arteries.
A build up
of plaque in the arteries results from a damaged blood vessel having calcium
deposited there so that you can remain alive.
Atherosclerosis, or the buildup
of plaque in the arteries was originally considered to be cholesterol sludge
depositing in the arterial walls.
Learn about the world's very best treatments for arteriosclerosis known as hardening
of the arteries via
plaque deposits.
Scientists have identified a basic pathologic process underlying Alzheimer's development that involves the formation
of abnormal protein
deposits in the brain known as beta - amyloid
plaques, but they still aren't entirely sure what causes this to happen.
Basically, if you have significant internal inflammation, this
plaque will be
deposited as a healing agent regardless
of whether you have high or low cholesterol.
TMAO is thought to encourage fatty
plaque deposits to form within arteries (atherosclerosis), and therefore, the more TMAO you have in your blood the greater your risk
of heart disease might be.
Over time, too much glucose in the blood can lead to increased
plaque deposits on the insides
of the blood vessel walls.
The skin on the shins gets thick with raised
plaques or nodules due to
deposits of altered material in the skin.
Even with regular brushing
plaque and tartar are
deposited on the tooth surface, out
of sight, above and below the gumline,.
Deposits of plaque build - up on your pet's teeth.
Made with North Atlantic alga Ascophyllum nodosum, their line
of food additive, treats, and bones for both cats and dogs prevent dental
plaque from sticking to the teeth and soften already existing tartar
deposits.
These products will wipe off
plaque deposits from the surface
of the tooth and, though they lack the ability to pick food particles out
of the gum socket, they are probably the next best thing to brushing and, like brushing, these products are best used daily.
It begins with
deposits of plaque which harden to form tartar.
Fourth, a POPD begins with the removal
of supra - gingival
deposits of plaque and calculus from the buccal, lingual and interproximal surfaces.
When the tooth encounters a piece
of kibble, the morsel shatters and subsequently provides no abrasive effect to scrape
plaque (invisible layer
of bacteria), tartar (yellow - brown
deposits from chronic bacterial growth), or other food debris from tooth surfaces.
STEP 4: Supra - gingival scaling — a POPD begins with the removal
of supra - gingival
deposits of plaque and calculus from the buccal, lingual and interproximal surfaces.
This
plaque, in turn leads to
deposits of rock - hard tartar along the gum line.