The total indirect effect was β = −.04, p <.01, and the total effect of self - esteem
on depressive symptoms at T5 was β = −.08, p =.01.
Not exact matches
Although none of the groups reported
on drug side effects, while Guo who tested only breastfed infants reported
on significant decrease in infant crying, and decrease in
depressive symptoms at one month and
at two months respectively, Sung who tested both formula fed and breastfed infants reported
on increase crying in the probiotic treated infants (particularly in the formula fed infants) compared to placebo with no effect
on maternal
depressive symptoms.
One randomized controlled trial comparing home - visited families with control participants who received other community services found a statistically significant difference in mean
depressive symptoms at two years post-enrollment, but this contrast was nonsignificant
at three years post - enrollment.15 A second study of Early Head Start found no differences in
depressive symptoms between intervention and control group participants post-intervention, although a difference was detected
at a longer - term follow - up prior to children's enrollment in kindergarten.10 Other randomized controlled trial studies have not found effects of home visitation
on maternal
depressive symptoms.12, 16,17
Mothers reported more
symptoms of psychological distress24, 25 and low self - efficacy.26, 27 And, although mothers report more
depressive symptoms at the time their infants are experiencing colic, 28,29 research
on maternal depression 3 months after the remittance of infant colic is mixed.30, 31 The distress mothers of colic infants report may arise out of their difficulties in soothing their infants as well as within their everyday dyadic interactions.32 The few studies to date that have examined the long - term consequences of having a colicky child, however, indicate that there are no negative outcomes for parent behaviour and, importantly, for the parent - child relationship.
For example,
on the Beck Depression Inventory (BDI), a widely used questionnaire in which a score of 19 or above indicates major depression, women in the study group saw their
depressive symptoms decline from an average of 27
at the beginning of therapy to 9.6 eight months after the program concluded.
«This study supports the use of a yoga and coherent breathing intervention in major
depressive disorder in people who are not
on antidepressants and in those who have been
on a stable dose of antidepressants and have not achieved a resolution of their
symptoms,» explained corresponding author Chris Streeter, MD, associate professor of psychiatry and neurology
at Boston University School of Medicine and a psychiatrist
at Boston Medical Center.
In a 2014 meta - analysis, Goyal and his partners
at Johns Hopkins found that mindfulness meditation might be
on par with antidepressants in treating
depressive symptoms.
Researchers
at the National University of Singapore reviewed 17 studies that examined the effects of music
on depression, and found that playing your favorite tunes as little as once a week can help reduce
depressive symptoms.
At both baseline and follow - up there was a high rate of
depressive symptoms with one third of the group scoring 14 or more
on the Beck Depression Inventory (a questionnaire designed to measure severity of
depressive symptoms).
At the same time, it is worth noting that some programs did identify small effects
on stress and
depressive symptoms and that others have specifically targeted reducing maternal
depressive symptoms and have obtained stronger results.99
At 12 weeks, the intervention group adjusted mean score for
depressive symptoms on the BDI - II was significantly lower than the control group by 5.8 points (95 % CI − 11.1 to − 0.5) after adjusting for baseline depression scores, anxiety, sociodemographics, psychotropic medication use and clustering by practice.
However, because the duration of the current maternal
depressive episode
at baseline was correlated with the number of children's internalizing and externalizing
symptoms at baseline (Cynthia Ewell - Foster, PhD, et al, unpublished data, December 2005), and the extent of children's improvement following maternal remission depended
on the magnitude of improvement in their mothers, reverse causation is not likely to fully account for the association between maternal remission and child improvement.
Depressive symptoms and chronic medical conditions have unique, additive effects
on adults» ability to function adequately
at home and in the community.40
Main effect of treatment and treatment by recruitment source interaction
on estimated mean change in self - rated
depressive symptoms (PHQ - 9) from baseline to post and follow - up and minimally clinically relevant change of PHQ - 9
at post-assessment.
Despite the negative effect maternal depression can have
on children, earlier studies in this area have focused
on smaller samples typically collected within 1 site or community.3, 8,20 The purpose of this multisite, cross-sectional study of 5820 children who attended a Head Start program and their families was to describe key aspects of the family environment and demographic factors related to maternal
depressive symptoms in a diverse national sample of low - income families whose children attended Head Start as preschoolers and were attending kindergarten
at the time of the interview.
The moderating effects of neighbourhood environmental attributes
on the associations between living arrangements and
depressive symptoms are summarised in table 4, where we report the ranges of values of the environmental attributes for which the associations between living arrangements and
depressive symptoms were significant
at the probability levels of 0.05, 0.01 or 0.001 (as appropriate).
The primary outcome measured
at 12 and 26 weeks follow - up was
depressive symptoms on the Beck Depression Inventory 2nd edition (BDI - II).26 Social functioning
on the Work and Social Adjustment Scale (WSAS) 27 and quality of life
on the EuroQol Five - item, Five - level (EQ - 5D - 5L) Scale28 were also measured
at 12 and 26 weeks follow - up.
As a result, they tend to spend more time onlooking (watching other children without joining) and hovering
on the edge of social groups.8, 11 There is some evidence to suggest that young
depressive children also experience social impairment.12 For example, children who display greater
depressive symptoms are more likely to be rejected by peers.10 Moreover, deficits in social skills (e.g., social participation, leadership) and peer victimization predict
depressive symptoms in childhood.13, 14 There is also substantial longitudinal evidence linking social withdrawal in childhood with the later development of more significant internalizing problems.15, 16,17 For example, Katz and colleagues18 followed over 700 children from early childhood to young adulthood and described a pathway linking social withdrawal
at age 5 years — to social difficulties with peers
at age 15 years — to diagnoses of depression
at age 20 years.
One randomized controlled trial comparing home - visited families with control participants who received other community services found a statistically significant difference in mean
depressive symptoms at two years post-enrollment, but this contrast was nonsignificant
at three years post - enrollment.15 A second study of Early Head Start found no differences in
depressive symptoms between intervention and control group participants post-intervention, although a difference was detected
at a longer - term follow - up prior to children's enrollment in kindergarten.10 Other randomized controlled trial studies have not found effects of home visitation
on maternal
depressive symptoms.12, 16,17
Figure 1a and b graphically presents the simple slopes for predicting functional limitations and
depressive symptoms, respectively, based
on poor versus good self - reported vision (plotted
at ± 1 SD around the mean) for those with low and high relationship satisfaction (computed
at ± 1 SD around the mean).
This finding of the lack of impact
on cognition is not surprising
at one level as cognition is not targeted in either CBT or ST.. In fact there is surprisingly little evidence regarding the impact of psychotherapies or pharmacotherapy for depression
on cognition, although the persistence of cognitive dysfunction despite improvement in
depressive symptoms following treatment suggests that these most current treatments for depression are having little impact in this area (1).
The purpose of this study was to examine the effects of the Strong African American Families (SAAF)
on a subset of 167 families in which the primary caregivers demonstrated elevated levels of
depressive symptoms at pretest as indicated by a score of 16 or higher
on the Center for Epidemiologic Studies — Depression scale (CES — D).
In addition, interventions to prevent youth depression may benefit from a focus
on enhancing family understanding of youth depression, improving parenting skills, and also
on addressing parental
depressive symptoms that may affect the efficacy of interventions targeting
at - risk youth.
In order to identify the number and patterns of these trajectories,
depressive symptoms should be measured
on at least three different times and modeled using a group based trajectory approach.
So we were able to look
at the differences between unhappily married people who divorced and those who stayed married
on a number of psychological variables, including: global happiness,
depressive symptoms, sense of personal mastery, self - esteem, hostility, autonomy, sense of purpose in life, and self - acceptance, as well as indirect measures such as days of drinking and number of drinks per day.
Model 1 included the marital status variable and covariates (age, education, equivalent household income,
at least one disability, number of chronic diseases, number of children, participation in economic activity, and number of social activities) as the second step in regressing
depressive symptoms (dependent variable)
on marital status (independent variable).
We plan to: (a) identify high risk adolescents based
on elevated scores
on a screening measure of
depressive symptoms that is delivered in primary care; (b) recruit 400 (200 per site) of these
at - risk adolescents to be randomized into either the CATCH - IT or the Educational group; and (c) assess outcomes
at 2, 6, 12, 18 and 24 months post intake
on measures of
depressive symptoms,
depressive diagnoses, other mental disorders, and
on measures of role impairment in education, quality of life, attainment of educational milestones, and family functioning; and to examine predictors of intervention response, and potential ethnic and cultural differences in intervention response.
Results indicated that maternal positive and negative parenting significantly concurrently predicted adolescent
depressive symptoms at all three waves, whereas TaqIA polymorphism had no main effect
on depressive symptoms.
Maternal
depressive symptoms were measured
at discharge using the Center for Epidemiologic Studies Depression Scale (CES - D; Radloff, 1977), a 20 - item self - report questionnaire of
depressive symptoms rated
on a 4 - point scale (0 = rarely / none of the time to 3 = all the time).
Mothers with an infant aged up to 12 months were recruited
at eight mental health centers in The Netherlands, if they met the following inclusion criteria: (a) having a diagnosis of a major
depressive episode or dysthymia according to the DSM - IV criteria [52](95 %) and / or scoring above 14
on the Beck Depression Inventory [53] indicating increased levels of
depressive symptoms (5 %); (b) having adequate fluency in Dutch; and (c) receiving professional outpatient treatment for their depression.
Children of mothers who are depressed or who have
depressive symptoms are
at increased risk for developmental delay, 1 behavioral problems, 2 depression, 3 asthma morbidity, 4 and injuries.5 Depressed mothers are less likely to engage in preventive parenting practices6 and are more likely to use child health care services.7 Though research initially focused
on postpartum depression, it is clear that maternal
depressive symptoms often persist after the postpartum period, 8 and this persistence further increases the effect
on children's health.9 As a result, the pediatric role in identifying and addressing maternal
depressive symptoms has received increasing attention.10 - 13
Of the sample of 9,600 mothers who had information
on alcohol use, 57.7 % (n = 5,539) of offspring provided
at least two measures of
depressive symptoms.
Mothers were identified as having persistent
depressive symptoms if their scores
on the CES - D were ≥ 16
at all 3 time points, as ever having
depressive symptoms if their scores were ≥ 16
at 1 or 2 time points, and as never having
depressive symptoms if their scores were always < 16.
Furthermore, despite a rich empirical and theoretical literature
on victimization and relationship dysfunction (O'Leary 1993; O'Leary and Vivian 1990; O'Leary et al. 1992; Stuart and Holtzworth - Munroe 2005) and between
depressive symptoms and relationship dysfunction (e.g., Beach et al. 1990; Coyne 1976; Whisman 2001), few studies have looked
at the three variables simultaneously.
We focused
on depressive symptoms in early adolescence (i.e.,
at age 14 years).
Participants were 142
at - risk Hispanic adolescents (54 % male, ages 14 — 19) who reported
on their anxious and
depressive symptoms, as well as their teachers who reported
on adolescents» ADHD
symptoms, ODD
symptoms, academic problems, and social problems.
Intraclass correlations
at Time 1 and Time 2 for examining socialization effects
on alcohol misuse and
depressive symptoms by dyad type and gender
We used prospective data from a large UK based population cohort (ALSPAC) to investigate the association between parental alcohol use, measured in units, (assessed
at ages 4 and 12 years) with childhood conduct trajectories, (assessed
on six occasions from 4 to 13.5 years, n = 6,927), and adolescent
depressive symptoms (assessed
on four occasions from ~ 13 to ~ 18 years, n = 5,539).
On the basis of CES - D scores
at all 3 time points, participants were divided into 3
symptom categories; 40 % of all participants never had
depressive symptoms, 48 % ever had
depressive symptoms, and 12 % had persistent
depressive symptoms.
We did not find significant effects of the fluctuation of maternal
depressive symptoms on FC
at 6 months, however, we did observe a negative relationship between the fluctuation of maternal
depressive symptoms and the right frontal FC
at 18 month.
We also compared this model with a model in which the direct effect of maternal
depressive symptoms on children's social preference was set
at zero (Model 1a).
We also compared these models with a model (Model 2a) in which the direct effect of maternal
depressive symptoms on peer social preference was set
at zero.
After adjusting for post-conceptual age
on the EEG visit day, greater - postnatal - than - prenatal maternal
depressive symptoms were significantly associated with greater right frontal activity (Table 2, Fig 2b) and greater relative right frontal asymmetry in infants
at 6 months of age (Table 2, Fig 2c).
Briefly, in the female sample, greater - postnatal - than - prenatal maternal
depressive symptoms were significantly associated with greater right frontal activity (β = -0.262, p = 0.020, df = 72) and greater relative right frontal asymmetry in infants
at 6 months of age (β = -0.426, p < 0.001, df = 72) after adjusting for post-conceptual age
on the EEG visit day.
The BDNF val (66) met × 5 - HTTLPR × Child Adversity interaction
on depressive symptoms: An attempt
at replication