Not exact matches
Current efforts focus
on elucidating the role of molecular chaperones compartmentalized in mitochondria in regulating tumor bioenergetics,
adaptation to
cellular stress and cell survival.
How do changes
on a
cellular level contribute to
adaptation processes that allow the brain (and with it its owner) to age successfully?
Our model should allow studies of the impact of aneuploidy
on molecular
adaptations and
cellular fitness.IMPORTANCE Aneuploidy is usually detrimental in multicellular organisms, but in several microorganisms, it can be tolerated and even beneficial.
Are there any respectable studies that show,
on a
cellular level, that slow fiber is slow, and nothing else, that fast fiber is fast and nothing else, or could these be just particular
adaptations that single fibers are capable of in response to the tasks put before them?