Since the amendments related to the article
on copper toxicosis, she felt that it may be necessary to have any suggested changes «examined» by her own independent examiners.
However, the Health Group has been in contact with «owner» of a recently posted website on «dog health» pointing out discrepancies in the information
on copper toxicosis and she has agreed to review this in the light of any suggestions made and to amend the site content, where necessary.
egrettably, it is noticeable that some websites have failed to update their information
on copper toxicosis to take into account the current situation, specifically in relation to testing.
Regrettably, it is noticeable that some websites have failed to update their information
on copper toxicosis to take into account the current situation, specifically in relation to testing.
It was reported that a member of the Health Group, acting as a «private breeder», had already contacted the website «owner» (Carol Fowler) expressing concern about various comments in the «linked article»
on copper toxicosis and that she had expressed the view that she was not adverse to amending the site content but would need a submission from the BTHG outlining the concerns.
Following a seminar
on copper toxicosis, jointly organised by the Animal Health Trust and the Kennel Club in June 2001, the secretary of the NBTC wrote to the Kennel Club seeking their advice on «the way forward».
Not exact matches
The BTHG Report for 2017 (including an update
on the situation re
Copper Toxicosis research) has now been produced.
The hunt is now
on for other
copper toxicosis gene (s).
Click
on «Canine DNA Testing» in the «Related Links» panel and then scroll down the page to make the appropriate links for «Bedlington terrier» - «
Copper Toxicosis».
This is a serious shortcoming because of the significance of dogs that would / could be classed as «carriers» and which would never develop
copper toxicosis but, never - the-less, had the potential to pass
on a copy of the mutant gene to their offspring.
This was an indirect test based
on the identification of a small section of DNA which had been found to be associated with the mutation thought to cause
copper toxicosis.