Not exact matches
After administering PET scans to 4000 people previously diagnosed with either
mild cognitive
impairment (MCI) or dementia and treated for Alzheimer's to test for the presence of amyloid plaque,
only 53.3 % of patients with MCI and 70.5 % with dementia tested positive.
In Alzheimer's disease, this loss of synapses occurs very early on, when people still
only have
mild cognitive
impairment, and long before the nerve cells themselves die.
This result corresponds with earlier findings that vitamin B supplements slow the rate of brain atrophy in
mild cognitive
impairment only in individuals with a good Omega - 3 level to begin with.
• Increased risk of heart attack • Increased risk of stroke • Increased risk of blood clots • Increased risk of breast cancer • Reduced risk of colorectal cancer • Fewer fractures • No protection against
mild cognitive
impairment and increased risk of dementia (study included
only women 65 and older)
The age - related decline in growth hormone and associated IGF - 1 has been linked to age - related muscle atrophy, increased adipose tissue, and neuronal dysfunction.2, 9 In fact, growth hormone replacement therapy in elderly men has been shown to increase lean body mass.9 Growth hormone therapy (1 mg / day — for 5 months) has also been used to improve cognitive function in healthy adults and adults with
mild cognitive
impairment.10 While
only modest amounts of growth hormone cross the blood - brain barrier, IGF - 1, which is actually responsible for the cognitive benefits, gets across just fine.
However, the decreases weren't yet seen in the cortices of patients with
only the
mild cognitive
impairment that precedes Alzheimer's.
In terms of an actual diagnosis of
mild cognitive
impairment, however, the researchers
only identified 37 people who developed that condition during the study, and the diets didn't have a significant effect on the risk for that diagnosis.