Sentences with phrase «onset antisocial»

The primary aim of this paper is to discuss the evidence that specific neurobiological systems are involved in the etiology of childhood - onset antisocial behavior.

Childhood - onset versus adolescent - onset antisocial conduct in males: Natural history from age 3 to 18

Distinguishing the early - onset / persistent and adolescence - onset antisocial behaviour types: from birth to sixteen years

A developmental model for early - and late - onset antisocial behavior.

[jounal] Moffit, T.E / 1996 / Childhood - onset versus adolescent - onset antisocial conduct problems in males: Natural history from ages 3 to 18 years.

Romantic relationships of young people with childhood and adolescent onset antisocial behavior problems

CONCLUSIONS: Early - onset antisocial behavior in a high - risk clinical group is predicted by a specific COMT gene variant previously linked with prefrontal cortical function and birth weight, and those possessing the val / val genotype are more susceptible to the adverse effects of prenatal risk as indexed by lower birth weight.

Moffit, T.E., Caspi, A., Dickson, N., Silva, P. and Stanton, W. (1996) «Childhood onset versus adolescent onset antisocial conduct problems in males: Natural history from ages 3 to 18 years», Development and Psychopathology, 8, pp399 - 424

OBJECTIVE: To test the a priori hypothesis that this genetic variant predicts early - onset antisocial behavior in a high - risk sample and further examine the effects of birth weight, an environmentally influenced index of prenatal adversity previously linked to childhood disruptive behaviors and genotype x birth weight interaction.

Distinguishing the early - onset / persistent and adolescence - onset antisocial behavior types: from birth to 16 years

Longitudinal follow - up of adolescents with late - onset antisocial behavior: a pathological yet overlooked group

Rather fewer meet the diagnostic criteria for research, which for the oppositional defiant type of conduct disorder seen in younger children require at least four specific behaviours to be present.7 The early onset pattern — typically beginning at the age of 2 or 3 years — is associated with comorbid psychopathology such as hyperactivity and emotional problems, language disorders, neuropsychological deficits such as poor attention and lower IQ, high heritability, 8 and lifelong antisocial behaviour.9 In contrast, teenage onset antisocial behaviour is not associated with other disorders or neuropsychological deficits, is more environmentally determined than inherited, and tends not to persist into adulthood.9

Given that early - onset antisocial behavior is associated with (1) subtle neurological impairment, (2) harsh, punitive, and neglectful parenting, and (3) family contexts characterized by substance abuse and criminal behavior,2 - 5 it is important to note that this program has affected these aspects of maternal, child, and family functioning at earlier phases in the child's development.6 - 11 Moreover, genetic vulnerability to impulsivity and aggression is expressed much more frequently when vulnerable rhesus monkeys experience aberrant rearing21 (also Allyson J. Bennett, PhD, K. Peter Lesch, Armin Heils, et al, unpublished data, 1998), adding to the plausibility of the findings reported here.

In general, these findings are consistent with program effects on early - onset antisocial behavior rather than on the more common and less serious antisocial behavior that emerges with puberty.3 The mere presence of arrests, convictions, and probation violations by the time the children were 15 years old suggests that these children started offending early and that they may be on life - course trajectories that portend recurrent and more serious offenses in the future.

The adolescent - onset variety, although sometimes expressed as aggression toward peers, is generally less serious (eg, shoplifting, lying to teachers and parents) and occurs so frequently that some consider it normative.3 Childhood - onset antisocial behavior is associated with neuropsychological deficits (eg, impaired language and intellectual functioning, attention - deficit / hyperactivity disorder) and harsh, rejecting parenting early in the child's life.4, 5 The adolescent - onset type has been hypothesized to be a reaction to the limited number of responsible roles for adolescents in Western societies.3

Not exact matches

The nature of the co-occurrence of multiple antisocial behaviors, including substance use, during adolescence, and the causal factors which contribute to the early onset and maintenance of youth substance use and substance abuse are discussed.

Notwithstanding these gender - specific risk and protective factors, in most cases, the same factors — ADHD, negative temperament, impulsivity, compromised intelligence — predict antisocial behavior in both males and females, as suggested by the substantial overlap shown in figure 4.99 Although some analysts have argued the need to concentrate on the commonalities in predictors of male and female offending, it is also important to note the areas in which risk factors differ by gender.100 Even if the differences between male and female offenders are confined to only a few key areas, the differences in these areas — for example, sensitivity to victimization, timing of onset of persistent offending, prevalence of mental health problems — can be substantial and can profoundly influence the effectiveness of risk assessments and treatment programs.

Two studies have identified groups of girls exhibiting chronically high levels of antisocial behavior across childhood and early adolescence and having an increased risk for continued antisocial behavior.60 In addition, Odgers and several colleagues found that 7.5 percent of all girls between the ages of seven and fifteen displayed an early - onset of offending that persisted into adolescence and that this pattern was similar to boys of the same age.61 Other studies suggest that although strongly aggressive behavior in girls before the age of seven is rare, continuity of offending for such girls may be stronger than that among comparable boys and that such early problem behavior in girls should be considered a significant warning sign of potential future problems.62

Age of Onset Some studies indicate that both boys and girls tend to begin their antisocial careers around the age of fifteen, with the average age of onset differing by no more than six months across genders.44 Other research, however, finds that females begin offending when they are younger than males are.45 Notably, gender differences in the age of onset tend to be most pronounced for serious or aggressive types of delinquency, while less serious problem behaviors, such as drug and alcohol - related offenses, have less gender - differentiated progressioOnset Some studies indicate that both boys and girls tend to begin their antisocial careers around the age of fifteen, with the average age of onset differing by no more than six months across genders.44 Other research, however, finds that females begin offending when they are younger than males are.45 Notably, gender differences in the age of onset tend to be most pronounced for serious or aggressive types of delinquency, while less serious problem behaviors, such as drug and alcohol - related offenses, have less gender - differentiated progressioonset differing by no more than six months across genders.44 Other research, however, finds that females begin offending when they are younger than males are.45 Notably, gender differences in the age of onset tend to be most pronounced for serious or aggressive types of delinquency, while less serious problem behaviors, such as drug and alcohol - related offenses, have less gender - differentiated progressioonset tend to be most pronounced for serious or aggressive types of delinquency, while less serious problem behaviors, such as drug and alcohol - related offenses, have less gender - differentiated progressions.46

Females who exhibit early - onset (by age seven) persistent offending are more likely than other girls to engage in antisocial behavior at age thirty - two.30 For example, 75 percent of these early - onset persistent female offenders had, by age thirty - two, engaged in one or more violent acts, including violence toward partners (44.8 percent) and children (41.7 percent).

These children are more likely to develop adult antisocial personality disorder than individuals with the adolescent - onset type.

Distinctions based on age of onset have proven important for understanding heterogeneity within attention - deficit / hyperactivity disorder51 and antisocial disorder, 52,53 in which childhood onset has worse implications for course, recurrence, familial transmission, and treatment resistance.54 Research on schizophrenia is also benefiting from a focus on childhood neurodevelopmental processes55 and juvenile - onset symptoms.56 The present study and others1 illustrate that the distinction between juvenile vs adult - onset MDD is important for understanding heterogeneity within depression as well.

Findings suggest that yearly FCU services within the context of social, health, and educational services in early childhood can potentially prevent early - onset trajectories of antisocial behavior.

Children with early - onset conduct problems (CPs) are at high risk for chronic antisocial and aggressive behaviour, and a variety of social and mental health problems in adolescence and adulthood (e.g. delinquency, psychiatric disorders, substance use, school dropout; Fergusson et al. 2005; Kratzer and Hodgins 1997).

Among ADHD girls, childhood - onset (< 12 years) CD was predicted by paternal antisocial personality disorder (ASPD), while adolescent - onset CD (⩾ 12 years) was predicted by family conflict.

Delinquents have repeatedly been shown to have an IQ that is 8 - 10 points lower than law abiding peers - and this is before the onset of antisocial behaviour.24 Other traits predisposing to conduct problems include irritability and explosiveness, lack of social awareness and social anxiety, and reward seeking behaviour.

Psychological characteristics include low IQ, impulsivity, hyperactivity, lack of empathy, and fearlessness.12, 13 Parental risks include low levels of education, antisocial behavior, poor parenting skills, maternal early onset of childbearing, and family discord.14 — 20 There is evidence of an intergenerational transmission of these problems through both genetic and environmental channels.18, 19,21 — 24 Developmental research also shows that the spontaneous onset of physical aggression in school - aged children is highly unusual.1, 7,25 Instead, the developmental precursors of chronic physical aggression are present before school entry.

Earlier onset of CD / ODD has been found to be correlated with the development of antisocial personality disorder, substance - related disorders, increased rates of drug use (tobacco and alcohol), mood disorders, anxiety disorders, somatoform disorders, and higher accident rates [3].

The likelihood of developing antisocial personality disorder in adult life is increased if the individual experienced childhood onset of conduct disorder (before age 10 years) and accompanying attention - deficit / hyperactivity disorder.

In about 40 percent of cases, childhood onset conduct disorder develops into adult antisocial personality disorder.8 Adolescent conduct disorder should be considered in social context.

Mesh terms: Adolescent Age Factors Age of Onset Antisocial Personality Disorder Attention Deficit Disorder with Hyperactivity Birth Weight Catechol O - Methyltransferase Child Child, Preschool Female Genetic Variation Genotype Humans Infant, Newborn Male Prefrontal Cortex Pregnancy Prenatal Exposure Delayed Effects Psychiatric Status Rating Scales Regression Analysis Risk Factors Valine

First, the findings confirm previous suggestions that prevention of the initial onset of conduct problems is key to preventing antisocial behavior (Tremblay and Japel 2003).

It is a period of biological, cognitive and social change of such magnitude and rapidity that it is no surprise to find that it is associated with the onset or exacerbation of a number of health - related problems including depression (1), eating disorders (2), substance abuse and dependence (3 — 5), risky sexual behaviour (6), antisocial and delinquent activity (7) and school dropout (8).

Abbreviations CP: Conduct problems; Conduct Disorder (CD); SDQ: Strengths and Difficulties Questionnaire; DAWBA: Development and Well Being Assessment; EOP: Early - Onset Persistent; AO: Adolescent - Onset; AL: Adolescence - Limited; CL: Childhood - Limited; ASB: Antisocial Behaviour; CBCL: Child Behaviour Checklist

Neighborhood contextual factors and the onset and progression of early - starting antisocial pathways

Antisocial personality disorder is only diagnosed in people over age 18, the symptoms are similar to those of conduct disorder, and the criteria for diagnosis include the onset of conduct disorder before the age of 15.

Not only can symptoms be distressing, AUD can trigger a cascade of lifelong adverse outcomes, such as: other mental disorders, suicide, serious unintentional injury, illicit drug use, antisocial behaviour, as well as early onset of heart disease, stroke and cancer.3 While the peak age for the onset for AUD is 18 — 24 years, the factors that predict the transition from alcohol use to AUD symptom onset and from symptom onset to diagnosable AUD remain largely unknown.

The life - course persistent antisocial group was characterized by high - risk childhood backgrounds (e.g., inadequate parenting, neurocognitive problems, and temperament and behavior problems) and appeared to be at highest risk for adverse outcomes in adulthood, whereas the adolescent onset group was considered to be quite normative, and thus at much lower risk for such adverse outcomes (Moffitt and Caspi 2001; Moffitt et al. 2002; Odgers et al. 2008).

In their taxonomy of antisocial behavior, Moffitt (1993) and Moffitt et al. (2002) demonstrated that two prototypes of offenders, the life - course - persistent (LCP: childhood onset and continuing into adulthood) and adolescent - limited group (AL: beginning in adolescence and desisting in young adulthood), account for the bulk of antisocial behavior (two additional groups were identified and labeled abstainers and low - level chronics, but these groups were relatively small, i.e., around 5 % and 8 % respectively).

Adolescent - onset alcohol abuse exacerbates the influence of childhood conduct disorder on late adolescent and early adult antisocial behaviour.

The onset and persistence of conduct problems is important, as the earlier the onset, the greater the (a) co-morbidity of adjustment problems, such as emotional difficulties and hyperactivity, and the (b) risk for a life - course trajectory of antisocial behaviour and lifestyle (Moffitt 2006).

Four antisocial behavior trajectory groups were identified among females and males using general growth mixture modeling and included life - course persistent (LCP), adolescent - onset, childhood - limited, and low trajectory groups.

Second, antisocial behavior apparently develops in at least two separate pathways — child - onset versus adolescent - onset — that differ markedly regarding types of antisocial behavior displayed, persistence, and perhaps etiology.